Academic Journal

Dexamethasone Modulates the Cytokine Response but Not COVID-19-Induced Coagulopathy in Critically Ill

Λεπτομέρειες βιβλιογραφικής εγγραφής
Τίτλος: Dexamethasone Modulates the Cytokine Response but Not COVID-19-Induced Coagulopathy in Critically Ill
Συγγραφείς: Dechamps, Mélanie, De Poortere, Julien, Octave, Marie, Ginion, Audrey, Robaux, Valentine, Pirotton, Laurence, Bodart, Julie, Gruson, Damien, Van Dievoet, Marie-Astrid, Douxfils, Jonathan, Haguet, Hélène, Morimont, Laure, Derive, Marc, Jolly, Lucie, Bertrand, Luc, Laterre, Pierre-François, Horman, Sandrine, Beauloye, Christophe
Πηγή: Dechamps, M, De Poortere, J, Octave, M, Ginion, A, Robaux, V, Pirotton, L, Bodart, J, Gruson, D, Van Dievoet, M-A, Douxfils, J, Haguet, H, Morimont, L, Derive, M, Jolly, L, Bertrand, L, Laterre, P-F, Horman, S & Beauloye, C 2023, 'Dexamethasone Modulates the Cytokine Response but Not COVID-19-Induced Coagulopathy in Critically Ill', International Journal of Molecular Sciences, vol. 24, no. 8, 7278. https://doi.org/10.3390/ijms24087278
Έτος έκδοσης: 2023
Συλλογή: Research Portal - University of Namur / Portail de la recherche de l'Université de Namur
Θεματικοί όροι: Humans, Cytokines, SARS-CoV-2, Critical Illness, COVID-19 Drug Treatment, COVID-19/complications, Dexamethasone/pharmacology, COVID-19, low dose dexamethasone, cytokine storm, inflammation, coagulopathy
Περιγραφή: Severe forms of coronavirus 2019 (COVID-19) disease are caused by an exaggerated systemic inflammatory response and subsequent inflammation-related coagulopathy. Anti-inflammatory treatment with low dose dexamethasone has been shown to reduce mortality in COVID-19 patients requiring oxygen therapy. However, the mechanisms of action of corticosteroids have not been extensively studied in critically ill patients in the context of COVID-19. Plasma biomarkers of inflammatory and immune responses, endothelial and platelet activation, neutrophil extracellular trap formation, and coagulopathy were compared between patients treated or not by systemic dexamethasone for severe forms of COVID-19. Dexamethasone treatment significantly reduced the inflammatory and lymphoid immune response in critical COVID-19 patients but had little effect on the myeloid immune response and no effect on endothelial activation, platelet activation, neutrophil extracellular trap formation, and coagulopathy. The benefits of low dose dexamethasone on outcome in critical COVID-19 can be partially explained by a modulation of the inflammatory response but not by reduction of coagulopathy. Future studies should explore the impact of combining dexamethasone with other immunomodulatory or anticoagulant drugs in severe COVID-19.
Τύπος εγγράφου: article in journal/newspaper
Περιγραφή αρχείου: application/pdf
Γλώσσα: English
ISSN: 1661-6596
1422-0067
Relation: info:eu-repo/semantics/altIdentifier/pmid/37108440; info:eu-repo/semantics/altIdentifier/pissn/1661-6596; info:eu-repo/semantics/altIdentifier/eissn/1422-0067
DOI: 10.3390/ijms24087278
Διαθεσιμότητα: https://researchportal.unamur.be/en/publications/af9342ce-0515-4af6-858c-5e8f13ad1cc6
https://doi.org/10.3390/ijms24087278
https://pure.unamur.be/ws/files/79857347/14959_2023_Dechamps.pdf
https://www.scopus.com/pages/publications/85156194201
Rights: info:eu-repo/semantics/openAccess
Αριθμός Καταχώρησης: edsbas.9BC5986B
Βάση Δεδομένων: BASE
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  Data: Dexamethasone Modulates the Cytokine Response but Not COVID-19-Induced Coagulopathy in Critically Ill
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  Data: Dechamps, M, De Poortere, J, Octave, M, Ginion, A, Robaux, V, Pirotton, L, Bodart, J, Gruson, D, Van Dievoet, M-A, Douxfils, J, Haguet, H, Morimont, L, Derive, M, Jolly, L, Bertrand, L, Laterre, P-F, Horman, S & Beauloye, C 2023, 'Dexamethasone Modulates the Cytokine Response but Not COVID-19-Induced Coagulopathy in Critically Ill', International Journal of Molecular Sciences, vol. 24, no. 8, 7278. https://doi.org/10.3390/ijms24087278
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  Data: Severe forms of coronavirus 2019 (COVID-19) disease are caused by an exaggerated systemic inflammatory response and subsequent inflammation-related coagulopathy. Anti-inflammatory treatment with low dose dexamethasone has been shown to reduce mortality in COVID-19 patients requiring oxygen therapy. However, the mechanisms of action of corticosteroids have not been extensively studied in critically ill patients in the context of COVID-19. Plasma biomarkers of inflammatory and immune responses, endothelial and platelet activation, neutrophil extracellular trap formation, and coagulopathy were compared between patients treated or not by systemic dexamethasone for severe forms of COVID-19. Dexamethasone treatment significantly reduced the inflammatory and lymphoid immune response in critical COVID-19 patients but had little effect on the myeloid immune response and no effect on endothelial activation, platelet activation, neutrophil extracellular trap formation, and coagulopathy. The benefits of low dose dexamethasone on outcome in critical COVID-19 can be partially explained by a modulation of the inflammatory response but not by reduction of coagulopathy. Future studies should explore the impact of combining dexamethasone with other immunomodulatory or anticoagulant drugs in severe COVID-19.
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