Academic Journal

Cytokeratin-18 increase as a signal for early liver cell changes in high-fat diet-induced obesity before overt steatohepatitis.

Λεπτομέρειες βιβλιογραφικής εγγραφής
Τίτλος: Cytokeratin-18 increase as a signal for early liver cell changes in high-fat diet-induced obesity before overt steatohepatitis.
Συγγραφείς: Özdemir Ö; Department of Internal Medicine, Medicine Faculty of Ordu University, Ordu, Türkiye., Akalın Ç; Department of General Surgery, Medicine Faculty of Ordu University, Ordu, Türkiye., Ekiz F; Department of Gastroenterology. Medical Park Ordu Hospital, Ordu, Türkiye., Öğüten PN; Department of Histology and Embryology. Medicine Faculty of Samsun University, Samsun, Türkiye., Baş O; Department of Anatomy, Medicine Faculty of Ordu University, Ordu, Türkiye., Bayrak A; Department of Medical Biochemistry, Medicine Faculty of Ordu University, Ordu, Türkiye., Kaşko Arıcı Y; Department of Biostatistics, Medicine Faculty of Ordu University, Ordu, Türkiye.
Πηγή: Frontiers in endocrinology [Front Endocrinol (Lausanne)] 2026 Jul 08; Vol. 17, pp. 1853417. Date of Electronic Publication: 2026 Jul 08 (Print Publication: 2026).
Τύπος έκδοσης: Journal Article
Γλώσσα: English
Στοιχεία περιοδικού: Publisher: Frontiers Research Foundation] Country of Publication: Switzerland NLM ID: 101555782 Publication Model: eCollection Cited Medium: Print ISSN: 1664-2392 (Print) Linking ISSN: 16642392 NLM ISO Abbreviation: Front Endocrinol (Lausanne) Subsets: MEDLINE
Imprint Name(s): Original Publication: [Lausanne : Frontiers Research Foundation]
Ιατρικοί όροι (MeSH): Keratin-18*/metabolism , Keratin-18*/blood , Diet, High-Fat*/adverse effects , Obesity*/metabolism , Obesity*/pathology , Obesity*/etiology , Non-alcoholic Fatty Liver Disease*/pathology , Non-alcoholic Fatty Liver Disease*/etiology , Non-alcoholic Fatty Liver Disease*/metabolism , Non-alcoholic Fatty Liver Disease*/blood , Liver*/pathology , Liver*/metabolism , Fatty Liver*/pathology , Fatty Liver*/etiology , Fatty Liver*/metabolism, Biomarkers/metabolism ; Animals ; Male ; Rats ; Rats, Wistar ; Insulin Resistance
Περίληψη: Objective: The major cause of non-alcoholic fatty liver disease (NAFLD) is obesity; in fact, this condition has been defined as metabolic dysfunction-associated steatotic liver disease (MASLD). Non-invasive diagnosis and grading of NAFLD are the subjects of the studies. Cytokeratin-18 (CK-18) is particularly highlighted as an indicator of liver cell death and steatohepatitis, but its relationship with obesity remains unclear. This study aimed to evaluate the metabolic parameters, CK-18 levels, and liver histopathology in high-fat diet-induced obesity in rats.
Methods: Rats were fed either a normal-fat diet (NFD) or a high-fat diet (HFD) for eight weeks. Body weights, metabolic-liver parameters and CK-18 levels were assessed at study end. The liver histopathological changes (steatosis, lobular inflammation, ballooning, fibrosis) and NAFLD activity score were examined.
Results: Increased final body weight (335.8 ± 20.2 vs 291.5 ± 6.25 g; p<0.001), glucose (186.4 ± 46.9 vs 130.0 ± 16.2 mg/dL; p=0.011), insulin (4.30 ± 0.70 vs 2.23 ± 0.82 IU/L; p<0.001), HOMA-IR (2.03 ± 0.79 vs 0.73 ± 0.34; p=0.003), ALT (72.34 ± 12.35 vs 55.43 ± 5.75 U/L; p=0.009) and CK-18 (1.46 ± 0.18 vs 0.88 ± 0.18 ng/mL; p<0.001) levels and decreased AST/ALT ratio (1.16 ± 0.09 vs 1.32 ± 0.14; p=0.020) were observed in HFD group compared to NFD group. Histologically, all HFD specimens showed ballooning (8/8) and half showed steatosis (4/8). NAFLD score remained ≤3 in all specimens nevertheless CK-18 values tended to co-occur with higher ALT levels and ballooning formation within the HFD group.
Conclusıon: HFD-induced obesity was associated with increased CK-18 and the hepatocellular changes as remarkable ballooning without the overt steatohepatitis. These findings support CK-18 interpreting as a signal for early hepatocellular injury in obesity.
(Copyright © 2026 Özdemir, Akalın, Ekiz, Öğüten, Baş, Bayrak and Kaşko Arıcı.)
Competing Interests: The author(s) declared that this work was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Contributed Indexing: Keywords: cytokeratin (CK) 18; liver cell; metabolic dysfunction associated fatty liver disease (MASLD); non alcoholic fatty liver disease (NAFLD); obesity
Substance Nomenclature: 0 (Keratin-18)
0 (Biomarkers)
Entry Date(s): Date Created: 20260723 Date Completed: 20260723 Latest Revision: 20260723
Update Code: 20260723
PubMed Central ID: PMC13388190
DOI: 10.3389/fendo.2026.1853417
PMID: 42488000
Βάση Δεδομένων: MEDLINE
Περιγραφή
ISSN:1664-2392
DOI:10.3389/fendo.2026.1853417