SIRT3-activating, biodegradable poly-honokiol with high drug loading for thoracic aortic dissection therapy.

Λεπτομέρειες βιβλιογραφικής εγγραφής
Τίτλος: SIRT3-activating, biodegradable poly-honokiol with high drug loading for thoracic aortic dissection therapy.
Συγγραφείς: Qiao X; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Wang D; School of Chemistry, Xi'an Jiaotong University, Xi'an, Shaanxi 710049, PR China., Zhu H; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Zhang B; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Sun H; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Wang J; School of Chemistry, Xi'an Jiaotong University, Xi'an, Shaanxi 710049, PR China., Tan G; School of Chemistry, Xi'an Jiaotong University, Xi'an, Shaanxi 710049, PR China., Jiang L; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Peng X; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Zhang L; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Wang L; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Han S; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China., Meng L; School of Chemistry, Xi'an Jiaotong University, Xi'an, Shaanxi 710049, PR China; Instrumental Analysis Center, Xi'an Jiaotong University, Xi'an, Shaanxi 710049, PR China. Electronic address: menglingjie@mail.xjtu.edu.cn., Duan W; Department of Cardiovascular Surgery, Xijing Hospital of Air Force Military Medical University, Xi'an, Shaanxi 710032, PR China. Electronic address: duanweixun@126.com.
Πηγή: Journal of advanced research [J Adv Res] 2026 Aug; Vol. 86, pp. 605-619. Date of Electronic Publication: 2025 Nov 14.
Τύπος έκδοσης: Journal Article
Γλώσσα: English
Στοιχεία περιοδικού: Publisher: Cairo University, production and hosting by Elsevier B. V Country of Publication: Egypt NLM ID: 101546952 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2090-1224 (Electronic) Linking ISSN: 20901224 NLM ISO Abbreviation: J Adv Res Subsets: MEDLINE
Imprint Name(s): Original Publication: [Giza, Egypt] : Cairo University, production and hosting by Elsevier B. V.
Ιατρικοί όροι (MeSH): Biphenyl Compounds*/pharmacology , Biphenyl Compounds*/chemistry , Lignans*/pharmacology , Lignans*/chemistry , Sirtuin 3*/metabolism , Aortic Dissection*/drug therapy , Aortic Dissection*/metabolism, Dissection, Thoracic Aorta/drug therapy ; Muscle, Smooth, Vascular/drug effects ; Muscle, Smooth, Vascular/metabolism ; Myocytes, Smooth Muscle/drug effects ; Myocytes, Smooth Muscle/metabolism ; Reactive Oxygen Species/metabolism ; Animals ; Phenols ; Mice ; Allyl Compounds ; Humans ; Male ; Disease Models, Animal
Περίληψη: Introduction: Thoracic aortic dissection (TAD) is a life-threatening cardiovascular emergency with limited pharmacological treatment options.
Objectives: Building on the strong correlation between TAD and SIRT3 observed in our preliminary studies, we aim to leverage pharmacological interventions to enhance the clinical translation of this mechanism.
Methods: Based on the SIRT3 activator honokiol, we synthesized a highly efficient, pH-responsive and biodegradable poly-honokiol prodrug synthesized via a metal-free phenol-yne click polymerization strategy, achieving an unprecedented drug-loading content of 83.65%, and subsequently investigated its pharmacological activity and underlying mechanisms using a β-aminopropionitrile (BAPN)-induced TAD model.
Results: Compared with honokiol, poly-honokiol markedly improved survival and reduced TAD incidence in mice by preventing vascular smooth muscle cell (VSMC) loss. Mechanistic investigations reveal that poly-honokiol activates the mitochondrial deacetylase SIRT3, which induce the deacetylation of both the antioxidant enzyme SOD2 and the key ferroptosis regulator COX2, thereby reducing ferrous ion (Fe2+) accumulation and reactive oxygen species (ROS) levels while suppressing mitochondrial permeability transition pore (mPTP) opening, ultimately attenuating ferroptosis-induced VSMC loss.
Conclusion: Collectively, these findings demonstrate that poly-honokiol is a potent preventive candidate for TAD, offering both high drug-loading efficiency and targeted mitochondrial protection.
(Copyright © 2025. Published by Elsevier B.V.)
Competing Interests: Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.
Contributed Indexing: Keywords: Ferroptosis; Mitochondria; Poly-honokiol; Sirtuin 3; Thoracic aortic dissection
Substance Nomenclature: 0 (Biphenyl Compounds)
0 (Lignans)
EC 3.5.1.- (Sirtuin 3)
11513CCO0N (honokiol)
0 (Phenols)
0 (Allyl Compounds)
0 (Sirt3 protein, mouse)
0 (Reactive Oxygen Species)
Entry Date(s): Date Created: 20251116 Date Completed: 20260730 Latest Revision: 20260730
Update Code: 20260731
DOI: 10.1016/j.jare.2025.11.028
PMID: 41242496
Βάση Δεδομένων: MEDLINE
Περιγραφή
ISSN:2090-1224
DOI:10.1016/j.jare.2025.11.028