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1Academic Journal
Συγγραφείς: Kazyra, I.A., Sukala, A.V.
Πηγή: Zdorovʹe Rebenka, Vol 14, Iss 2, Pp 115-122 (2019)
CHILD`S HEALTH; Том 14, № 2 (2019); 115-122
Здоровье ребенка-Zdorovʹe rebenka; Том 14, № 2 (2019); 115-122
Здоров'я дитини-Zdorovʹe rebenka; Том 14, № 2 (2019); 115-122Θεματικοί όροι: IgA nephropathy, children, factors of progression, Pediatrics, IgA-нефропатия, дети, факторы прогрессирования, RJ1-570, IgA-нефропатія, діти, фактори прогресування, 3. Good health
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Σύνδεσμος πρόσβασης: http://childshealth.zaslavsky.com.ua/article/download/165548/167237
https://doaj.org/article/dd82ea0fe8ce460bb0b27d55bb639b20
http://childshealth.zaslavsky.com.ua/article/download/165548/167237
http://childshealth.zaslavsky.com.ua/article/view/165548
http://childshealth.zaslavsky.com.ua/article/view/165548 -
2Academic Journal
Συγγραφείς: L. A. Burova, A. N. Suvorov, A. A. Totolian, Л. А. Бурова, А. Н. Суворов, А. А. Тотолян
Πηγή: Medical Immunology (Russia); Том 24, № 2 (2022); 217-234 ; Медицинская иммунология; Том 24, № 2 (2022); 217-234 ; 2313-741X ; 1563-0625
Θεματικοί όροι: IgA-нефропатия, streptococcal IgFc-binding proteins, post-streptococcal glomerulonephritis, myocarditis, IgA nephropathy, Fc-связывающая активность стрептококков, аутоиммунные осложнения, постстрептококковый гломерулонефрит, миокардит
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Relation: https://www.mimmun.ru/mimmun/article/view/2450/1517; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8945; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8946; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8947; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8948; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8949; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8950; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8951; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8952; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8953; https://www.mimmun.ru/mimmun/article/downloadSuppFile/2450/8954; Бурова Л.А., Гаврилова Е.А., Пигаревский П.В., Тотолян Артем А. Роль стрептокиназы в моделировании постстрептококкового гломерулонефрита // Инфекция и иммунитет, 2021. Т. 11, № 5. С. 853-864. doi:10.15789/2220-7619-ARO-1594.; Бурова Л.А., Пигаревский П.В., Снегова В.А., Тотолян А.А. 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PloS One, 2014, Vol. 9, no. 2, e89707. doi:10.1371/journal.pone.0089707; Nilson B.H., Frick I.M., Akesson P., Forsen S., Bjorck L., Akerstrom B., Wikström M. Structure and stability of protein H and the M1 protein from Streptococcus pyogenes. Implications for other surface proteins of grampositive bacteria. Biochemistry, 1995, Vol. 34, no. 41, pp. 13688-13698.; Nordstrand A., McShan W.M., Ferretti J.J., Holm S. E., Norgren M. Allele substitution of the streptokinase gene reduces the nephritogenic capacity of Group A streptococcal strain NZ131. Infect. Immun., 2000, Vol. 68, no. 3, pp. 1019-1025.; Nordstrand A., Norgren M., Ferretti J.J., Holm S.E. Streptokinase as a mediator of acute post-streptococcal glomerulonephritis in an experimental mouse model. Infect. Immun., 1998, Vol. 66, no. 1, pp. 315-321.; Nordstrand A., Norgren M., Holm S.E. An experimental model for acute glomerulonephritis in mice. 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Immunol., 2014, Vol. 193, no. 1, pp. 317-326.; Schmitt R., Carlsson F., Mörgelin M., Tati R., Lindahl G., Karpman D. Tissue deposits of IgA-binding streptococcal M proteins in IgA nephropathy and Henoch-Schonlein purpura. Am. J. Pathol., 2010, Vol. 176, no. 2, pp. 608-618.; Schroder A.K., Nardella F.A., Mannik M., Johansson P.J., Christensen P. Identification of the site on IgG Fc for interaction with streptococci of groups A, C end G. Immunology, 1987, Vol. 62, no. 4, pp. 523-527.; Stenberg L., O’Toole P., Lindahl G. Many group A streptococcal strains express two different immunoglobulinbinding proteins, encoded by closely linked genes: characterization of the proteins expressed by four strains of different M-type. Mol. Microbiol., 1992, Vol. 6, no. 9, pp. 1185-1194.; Sun H., Ringdahl U., Homeister J.W., Fay W.P., Engleberg N.C., Yang A.Y., Rozek L.S., Wang X., Sjöbring U., Ginsburg D. Plasminogen is a critical host pathogenicity factor for group A streptococcal infection. Science, 2004, Vol. 305, no. 5688, pp. 1283-1286.; Suzuki H., Suzuki Y., Novak J., Tomino Y. Development of animal models of human IgA Nephropathy. Drug Discov. Today Dis. Models, 2014, no. 11, pp. 5-11.; Suzuki H., Kiryluk K., Novak J., Moldoveanu Z., Herr A.B., Renfrow M.B., Wyatt R.J., Scolari F., Mestecky J., Gharavi A.G., Bruce A.J. The Pathophysiology of IgA Nephropathy. J. Am. Soc. Nephrol., 2011, Vol. 22, pp. 1795-1803.; Suzuki H., Fan R., Zhang Z., Brown R., Hall S., Bruce A.J., Chatham W.W., Suzuki Y., Wyatt R.J., Moldoveanu Z., Lee J.Y., Robinson J., Tomana M., Tomino Y., Mestecky J., Novak J. Aberrantly glycosylated IgA1 in IgA nephropathy patients is recognized by IgG antibodies with restricted heterogeneity. J. Clin. Invest., 2009, Vol. 119, no. 6, pp. 1668-1677.; Swanson J., Hsu K.C., Gotschlich E.C. Electron microscopic studies on streptococci. I. M antigen. J. Exp. Med., 1969, Vol. 130, no. 5, pp. 1063-1091.; Tanaka M., Seki G., Someya T., Nagata M., Fujita T. 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3Academic Journal
Συγγραφείς: Наталья Яковлевна Дзеранова, Владимир Анатольевич Исаков, Юрий Романович Ковалев, Дмитрий Дмитриевич Зотов
Πηγή: University Therapeutic Journal, Vol 1, Iss 1 (2020)
Θεματικοί όροι: гломерулонефрит, псориаз, псориатический артрит, псориатическая нефропатия, IgA нефропатия, Medicine
Περιγραφή αρχείου: electronic resource
Relation: http://ojs3.gpmu.org/index.php/Un-ther-journal/article/view/2427; https://doaj.org/toc/2713-1912; https://doaj.org/toc/2713-1920
Σύνδεσμος πρόσβασης: https://doaj.org/article/963da030b1204243bea25bc2965b52be
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4Academic Journal
Συγγραφείς: Юрий Романович Ковалев, Дмитрий Дмитриевич Зотов
Πηγή: University Therapeutic Journal, Vol 1, Iss 1 (2020)
Θεματικοί όροι: гломерулонефрит, псориаз, псориатический артрит, псориатическая нефропатия, IgA нефропатия, Medicine
Relation: http://ojs3.gpmu.org/index.php/Un-ther-journal/article/view/2427; https://doaj.org/toc/2713-1912; https://doaj.org/toc/2713-1920; https://doaj.org/article/963da030b1204243bea25bc2965b52be
Διαθεσιμότητα: https://doaj.org/article/963da030b1204243bea25bc2965b52be
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5Academic Journal
Πηγή: University Therapeutic Journal, Vol 1, Iss 1 (2020)
Θεματικοί όροι: псориатический артрит, IgA нефропатия, Medicine, псориаз, гломерулонефрит, псориатическая нефропатия
Σύνδεσμος πρόσβασης: https://doaj.org/article/963da030b1204243bea25bc2965b52be
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6Academic Journal
Συγγραφείς: S.N. Mamaeva, G.V. Maksimov, A.S. Golderova, Ya.A. Munkhalova, A.N. Pavlov, A.L. Fedorov
Πηγή: Yakut Medical Journal. :33-35
Θεματικοί όροι: хронический гломерулонефрит, hematuria, chronic glomerulonephritis, гематурия, ИК-спектроскопия, IgA-nephropathy, IgA-нефропатия, acute glomerulonephritis, infrared spectroscopy, 3. Good health, острый гломерулонефрит
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7Academic Journal
Συγγραφείς: E. V. Ilyinykh, M. S. Eliseev, Е. В. Ильиных, М. С. Елисеев
Πηγή: Modern Rheumatology Journal; Том 13, № 3 (2019); 71-75 ; Современная ревматология; Том 13, № 3 (2019); 71-75 ; 2310-158X ; 1996-7012 ; 10.14412/1996-7012-2019-3
Θεματικοί όροι: аллопуринол, canakinumab, arthritis, uric acid, IgA nephropathy, chronic kidney disease, allopurinol, канакинумаб, артрит, мочевая кислота, IgA-нефропатия, хроническая болезнь почек
Περιγραφή αρχείου: application/pdf
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Научно-практическая ревматология. 2018;56(6):760-6. doi:10.14412/1995-4484-2018-760-766; Schlesinger N, Mysler E, Lin HY, et al. Canakinumab reduces the risk of acute gouty arthritis flares during initiation of allopurinol treatment: results of a double-blind, randomised study. Ann Rheum Dis. 2011 Jul; 70(7):1264-71. doi:10.1136/ard.2010.144063. Epub 2011 May 3.; Елисеев МС, Желябина ОВ, Мукагова МВ, Насонов ЕЛ. Клинический опыт применения блокатора интерлейкина 1 в канакинумаба у больных хронической тофусной подагрой: купирование артрита и профилактика обострений при назначении аллопуринола. Современная ревматология. 2015;9(2):16-22. doi: 1996-7012-2015-2-16-22; Akahoshi T, Murakami Y, Kitasato H. Recent advances in crystal-induced acute inflammation. Curr Opin Rheumatol. 2007 Mar;19(2):146-50.; Martinon F, P?trilli V, Mayor A, et al. Gout-associated uric acid crystals activate the NALP3 inflammasome. Nature. 2006 Mar 9;440(7081):237-41. Epub 2006 Jan 11.; So A, Meulemeester De, Pikhlak A, et al. Canakinumab for the treatment of acute flares in difficult-to-treat gouty arthritis: results of a multicenter, phase II, dose-ranging study. Arthritis Rheum. 2010 Oct;62(10): 3064-76. doi:10.1002/art.27600.; Schlesinger N, Alten R, Bardin T, et al. Canakinumab in frequently flaring gouty arthritis patients, contraindicated, intolerant or unresponsive to non-steroidal anti-inflammatory drugs and/or colchicine: safety and efficacy results from long term follow-up. Arthritis Rheum. 2014;66:S66—S67.; Bardin T, So A, Alten R, et al. Efficacy and safety of canakinumab vs triamcinolone acetonide in patients with gouty arthritis unable to use nonsteroidal anti-inflammatory drugs and colchicine, and on stable urate lowering therapy (ULT) or unable to use ULT. Arthritis Rheum. 2012;64:S811—S812.; Schlesinger N, De Meulemeester M, Pikhlak A, et al. Canakinumab relieves symptoms of acute flares and improves health-related quality of life in patients with difficult-to-treat gouty arthritis by suppressing inflammation: results of a randomized, dose-ranging study. Arthritis Res Ther. 2011 Mar 25;13(2):R53. doi:10.1186/ar3297; Елисеев МС, Желябина ОВ, Барскова ВГ, Насонов ЕЛ. Опыт применения ингибитора интерлейкина 1 в канакинумаба у больного с хронической тофусной подагрой. Научно-практическая ревматология. 2014;52(1):99—101. doi:10.14412/1995-4484-2014-99-101; Schlesinger N, Alten RE, Bardin T, et al. Canakinumab for acute gouty arthritis in patients with limited treatment options: results from two randomised, multicentre, active-controlled, double-blind trials and their initial extensions. Ann Rheum Dis. 2012 Nov;71(11):1839-48. doi:10.1136/annrheumdis-2011-200908. Epub 2012 May 14.; Lowry F. FDA panel says no to canakinumab for gout attacks. 2011. www.medscape.com/viewarticle/745076; Vianna HR, Soares CM, Tavares MS, et al. Inflammation in chronic kidney disease: the role of cytokines. J Bras Nefrol. 2011 Jul-Sep;33(3):351-64.; Vilaysane A, Chun J, Seamone ME, et al. The NLRP3 inflammasome promotes renal inflammation and contributes to CKD. J Am Soc Nephrol. 2010 Oct;21(10):1732-44. doi:10.1681/ASN.2010020143. Epub 2010 Aug 5.; Fessel WJ. Renal outcomes of gout and hyperuricemia. Am J Med. 1979 Jul;67(1):74-82.; Roughley MJ, Belcher J, Mallen CD, Roddy E. Gout and risk of chronic kidney disease and nephrolithiasis: meta-analysis of observational studies. Arthritis Res Ther. 2015 Apr 1;17:90. doi:10.1186/s13075-015-0610-9.; Singh JA, Yu S. Are allopurinol dose and duration of use nephroprotective in the elderly? A Medicare claims study of allopurinol use and incident renal failure. Ann Rheum Dis. 2017 Jan;76(1):133-139. doi:10.1136/annrheumdis-2015-209046. Epub 2016 Jun 13.; Levy GD, Rashid N, Niu F, Cheetham TC. Effect of urate-lowering therapies on renal disease progression in patients with hyperuricemia. J Rheumatol. 2014 May;41(5):955-62. doi:10.3899/jrheum.131159. Epub 2014 Apr 1.; Bakan A, Oral A, Elcioglu OC, et al. Hyperuricemia is associated with progression of IgA nephropathy. Int Urol Nephrol. 2015 Apr;47(4):673-8.; Caliskan Y, Ozluk Y, Celik D, et al. The Clinical Significance of Uric Acid and Complement Activation in the Progression of IgA Nephropathy. Kidney Blood Press Res. 2016;41(2):148-57. doi:10.1159/000443415. Epub 2016 Feb 26.; Shi Y, Chen W, Jalal D, et al. Clinical outcome of hyperuricemia in IgA nephropa-thy: a retrospective cohort study and random-ized controlled trial. Kidney Blood Press Res. 2012;35(3):153-60. doi:10.1159/000331453. Epub 2011 Nov 23.; Le W, Liang S, Hu Y, et al. Long-term renal survival and related risk factors in patients with IgA nephropathy: results from a cohort of 1155 cases in a Chinese adult population. Nephrol Dial Transplant. 2012 Apr;27(4):1479-85. doi:10.1093/ndt/gfr527. Epub 2011 Sep 29.; Nasri H, Baradaran A. Aggravation of immunoglobulin a nephropathy by hyperuricemia: a mini-review on current findings and new concepts. Iran J Kidney Dis. 2014 Sep;8(5):359-62.
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8Academic Journal
Συγγραφείς: L. A. Burova, P. V. Pigarevsky, V. A. Snegova, N. V. Duplik, Klaas Schalen, Artem A. Totolian, Л. А. Бурова, П. В. Пигаревский, В. А. Снегова, Н. В. Дуплик, Клаас Шален, Артем А. Тотолян
Πηγή: Medical Immunology (Russia); Том 18, № 3 (2016); 221-230 ; Медицинская иммунология; Том 18, № 3 (2016); 221-230 ; 2313-741X ; 1563-0625 ; 10.15789/1563-0625-2016-3
Θεματικοί όροι: экспериментальная модель, IgA-binding streptococci, IgA nephropathy, experimental model, стрептококковая IgAFc-связывающая активность, IgA-нефропатия
Περιγραφή αρχείου: application/pdf
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Aujourd’hui, 2013, Vol. 207, no. 4, pp. 241-247.; Boyd J.K., Cheung C.K, Molyneux K., Feehally J., Barratt J. An update on the pathogenesis and treatment of IgA nephropathy. Kidney International, 2012, Vol. 81, pp. 833-843.; Burova L., Thern A., Pigarevsky P., Seliverstova V., Gavrilova E., Nagornev V., Schalen C., Totolian A. Role of group A streptococcal IgG-binding protein in triggering experimental glomerulonephritis in the rabbit. APMIS, 2003, Vol. 111, pp. 955-962.; Burova L., Pigarevsky P., Seliverstova V., Gupalova T., Schalen C., Totolian A. Experimental poststreptococcal glomerulonephritis elicited by IgGFc-binding M family proteins and blocked by IgG Fc fragment. APMIS, 2012, Vol. 120, no. 3, pp. 221-230.; Collan Y. Morphometry in pathology: another look at diagnostic histopathology. Pathol. Res. Pract., 1984, Vol. 1, pp. 189-192.; Coppo R. The pathogenetic potential of environmental antigens in IgA nephropathy. Am. J. Kidney Dis., 1988, Vol. 12, no. 5, pp. 420-424.; Coppo R. The intestine-renal connection in IgA nephropathy. Nephrol. Dial. Transplant., 2015, Vol. 30, no. 3, pp. 360-366.; Hashimoto A., Suzuki Y., Suzuki H., Ohsawa I., Brown R., Hall S., Tanaka Y., Novak J., Ohi H., Tomino H. Determination of severity of murine IgA nephropathy by glomerular complement activation by aberrantly glycosylated IgA and immune complexes. Am. J. Pathol., 2012, Vol. 181, no. 4, pp. 1338-1347.; Jessen R.H., Emancipator S.N., Jacobs G.H., Nedrud J.G. Experimental IgA-IgG nephropathy induced by a viral respiratory pathogen. Dependence on antigen form and immune status. Lab. Invest., 1992, Vol. 67, no. 3, pp. 379-386.; Kovalenko P., Fujinaka H., Yoshida Y., Kawamura H., Qu Z., El-Shemi A., Li H., Matsuki A., Bilim V., Yaoita E., Abo T., Uchiyama M., Yamamoto T. Fc receptor-mediated accumulation of macrophages in crescentic glomerulonephritis induced by antiglomerular basement membrane antibody administration in WKY rats. International Immunology, 2004, Vol. 16, no. 5, pp. 625-634.; Liu H., Peng Y., Liu F., Xiao W., Zhang Y., Li W. Expression of IgA class switching gene in tonsillar mononuclear cells in patients with IgA nephropathy. Inflamm Res., 2011, Vol. 60, no. 9, pp. 869-878.; Meng H., Ohtake H., Ishida A., Ohta N., Kakehata S., Yamakawa M. IgA production and tonsillar focal infection in IgA nephropathy. J. Clin. Exp. Hematop., 2012, Vol. 52, no. 3, pp. 161-170.; Nakata J., Suzuki Y., Suzuki H., Sato D., Kano T., Yanagawa H., Matsuzaki K., Horikoshi S., Novak J., Tomino Y. Changes in nephritogenic serum galactose-deficient IgA1 in IgA nephropathy following tonsillectomy and steroid therapy. PloS One, 2014, Vol. 9, no. 2, e89707.; Okazaki K., Suzuki Y., Otsuji M., Suzuki H., Kihara M., Kajiyama T., Hashimoto A., Nishimura H., Brown R., Hall S., Novak J., Izui S., Hirose S., Tomino Y. Development of a model of early-onset IgA nephropathy. J. Am. Soc. Nephrol., 2012, Vol. 23, no. 8, pp. 1364-1374.; Piccolo M., De Angelis M., Lauriero G., Montemurno E., Di Cagno R., Gobbetti M. Salivary Microbiota associated with immunoglobulin A. Nephropathy. Microb. Ecol., 2015, Vol. 70, no. 2, pp. 557-565.; Schmitt R., Carlsson F., Mörgelin M., Tati R., Lindahl G., Karpman D. Tissue deposits of IgA-binding streptococcal M proteins in IgA nephropathy and Henoch-Schonlein purpura. Am. J. Pathol., 2010, Vol. 176, no. 2, pp. 608-618.; Schmitt R., Ståhl A., Olin A., Kristoffersson A.-C., Rebetz A., Novak J., Lindahl G., Karpman D. The combined role of galactose-deficient IgA1 and Streptococcal IgA-binding M protein in inducing IL-6 and C3 secretion from human mesangial cells: Implications for IgA nephropathy. J. Immunol., 2014, Vol. 193, no. 1, pp. 317-326.; Stenberg L., O’Toole P., Lindahl G. Many group A streptococcal strains express two different immunoglobulin-binding proteins, encoded by closely linked genes: characterization of the proteins expressed by four strains of different M-type. Molecular Microbiology, 1992, Vol. 6, no. 9, pp. 1185- 1194.; Suzuki H., Run Fan, Zhixin Zhang, Brown R., Hall S., Bruce A.J., Chatham W.W., Suzuki Y., Wyatt R.J.; Moldoveanu Z., Lee J.Y., Robinson J., Tomana M., Tomino Y., Mestecky J., Novak J. Aberrantly glycosylated IgA1 in IgA nephropathy patients is recognized by IgG antibodies with restricted heterogeneity. J. Clin. Invest., 2009, Vol. 119, no. 6, pp. 1668-1677.; Suzuki H., Kiryluk K., Novak J., Moldoveanu Z., Herr A.B., Renfrow M.B., Wyatt R.J., Scolari F., Mestecky J., Gharavi A.G., Bruce A.J. The pathophysiology of IgA nephropathy. J. Am. Soc. Nephrol., 2011, Vol. 22, pp. 1795-1803.; Suzuki H., Suzuki Y., Novak J., Tomino Y. Development of animal models of human IgA nephropathy. Drug Discov. Today Dis. Models, 2014, Vol. 11, pp. 5-11.; Tanaka M., Seki G., Someya T., Nagata M., Fujita T. Aberrantly glycosylated IgA1 as a factor in the pathogenesis of IgA nephropathy. Clin. Dev. Immunol., 2011, Vol. 2011, ID 470803.; Tian J., Wang Y., Zhou X., Li Y., Wang C., Li J., Li R. Rapamycin slows IgA nephropathy progression in the rat. Am. J. Nephrol., 2014, Vol 39. no. 3, pp. 218-229.; Tomana M., Matousovic K., Julian B.A., Radl J., Konecny K., Mestecky J. Galactose-deficient IgA1 in sera of IgA nephropathy patients is present in complexes with IgG. Kidney Int., 1997, Vol. 52, no. 2, pp. 509-516.; Wyatt R.J., Bruce A.J. IgA Nephropathy. N. Engl. J. Med., 2013, Vol. 368, pp. 202-214.; https://www.mimmun.ru/mimmun/article/view/1034
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9Academic Journal
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10Academic Journal
Συγγραφείς: Сиповский, В., Добронравов, В., Корунная, А., Смирнов, А.
Θεματικοί όροι: IGA-НЕФРОПАТИЯ, ЛЕКТИНОВЫЙ ПУТЬ АКТИВАЦИИ КОМПЛЕМЕНТА, ТУБУЛОИНТЕРСТИЦИЙ
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Διαθεσιμότητα: http://cyberleninka.ru/article/n/kliniko-morfologicheskiy-analiz-izmeneniy-peritubulyarnyh-mikrososudov-interstitsiya-pochek-u-bolnyh-s-iga-nefropatiey-igan-s-otsenkoy
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11Academic Journal
Συγγραφείς: Наушабаева, А., Абеуова, Б., Чингаева, Г., Кабулбаев, К.
Θεματικοί όροι: БЫСТРОПРОГРЕССИРУЮЩИЙ ГЛОМЕРУЛОНЕФРИТ, ЭКСТРАКАПИЛЛАРНЫЙ ГЛОМЕРУЛОНЕФРИТ, IGA-НЕФРОПАТИЯ, НЕФРИТ ШЕНЛЕЙН-ГЕНОХА, ПУЛЬС-ТЕРАПИЯ КОРТИКОСТЕРОИДАМИ, ПУЛЬС-ТЕРАПИЯ ЦИКЛОФОСФАМИДОМ, МОФЕТИЛА МИКОФЕНОЛАТ, 5-ЛЕТНЯЯ ВЫЖИВАЕМОСТЬ
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12Academic Journal
RENAL INVOLVEMENT IN CHILDREN WITH CELIAC DISEASE ; ОСОБЕННОСТИ СОСТОЯНИЯ ПОЧЕК ПРИ ЦЕЛИАКИИ У ДЕТЕЙ
Συγγραφείς: E. A. Trifonova, N. V. Rusakova, Е. А. Трифонова, Н. В. Русакова
Πηγή: Current Pediatrics; Том 11, № 5 (2012); 45-49 ; Вопросы современной педиатрии; Том 11, № 5 (2012); 45-49 ; 1682-5535 ; 1682-5527
Θεματικοί όροι: IgA-нефропатия, kidneys, celiac disease, dysmetabolic nephropathy, IgA-nephropathy, почки, целиакия, дисметаболическая нефропатия
Περιγραφή αρχείου: application/pdf
Relation: https://vsp.spr-journal.ru/jour/article/view/426/331; Полачек Э. Нефрология детского возраста. Прага: Авиценум. 1980. 330 с.; Волкова О. В., Елецкий Ю. К. Гистология, цитология и эмбриология. М. 1996. 544 с.; Collin P., Syrjanen J., Partanen J. et al. Celiac disease and HLADQ in patients with IgA nephropathy. Am. J. Gastroenterol. 2002; 97: 2572–2576.; Фадеева М. А., Лебедев В. П., Вербицкий В. И. и др. Патология мочевой системы при синдроме нарушенного кишечного всасывания. В кн.: Синдром нарушенного кишечного всасывания у детей. М. 1985. С. 53–55.; Бельмер С. В., Ревнова М. О. Целиакия у детей. М.: Медпрактика-М. 2010. 391 с.; Малкоч А. В. Дисметаболические нефропатии и мочекаменная болезнь. В кн.: Нефрология детского возраста: Практическое руководство по детским болезням. М.: Медпрактика-М. 2005. С. 472–516.; Фрейтаг Д., Хрустка К. Патофизиология нефролитиаза. В кн.: Почки и гомеостаз в норме и при патологии. Под ред. С. Клара: Пер. с англ. М. 1987. С. 390–419.; Возианов А. Ф., Майданник В. Г., Бидный В. Г., Багдасарова И. В. Основы нефрологии детского возраста. Киев: Книга-плюс. 2002. С. 214–225.; Eversmann J. J., Farmer R. G., Brown C. H. et al. Gastrointestinal manifestations of Hyperparathyroidism. Arch. Intern. Med. 1967; 119: 605.; Saunders D. R., Sillery J., McDonald G. B. Regional differences in oxalate absorption by rat intestine: evidence for excessive absorption by the colon in steatorrhea. Gut. 1975; 16: 543.; Stauffer J. Q. Hyperoxaluria and calcium oxalate nephrolithiasis after jejunoileal bypass. Am. J. Clin. Nutr. 1977; 30: 64–71.; Binder H. J. Intestinal oxalate absorption. Gastroenterology. 1974; 67: 441.; Dobbins J. W., Binder H. J. Effect of bile salts and fatty acids on the colonic absorption of oxalate. Gastroenterology. 1976; 70: 1096.; Massey L. K. Dietary influences on urinary oxalate and risk of kidney stones. Front Biosci. 2003; 8: 584–594.; Mittal R. D., Kumar R. Gut-inhabiting bacterium Oxalobacter formigenes: role in calcium oxalate urolithiasis. J. Endourol. 2004; 18 (5): 418–424.; Kwak C., Kim H. K., Kim E. C. et al. Urinary oxalate levels and the Urolithiasis. Eur. Urol. 2003; 44 (4): 475–481.; Troxel S. A., Sidhu H., Kaul P., Low R. K. Intestinal Oxalobacter formigenes colonization in calcium oxalate stone formers and its relation to urinary oxalate. J. Endourol. 2003; 17 (3): 173–176. 18. Weese J. S., Weese H. E., Yuricek L., Rousseau J. Oxalate degradation by intestinal lactic acid bacteria in dogs and cats. Vet. Microbiol. 2004; 101 (3): 161–166.; Коровина Н. А., Захарова И. Н., Гаврюшова Л. П., Мумладзе Э. Б. и др. Дисметаболические нефропатии у детей: диагностика и лечение. Рук-во для врачей. М.: Медпрактика-М. 2007. С. 5–6.; Laurent J., Branellec A., Heslan J. M. et al. An increase in circulating Ig A antibodies to gliadin in IgA mesangial gbomerulonephritis. Am. J. Nephrol. 1987; 7: 178–183.; Nagy J., Scott H., Brandtzaeg P. Antibodies to dietary antigens in IgA nephropathy. Clin. Nephrol. 1988; 29: 275–279.; Fornasieri A., Sinico P. A., Maldifassi P. et al. IgA-antigliadin antibodies in IgA mesangial nephropathy (Berger’s disease). Br. Med. J. 1988; 295: 78–80.; Rodriguez-Soriano J., Arrieta A., Vallo A. et al. IgA-antigliadin anti bodies in children with IgA mesangial glomerulonephritis. Lancet. 1988; 1: 1109–1110.; Колина И. Б. Болезнь Берже. Лечащий врач. 2011; 8: 24–27.; Haddad E., Moura I. С., Arcos-Fajardo M. et al. Enhanced expres sion of the CD71 mesangial IgAl receptor in Berger disease and Henoch-Schnnlein nephritis: association between CD71 expression and IgA deposits. J. Am. Soc. Nephrol. 2003; 14: 327–337.; Lai K. N., Chan L. Y. Y., Tang S. C. W. et al. Characteristics of polymeric R-IgA binding to leukocytes in IgA nephropathy. J. Am. Soc. Nephrol. 2002; 13: 2309–2319.; Barratt J., Feehally J., Smith A. C. Patogenesis of IgA nephropathy. Seminar in Nephrology. 2004; 24 (3): 197–217.; Coppo R., Amore A., Roccatelbo D. et al. IgA antibodies to dietary antigens and bectinbinding Ig A in sera from Italian, Australian and Japanese Ig A nephropathy patients. Am. J. Kidney Dis. 1991; 17: 480–487.; Coppo R., Amore A., Roccatelbo D. Dietary antigens and primary immunoglobulin A nephropathy. J. Am. Soc. Nephrol. 1992; 2: 173–180.; Kondoh H., Kobayashi K., Kajii T. Jacalin, a jackfruit lectin, precipitates IgA1 but not IgA2 subclass on gel diffusion reaction. J. Immunol. Methods. 1986; 80: 171–173.; Stockert R. J., Kressner M. S., Collins J. C. IgA interaction with the asiabogbycoprotein receptor. Proc. Natl. Acad. Sci. USA. 1982; 79: 6229–6235.; Dav in J. C., Dechenne C., Lombet J. et al. Acute experimental glomerulonephritis induced by the glomerulan deposition of circulating polymeric IgA-concanavalin A complexes. Virchows Arch. 1989; 415: 7–20.; Douglas A. P. The binding of a glycopeptide component of wheat gluten to intestinal mucosa of normal and celiac human subjects. Clin. Chem. Acta. 1976; 73: 357–361.; Lorenzsonn V., Olsen W. A. In vivo responses of rat intestinal epithelium to intrabuminal dietary lectins. Gastroenterology. 1982; 82: 839–848.; Forsdyke D. R. Role of complement in the toxicity of the dietary legumes. Med. Hypotheses. 1978; 4: 97–100.; Amore A., Emancipator S. N., Coppo R. et al. Specific binding of gliadin to rat mesangial cell in culture. Nephrol. Dial. Transplant. 1988; 3: 524.
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13Academic Journal
Συγγραφείς: A. P. Potemkina, T. V. Margieva, O. V. Komarova, P. E. Povilaitite, L. V. Leonova, T. V. Vashurina, O. I. Zrobok, A. N. Tsygin, А. П. Потемкина, Т. В. Маргиева, О. В. Комарова, П. Е. Повилайтите, Л. В. Леонова, Т. В. Вашурина, О. И. Зробок, А. Н. Цыгин
Πηγή: Pediatric pharmacology; Том 9, № 3 (2012); 46-51 ; Педиатрическая фармакология; Том 9, № 3 (2012); 46-51 ; 2500-3089 ; 1727-5776
Θεματικοί όροι: дети, causes, diagnosis, glomerular disease, mutations of collagen type IV, Alport syndrome, thin basement membrane disease, IgA-nephropathy, children, диагностика, гломерулярные болезни, мутации коллагена IV типа, синдром Альпорта, болезнь тонких базальных мембран, IgA-нефропатия
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Relation: https://www.pedpharma.ru/jour/article/view/324/591; Борисов В. В., Вашурина Т. В., Вознесенская Т. С. и др. Нефрология: национальное руководство / под ред. Н. А. Мухина. М.: ГЭОТАР-Медиа. 2009. С. 112–118.; Лойманн Э., Цыгин А. Н., Саркисян А. А. Практическое руководство. Детская нефрология. Москва: Литтерра. 2010. С. 84–85, 172–178.; Cameron J. S. The patient with proteinuria and/or haematuria. In: Oxford textbook of Clinikal nephrology, 2-nd ed. Oxford University, Press London. 1998. Р. 441–455.; Wilcox C. S., Berl T., Himmelfarb J. et al. Therapy in nephrology and hypertension: a companion to Brenner and Rector's. The kidney. 2008 P. 503–507.; Комарова О. В., Маргиева Т. В., Цыгин А. Н. Синдром гематурии у детей. Вопросы диагностики в современной педиатрии. 2009; 1 (4): 28–31.; Комарова О. В. Синдром гематурии у детей. Медицинский научный и учебно-методический журнал. 2006; 30.; Geary D. F., Schaefer F. Comprehensive Pediatric Nephrology. Philadelphia: Mosby. 2008. Р. 179–184, 229–235.; Roth S., Renner E., Rathert P. Microscopic hematuria: advances in identification of glomerular dysmorphic erythrocytes. J Urol. 1991; 146 (3): 680–684.; Приходина Л. С., Малашина О. А. Современные представления о гематурии у детей. Нефрология и диализ. 2000; 3: 42–49.; Сергеева Т. В., Дмитриенко С. В., Цыгин А. Н. Синдром гематурии у детей. Качество жизни. 2006; 4 (15): 68–71.; Bohler W. J., Luft F. C. et al. Nephrologie. Pathologie-KlinikNierenersetzverfahren. Thieme-Stuttgart. 2008. Р. 12.; Zaman Z., Proesmans W. Dysmorphic erythrocytes and G1 cells as markers of glomerular hematuria. Pediatric. Nephrology. 2000; 14: 980–984.; Gubler M. C. Inherited diseases of the glomerular basement membrane. Nat Clin Pract Nephrol. 2008; 4 (1): 24–37.; Jedlicka J. N. Die interstitielle entzundung im rahmen des alport syndroms. Munchen-Pasing. 2011. Р. 74.; D'Amico G. Natural history of idiopathic IgA nephropathy: role of clinical and histological prognostic factors. Am J Kidney Dis. 2000; 36 (2): 227–237.; Tumlin J. A., Madaio M. P., Hennigar R. Idiopathic IgA nephropathy: pathogenesis, histopathology and therapeutic options. CJASN. 2007; 2 (5): 1054–1061.; Schroder C. H., Bontemps C. M., Assmann J. M. et al. Renal biopsy and family studies in 65 children with isolated hematuria. Acta Paediatrica. 1990; 79 (6–7): 630–636.; Игнатова М. С. Гематурия при наследственных нефропатиях. Нефрология и диализ. 2006; 4: 64–71.; Jais J., Knebelmann B., Giatras I. et al. X-linked alport syndrome: natural history and genotype-phenotype correlation in girls and women belonging to 195 families: A «European community alport syndrome concerted action» study. J Am Soc Nephrol. 2003; 14: 2603–2610.; https://www.pedpharma.ru/jour/article/view/324
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14Academic Journal
Συγγραφείς: Потемкина, Александра, Маргиева, Т., Комарова, О., Повилайтите, П., Леонова, Л., Вашурина, Т., Зробок, О., Цыгин, А.
Θεματικοί όροι: ГЕМАТУРИЯ, ПРИЧИНЫ, ДИАГНОСТИКА, ГЛОМЕРУЛЯРНЫЕ БОЛЕЗНИ, МУТАЦИИ КОЛЛАГЕНА IV ТИПА, СИНДРОМ АЛЬПОРТА, БОЛЕЗНЬ ТОНКИХ БАЗАЛЬНЫХ МЕМБРАН, IGA-НЕФРОПАТИЯ, ДЕТИ
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15Academic Journal
Συγγραφείς: Трифонова, Елена, Русакова, Н.
Θεματικοί όροι: ДЕТИ, ПОЧКИ, ЦЕЛИАКИЯ, ДИСМЕТАБОЛИЧЕСКАЯ НЕФРОПАТИЯ, IGA-НЕФРОПАТИЯ
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16Academic Journal
Συγγραφείς: Вознесенская, Татьяна, Сергеева, Тамара
Θεματικοί όροι: IGA-НЕФРОПАТИЯ, ХРОНИЧЕСКАЯ БОЛЕЗНЬ ПОЧЕК, ДЕТИ
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17Academic Journal
Συγγραφείς: Комиссаров, К., Дмитриева, М., Цвирко, Т., Ефремова, К., Пилотович, В.
Θεματικοί όροι: НЕФРОТИЧЕСКИЙ СИНДРОМ, АУТОСОМНО ДОМИНАНТНЫЙ ПОЛИКИСТОЗ ПОЧЕК, ГЛЮКОКОРТИКОИДЫ, ЦИТОСТАТИКИ, IGA-НЕФРОПАТИЯ, МЕМБРАНОЗНАЯ НЕФРОПАТИЯ
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18Academic Journal
Συγγραφείς: Рябова, Татьяна, Ракитянская, Ирина, Рябов, Сергей
Θεματικοί όροι: IGA-НЕФРОПАТИЯ, ЦИТОКИНЫ, БИОПСИЙНАЯ ТКАНЬ, ВОЗРАСТ
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19Academic Journal
Συγγραφείς: Рябов, Сергей, Ракитянская, Ирина, Рябова, Татьяна
Θεματικοί όροι: IGA-НЕФРОПАТИЯ, ЛИМФОИДНЫЙ ИНФИЛЬТРАТ, КЛИНИКА, ВОЗРАСТ
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Διαθεσιμότητα: http://cyberleninka.ru/article/n/vliyanie-limfoidnoy-infiltratsii-pochechnoy-tkani-bolnyh-iga-nefropatiey-na-kliniko-laboratornuyu-kartinu-zabolevaniya-s-uchetom
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20Academic Journal
Συγγραφείς: Рябова, Т. С., Ракитянская, И. А.
Θεματικοί όροι: медицина, внутренние болезни, нефрология, болезни почек, IgA-нефропатия, морфологические изменения, лимфоидный инфильтрат, пожилой возраст
Διαθεσιμότητα: http://dspace.bsu.edu.ru/handle/123456789/53819
