Εμφανίζονται 1 - 20 Αποτελέσματα από 1.343 για την αναζήτηση '"репродуктивное здоровье"', χρόνος αναζήτησης: 0,81δλ Περιορισμός αποτελεσμάτων
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    Academic Journal

    Πηγή: Obstetrics, Gynecology and Reproduction; Online First ; Акушерство, Гинекология и Репродукция; Online First ; 2500-3194 ; 2313-7347

    Περιγραφή αρχείου: application/pdf

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Sphingolipids as multifaceted mediators in ovarian cancer. Cell Signal. 2021;81:109949. https://doi.org/10.1016/j.cellsig.2021.109949.; Quinville B.M., Deschenes N.M., Ryckman A.E., Walia J.S. A comprehensive review: sphingolipid metabolism and implications of disruption in sphingolipid homeostasis. Int J Mol Sci. 2021;22(11):5793. https://doi.org/10.3390/ijms22115793.; Sukocheva O., Wadham C., Holmes A. et al. Estrogen transactivates EGFR via the sphingosine 1-phosphate receptor Edg-3: the role of sphingosine kinase-1. J Cell Biol. 2006;173(2):301–10. https://doi.org/10.1083/jcb.200506033.; Chou C.H., Chen M.J. The effect of steroid hormones on ovarian follicle development. Vitam Horm. 2018;107:155–75. https://doi.org/10.1016/bs.vh.2018.01.013.; Zeleznik O.A., Clish C.B., Kraft P. et al. Circulating lysophosphatidylcholines, phosphatidylcholines, ceramides, and sphingomyelins and ovarian cancer risk: a 23-year prospective study. 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Steroids. 2010;75(6):390–9. https://doi.org/10.1016/j.steroids.2010.01.020.; Roth Z. Symposium review: reduction in oocyte developmental competence by stress is associated with alterations in mitochondrial function. J Dairy Sci. 2018;101(4):3642–54. https://doi.org/10.3168/jds.2017-13389.; Протопопов В.А., Секунов А.В., Панов А.В., Брындина И.Г. Взаимосвязь сфинголипидных механизмов с окислительным стрессом и изменениями митохондрий при функциональной разгрузке постуральных мышц. Acta Biomedica Scientifica. 2024;9(2):228–42. https://doi.org/10.29413/ABS.2024-9.2.23.; Kujjo L.L., Perez G.I. Ceramide and mitochondrial function in aging oocytes: joggling a new hypothesis and old players. Reproduction. 2012;143(1):1–10. https://doi.org/10.1530/REP-11-0350.; Zigdon H., Kogot-Levin A., Park J.W. et al. Ablation of ceramide synthase 2 causes chronic oxidative stress due to disruption of the mitochondrial respiratory chain. 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Roles of sphingosine-1-phosphate in follicle development and oocyte maturation. Anim Res One Health. 2024;2(3):314–22. https://doi.org/10.1002/aro2.53.; Park J.Y., Su Y.Q., Ariga M. et al. EGF-like growth factors as mediators of LH action in the ovulatory follicle. Science. 2004;303(5658):682–4. https://doi.org/10.1126/science.1092463.; Yamanaka M., Shegogue D., Pei H. et al. Sphingosine kinase 1 (SPHK1) is induced by transforming growth factor-beta and mediates TIMP-1 up-regulation. J Biol Chem. 2004;279(52):53994–4001. https://doi.org/10.1074/jbc.M410144200.; Squecco R., Sassoli C., Nuti F. et al. Sphingosine 1-phosphate induces myoblast differentiation through Cx43 protein expression: a role for a gap junction-dependent and -independent function. Mol Biol Cell. 2006;17(11):4896–910. https://doi.org/10.1091/mbc.e06-03-0243.; Giepmans B.N., Verlaan I., Hengeveld T. et al. Gap junction protein connexin-43 interacts directly with microtubules. 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Absence of estrogen receptor-beta expression in metastatic ovarian cancer. Obstet Gynecol. 2000;96(3):417–21. https://doi.org/10.1016/s0029-7844(00)00917-0.; Jeon S.-Y., Hwang K.-A., Choi K.-C. Effect of steroid hormones, estrogen and progesterone, on epithelial mesenchymal transition in ovarian cancer development. J Steroid Biochem Mol Biol. 2016;158:1–8. https://doi.org/10.1016/j.jsbmb.2016.02.005.; Mungenast F., Thalhammer T. Estrogen biosynthesis and action in ovarian cancer. Front Endocrinol (Lausanne). 2014;5:192. https://doi.org/10.3389/fendo.2014.00192.; Giaccari C., Antonouli S., Anifandis G. et al. An update on physiopathological roles of Akt in the reprodAKTive mammalian ovary. Life (Basel). 2024;14(6):722. https://doi.org/10.3390/life14060722.; Yang Y., Lang P., Zhang X. et al. Molecular characterization of extracellular vesicles derived from follicular fluid of women with and without PCOS: integrating analysis of differential miRNAs and proteins reveals vital molecules involving in PCOS. J Assist Reprod Genet. 2023;40(3):537–52. https://doi.org/10.1007/s10815-023-02724-z.; Liu L., Yin T.L., Chen Y. et al. Follicular dynamics of glycerophospholipid and sphingolipid metabolisms in polycystic ovary syndrome patients. J Steroid Biochem Mol Biol. 2019;185:142–9. https://doi.org/10.1016/j.jsbmb.2018.08.008.; Shi Y., Zhao H., Shi Y. et al. Genome-wide association study identifies eight new risk loci for polycystic ovary syndrome. Nat Genet. 2012;44(9):1020–5. https://doi.org/10.1038/ng.2384.; Parasar P., Ozcan P., Terry K.L. Endometriosis: epidemiology, diagnosis and clinical management. Curr Obstet Gynecol Rep. 2017;6(1):34–41. https://doi.org/10.1007/s13669-017-0187-1.; Lee Y.H., Tan C.W., Venkatratnam A. et al. Dysregulated sphingolipid metabolism in endometriosis. J Clin Endocrinol Metab. 2014;99(10):E1913–21. https://doi.org/10.1210/jc.2014-1340.; Zhang Q., Duan J., Liu X., Guo S.W. Platelets drive smooth muscle metaplasia and fibrogenesis in endometriosis through epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation. Mol Cell Endocrinol. 2016;428:1–16. https://doi.org/10.1016/j.mce.2016.03.015.; Bernacchioni C., Capezzuoli T., Vannuzzi V. et al. Sphingosine 1-phosphate receptors are dysregulated in endometriosis: possible implication in transforming growth factor β-induced fibrosis. Fertil Steril. 2021;115(2):501–11. https://doi.org/10.1016/j.fertnstert.2020.08.012.; Turathum B., Gao E.M., Grataitong K. et al. Dysregulated sphingolipid metabolism and autophagy in granulosa cells of women with endometriosis. Front Endocrinol (Lausanne). 2022;13:906570. https://doi.org/10.3389/fendo.2022.906570.; Itami N., Shirasuna K., Kuwayama T., Iwata H. Palmitic acid induces ceramide accumulation, mitochondrial protein hyperacetylation, and mitochondrial dysfunction in porcine oocytes. Biol Reprod. 2018;98(5):644–53. https://doi.org/10.1093/biolre/ioy023.; Fucho R., Casals N., Serra D., Herrero L. Ceramides and mitochondrial fatty acid oxidation in obesity. FASEB J. 2017;31(4):1263–72. https://doi.org/10.1096/fj.201601156R.; Torretta E., Barbacini P., Al-Daghri N.M., Gelfi C. Sphingolipids in obesity and correlated co-morbidities: the contribution of gender, age and environment. Int J Mol Sci. 2019;20(23):5901. https://doi.org/10.3390/ijms20235901.; Samad F., Hester K.D., Yang G. et al. Altered adipose and plasma sphingolipid metabolism in obesity: a potential mechanism for cardiovascular and metabolic risk. Diabetes. 2006;55(9):2579–87. https://doi.org/10.2337/db06-0330.; Shibahara H., Ishiguro A., Inoue Y. et al. Mechanism of palmitic acid-induced deterioration of in vitro development of porcine oocytes and granulosa cells. 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Компаративная оценка провоспалительных цитокинов у женщин с диагностированными наследственными тромбофилиями различного генеза и их ассоциация с ранними и поздними эмбриональными потерями. Проблемы репродукции. 2022;28(3):10–7. https://doi.org/10.17116/repro20222803110.; Cianci A., Calogero A.E., Palumbo M.A. et al. Relationship between tumour necrosis factor alpha and sex steroid concentrations in the follicular fluid of women with immunological infertility. Hum Reprod. 1996;11(2):265–8. https://doi.org/10.1093/humrep/11.2.265.; Banaras S., Paracha R.Z., Nisar M. et al. System level modeling and analysis of TNF-α mediated sphingolipid signaling pathway in neurological disorders for the prediction of therapeutic targets. Front Physiol. 2022;13:872421. https://doi.org/10.3389/fphys.2022.872421.; Sukocheva O.A., Neganova M.E., Aleksandrova Y/ et al. 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    Academic Journal

    Συνεισφορές: The authors declare no funding, Авторы заявляют об отсутствии финансовой поддержки

    Πηγή: Obstetrics, Gynecology and Reproduction; Vol 19, No 5 (2025); 626-631 ; Акушерство, Гинекология и Репродукция; Vol 19, No 5 (2025); 626-631 ; 2500-3194 ; 2313-7347

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