Hypothalamic glucose-sensing mechanisms

Bibliographic Details
Title: Hypothalamic glucose-sensing mechanisms
Authors: Nal Ae Yoon, Sabrina Diano
Source: Diabetologia. 64:985-993
Publisher Information: Springer Science and Business Media LLC, 2021.
Publication Year: 2021
Subject Terms: Neurons, 0301 basic medicine, 0303 health sciences, Diabetes, Tanycytes, Hypothalamus, Brain, Review, Counterregulatory responses, 3. Good health, 03 medical and health sciences, Glucose, Diabetes Mellitus, Type 2, Astrocytes, Glucose-sensing, Obesity, Risk Factors, Animals, Homeostasis, Humans, Energy Metabolism
Description: Chronic metabolic diseases, including diabetes and obesity, have become a major global health threat of the twenty-first century. Maintaining glucose homeostasis is essential for survival in mammals. Complex and highly coordinated interactions between glucose-sensing mechanisms and multiple effector systems are essential for controlling glucose levels in the blood. The central nervous system (CNS) plays a crucial role in regulating glucose homeostasis. Growing evidence indicates that disruption of glucose sensing in selective CNS areas, such as the hypothalamus, is closely interlinked with the pathogenesis of obesity and type 2 diabetes mellitus. However, the underlying intracellular mechanisms of glucose sensing in the hypothalamus remain elusive. Here, we review the current literature on hypothalamic glucose-sensing mechanisms and discuss the impact of alterations of these mechanisms on the pathogenesis of diabetes.
Document Type: Article
Language: English
ISSN: 1432-0428
0012-186X
DOI: 10.1007/s00125-021-05395-6
Access URL: https://link.springer.com/content/pdf/10.1007/s00125-021-05395-6.pdf
https://pubmed.ncbi.nlm.nih.gov/33544170
https://hdl.handle.net/11588/990010
https://link.springer.com/article/10.1007/s00125-021-05395-6
https://europepmc.org/article/PMC/PMC8087998
https://www.scilit.net/article/b8aac2f3daaa42c79a35d406e7751f79?action=show-references
https://pubmed.ncbi.nlm.nih.gov/33544170/
https://www.ncbi.nlm.nih.gov/pubmed/33544170
https://link.springer.com/content/pdf/10.1007/s00125-021-05395-6.pdf
https://hdl.handle.net/11588/990010
https://doi.org/10.1007/s00125-021-05395-6
Rights: Springer TDM
Accession Number: edsair.doi.dedup.....347c13da9ab852d779332676d16cbd26
Database: OpenAIRE
Description
ISSN:14320428
0012186X
DOI:10.1007/s00125-021-05395-6