-
1Academic Journal
Συγγραφείς: N. V. Maychuk, A. A. Naumtseva, N. Sh. Sarhadov, Н. В. Майчук, A. А. Наумцева, Н. Ш. Сархадов
Συνεισφορές: Тhe article was prepared with the support of Bausch Health LLC. The position of the authors of the article may differ from the position of Bausch Health LLC, Статья подготовлена при поддержке компании ООО «Бауш Хелс». Позиция авторов статьи может отличаться от позиции компании ООО «Бауш Хелс»
Πηγή: Ophthalmology in Russia; Том 21, № 4 (2024); 793-801 ; Офтальмология; Том 21, № 4 (2024); 793-801 ; 2500-0845 ; 1816-5095 ; 10.18008/1816-5095-2024-4
Θεματικοί όροι: роговица, vitamin C, epithelium, regenerative regeneration, deficiency, proinflammatory factors, cornea, corneal surgery, витамин С, эпителий, репаративная регенерация, дефицит, провоспалительные факторы
Περιγραφή αρχείου: application/pdf
Relation: https://www.ophthalmojournal.com/opht/article/view/2505/1280; MollerPedersen T, Cavanagh HD, Petroll WM, Jester JV. Stromal wound healing explains refractive instability and haze development after photorefractive keratectomy: a 1year confocal microscopic study. Ophthalmology. 2000 Jul;107(7):1235– 1245. doi:10.1016/s0161-6420(00)00142-1. PMID: 10889092.; Федоров АА, Куренков ВВ, Каспаров АА, Полунин ГС. Особенности регенераторных процессов в роговице после фоторефракционной кератэктомии. Тезисы докладов 7 съезда офтальмологов России. 2000 ч 2:49.; Румянцева ОА, Ухина ТВ. Изучение патогенеза гиперплазии эпителия и регресса рефракции после фоторефракционной хирургии. Клиническая офтальмология. 2001;1(4):101–104.; Куренков ВВ. Эксимерлазерная хирургия роговицы. М.: Медицина, 1998:134–138. Kurenkov VV. Excimerlaser surgery of the cornea. Moscow, Medicine, 1998:134– 138 (In Russ.).; Ramachandran GN, Bansal M, Bhatnagar RS. A hypothesis on the role of hydroxyproline in stabilizing collagen structure. Biochim Biophys Acta. 1973 Sep 21;322(1):166–171. doi:10.1016/0005-2795(73)90187-6.; Lambiase A, Manni L, Bonini S, Rama P, Micera A, Aloe L. Nerve growth factor promotes corneal healing: structural, biochemical, and molecular analyses of rat and human corneas. Invest Ophthalmol Vis Sci. 2000 Apr;41(5):1063–1069.; Amayem A, AliAT, Waring GO 3rd, Ibrahim O. Bacterial keratitis after photorefractive keratectomy. J Refract Surg. 1996 JulAug;12(5):642–644. doi:10.3928/1081-597X-19960701-19.; Hovanesian JA, Shah SS, Maloney RK. Symptoms of dry eye and recurrent erosion syndrome after refractive surgery. J Cataract Refract Surg. 2001 Apr;27(4):577–584. doi:10.1016/s0886-3350(00)00835-x.; Cordeiro MF, Bhattacharya SS, Schultz GS, Khaw PT. TGFbeta1, beta2, and beta3 in vitro: biphasic effects on Tenon’s fibroblast contraction, proliferation, and migration. Invest Ophthalmol Vis Sci. 2000 Mar;41(3):756–763.; Werner S, Grose R. Regulation of wound healing by growth factors and cytokines. Physiol Rev. 2003 Jul;83(3):835–870. doi:10.1152/physrev.2003.83.3.835.; Di Girolamo N, Bobba S, Raviraj V, Delic NC, Slapetova I, Nicovich PR, Halliday GM, Wakefield D, Whan R, Lyons JG. Tracing the fate of limbal epithelial progenitor cells in the murine cornea. Stem Cells. 2015 Jan;33(1):157–169. doi:10.1002/stem.1769.; Brunette I, Gresset J, Boivin JF, Pop M, Thompson P, Lafond GP, Makni H. Functional outcome and satisfaction after photorefractive keratectomy. Part 2: survey of 690 patients. Ophthalmology. 2000 Sep;107(9):1790–1796. doi:10.1016/s0161-6420(00)002670.; Stojanovic A, Nitter TA. Correlation between ultraviolet radiation level and the incidence of lateonset corneal haze after photorefractive keratectomy. J Cataract Refract Surg. 2001 Mar;27(3):404–410. doi:10.1016/s0886-3350(00)00742-2.; Patel S, Maheshwari A, Chandra A. Biomarkers for wound healing and their evaluation. J Wound Care. 2016 Jan;25(1):46–55. doi:10.12968/jowc.2016.25.1.46.; Kim JH, Kim MS, Hahn TW, Lee YC, Sah WJ, Park CK. Five years results of photorefractive keratectomy for myopia. J Cataract Refract Surg. 1997 Jun;23(5):731– 735. doi:10.1016/s0886-3350(97)80282-9.; Naidu KA. Vitamin C in human health and disease is still a mystery? An overview. Nutr J. 2003 Aug 21;2:7. doi:10.1186/1475-2891-2-7.; Mohammed BM, Fisher BJ, Huynh QK, Wijesinghe DS, Chalfant CE, Brophy DF, Fowler AA 3rd, Natarajan R. Resolution of sterile inflammation: role for vitamin C. Mediators Inflamm. 2014;2014:173403. doi:10.1155/2014/173403.; Savini I, Rossi A, Pierro C, Avigliano L, Catani MV. SVCT1 and SVCT2: key proteins for vitamin C uptake. Amino Acids. 2008 Apr;34(3):347–355. doi:10.1007/s00726-007-0555-7.; Khurana V, Vadlapudi AD, Vadlapatla RK, Pal D, Mitra AK. Functional characterization and molecular identification of vitamin C transporter (SVCT2) in human corneal epithelial (HCEC) and retinal pigment epithelial (D407) cells. Curr Eye Res. 2015 May;40(5):457-69. doi:10.3109/02713683.2014.93544320.; Tsukaguchi H, Tokui T, Mackenzie B, Berger UV, Chen XZ, Wang Y, Brubaker RF, Hediger MA. A family of mammalian Na+dependent Lascorbic acid transporters. Nature. 1999 May 6;399(6731):70–75. doi:10.1038/19986.; Taniguchi M, Arai N, Kohno K, Ushio S, Fukuda S. Antioxidative and antiaging activities of 2OαglucopyranosylLascorbic acid on human dermal fibroblasts. Eur J Pharmacol. 2012 Jan 15;674(2-3):126–131. doi:10.1016/j.ejphar.2011.11.013.; Gonzalez G, Sasamoto Y, Ksander BR, Frank MH, Frank NY. Limbal stem cells: identity, developmental origin, and therapeutic potential. Wiley Interdiscip Rev Dev Biol. 2018 Mar;7(2):10.1002/wdev.303. doi:10.1002/wdev.303.; Kim JE, Jin DH, Lee SD, Hong SW, Shin JS, Lee SK, Jung DJ, Kang JS, Lee WJ. Vitamin C inhibits p53induced replicative senescence through suppression of ROS production and p38 MAPK activity. Int J Mol Med. 2008 Nov;22(5):651–655.; Moores J. Vitamin C: a wound healing perspective. Br J Community Nurs. 2013 Dec;Suppl:S6, S8–11. doi:10.12968/bjcn.2013.18.sup12.s6.; Brubaker RF, Bourne WM, Bachman LA, McLaren JW. Ascorbic acid content of human corneal epithelium. Invest Ophthalmol Vis Sci. 2000 Jun;41(7):1681–1683.; Diegelmann RF, Evans MC. Wound healing: an overview of acute, fibrotic and delayed healing. Front Biosci. 2004 Jan 1;9:283–289. doi:10.2741/1184. .; Mohammed BM, Fisher BJ, Kraskauskas D, Ward S, Wayne JS, Brophy DF, Fowler AA 3rd, Yager DR, Natarajan R. Vitamin C promotes wound healing through novel pleiotropic mechanisms. Int Wound J. 2016 Aug;13(4):572–584. doi:10.1111/iwj.12484.; Chen J, Lan J, Liu D, Backman LJ, Zhang W, Zhou Q, Danielson P. Ascorbic Acid Promotes the Stemness of Corneal Epithelial Stem/Progenitor Cells and Accelerates Epithelial Wound Healing in the Cornea. Stem Cells Transl Med. 2017 May;6(5):1356–1365. doi:10.1002/sctm.16-0441.; Csorba A, Katona G, BudaiSzűcs M, BaloghWeiser D, Fadda AM, Caddeo C, Takács ÁI, Mátyus P, Balogh GT, Nagy ZZ. Effect of liposomal formulation of ascorbic acid on corneal permeability. Sci Rep. 2023 Mar 1;13(1):3448. doi:10.1038/s41598-023-29290-9.; Bilgihan A, Bilgihan K, Toklu Y, Konuk O, Yis O, Hasanreisoğlu B. Ascorbic acid levels in human tears after photorefractive keratectomy, transepithelial photorefractive keratectomy, and laser in situ keratomileusis. J Cataract Refract Surg. 2001 Apr;27(4):585–588. doi:10.1016/s0886-3350(00)00877-4.; Li M, Chen Z, Liu L, Ma X, Zou J. Topical Vitamin C Promotes the Recovery of Corneal Alkali Burns in Mice. J Ophthalmol. 2021 Dec 23;2021:2406646. doi:10.1155/2021/2406646.; Hayes S, Cafaro TA, Boguslawska PJ, KammaLorger CS, Boote C, Harris J, Young R, Hiller J, Terrill N, Meek KM, Serra HM. The effect of vitamin C deficiency and chronic ultravioletB exposure on corneal ultrastructure: a preliminary investigation. Mol Vis. 2011;17:3107–3015.; Stojanovic A, Ringvold A, Nitter T. Ascorbate prophylaxis for corneal haze after photorefractive keratectomy. J Refract Surg. 2003 MayJun;19(3):338–343. doi:10.3928/1081-597X-20030501-11.; Marriott MP, Birt DF, Stallings VA, Yates AA, eds. “Vitamin C”. Present Knowledge in Nutrition, Eleventh Edition. London, United Kingdom: Academic Press (Elsevier), 2020. P. 155–170. ISBN 978-0-323-66162-1.; Методические рекомендации MP 2.3.1.025321 «Нормы физиологических потребностей в энергии и пищевых веществах для различных групп населения Российской Федерации» (утв. Федеральной службой по надзору в сфере защиты прав потребителей и благополучия человека 22 июля 2021 г.).; Carr AC, Lykkesfeldt J. Discrepancies in global vitamin C recommendations: a review of RDA criteria and underlying health perspectives. Crit Rev Food Sci Nutr. 2021;61(5):742–755. doi:10.1080/10408398.2020.1744513.; Chew EY, Clemons TE, Agrón E, Domalpally A, Keenan TDL, Vitale S, Weber C, Smith DC, Christen W; AREDS2 Research Group. Longterm Outcomes of Adding Lutein/Zeaxanthin and ω3 Fatty Acids to the AREDS Supplements on AgeRelated Macular Degeneration Progression: AREDS2 Report 28. JAMA Ophthalmol. 2022 Jul 1;140(7):692–698. doi:10.1001/jamaophthalmol.2022.1640.; Schleicher RL, Carroll MD, Ford ES, Lacher DA. Serum vitamin C and the prevalence of vitamin C deficiency in the United States: 2003–2004 National Health and Nutrition Examination Survey (NHANES). The American Journal of Clinical Nutrition. 2009;90(5):1252–1263.; AgeRelated Eye Disease Study Research Group. A randomized, placebocontrolled, clinical trial of highdose supplementation with vitamins C and E, beta carotene, and zinc for agerelated macular degeneration and vision loss: AREDS report no. 8. Arch Ophthalmol. 2001 Oct;119(10):1417–1436. doi:10.1001/archopht.119.10.1417. Erratum in: Arch Ophthalmol. 2008 Sep;126(9):1251.; World Health Organization. “Chapter 7: Vitamin C”. Vitamin and mineral requirements in human nutrition (2nd ed.). Geneva: World Health Organization, 2005. https://en.wikipedia.org/wiki/Vitamin_C; Narayanan S, Kumar SS, Manguvo A, Friedman E. Current Estimates of Serum Vitamin C and Vitamin C Deficiency in the United States. Curr Dev Nutr. 2021 Jun 7;5(Suppl 2):1067. doi:10.1093/cdn/nzab053_060. PMCID: PMC8180804.; https://www.ophthalmojournal.com/opht/article/view/2505
-
2Academic Journal
Συγγραφείς: I. Sh. Khassanov, Ильдар Шамильевич Хасанов
Πηγή: Siberian Journal of Clinical and Experimental Medicine; Том 30, № 1 (2015); 7-16 ; Сибирский журнал клинической и экспериментальной медицины; Том 30, № 1 (2015); 7-16 ; 2713-265X ; 2713-2927 ; 10.29001/2073-8552-2015-30-1
Θεματικοί όροι: non-specific inflammation factors, физиологичность лечения, нейрогуморальная регуляция, инотропная функция миокарда, провоспалительные факторы, electrotherapy of the heart, physiological therapy, neurohumoral regulation, inotropic myocardium function
Περιγραφή αρχείου: application/pdf
Relation: https://www.sibjcem.ru/jour/article/view/135/136; Selye H. Stress and disease // Science. - 1955. - Vol. 122 (3171). - P. 625-631.; Selye H. A Syndrome produced by nocuous agents // Nature. - 1936. - Vol. 138(3479). - P. 32.; Баевский Р.М., Берсенева А.П. Оценка адаптационных возможностей организма и риск развития заболеваний. -М.: Медицина, 1997. - С. 265.; Nathan D.A., Centre S., Wu C. et al. An implantable synchronous pacemaker for the long term correction of complete heart block // Am. J. Cardiol. - 1963. - Vol. 11. - P. 362-367.; Rediker D.E., Eagle K.A., Homma S. et al. Clinical and haemodynamic comparison of VVI versus DDD pacing in patients with DDD pacemakers // Am. J. Cardiol. - 1988. -Vol. 61. - P. 323-329.; Perrins J.E., Morley C.A., Chan S.L. et al. Randomized controlled trial of physiological and ventricular pacing // Br. Heart J. -1983. - Vol. 50. - P. 112-117.; Pehrsson S.K., Hjemdhal P., Norlander R. et al. A comparison of sympathoadrenal activity and cardiac performance at rest and during exercise in patients with ventricular demand or atrial synchronous pacing // Br. Heart J. - 1988. - Vol. 60. -P. 212-220.; He G.X., Song Z.Y., Zhou L.X. et al. Effects of physiological pacing and non-physiological pacing on hemodynamics, atrial natriuretic peptide and renin-angiotensin // J. HK Coll. Cardiol. - 1996. - Vol. 4. - P. 17-20.; Wiegand U.K.H.; Bode F., Schunkert H. Physiological pacing -quo vadis // Current Cardiology Reviews. - 2005. - Vol. 1(3). - P. 225-230.; Sweeney M.O., Hellkamp A.S., Ellenbogen K.A. et al. Adverse effect of ventricular pacing on heart failure and atrial fibrillation among patients with normal baseline QRS duration in a clinical trial of pacemaker therapy for sinus node dysfunction // Circulation. - 2003. - Vol. 107(23). - P. 2932-2937.; Lamas G.A., Lee K.L., Sweeney M.O. et al. Ventricular pacing or dual-chamber pacing for sinus-node dysfunction // N. Engl. J. Med. - 2002. - Vol. 346. - P. 1854-1862.; Steinberg J.S., Fischer A., Wang P. et al. The clinical implications of cumulative right ventricular pacing in the multicenter automatic defibrillator trial II // J. Cardiovasc. Electrophysiol. - 2005. - Vol. 16. - P. 359-365.; Nielsen J.C., Kristensen L., Andersen H.R. et al. A randomized comparison of atrial and dual-chamber pacing in 177 consecutive patients with sick sinus syndrome: echocardiographic and clinical outcome // J. Am. Coll. Cardiol. - 2003. - Vol. 42. - P. 614-623.; Wilkoff B.L., Cook J.R., Epstein A.E. et al. Dual-chamber pacing or ventricular backup pacing in patients with an implantable defibrillator: the Dual Chamber and VVI Implantable Defibrillator (DAVID) Trial // JAMA. - 2002. - Vol. 288. -P. 3115-3123.; Sweeney M.O., Hellkamp A.S. Heart failure during cardiac pacing // Circulation. - 2006. - Vol. 113. - P. 2082-2088.; Shukla H.H., Hellkamp A.S., James E.A. et al. Heart failure hospitalization is more common in pacemaker patients with sinus node dysfunction and a prolonged paced QRS duration // Heart Rhythm. - 2005. - Vol. 2. - P. 245-251.; Sweeney M.O., Hellkamp A.S., Lee K.L. et al. Association of prolonged QRS duration with death in a clinical trial of pacemaker therapy for sinus node dysfunction // Circulation. - 2005. - Vol. 111. - P. 2418-2423.; Sweeney M.O., Prinzen F.W. A new paradigm for physiologic ventricular pacing // J. Am. Coll. Cardiol. - 2006. - Vol. 47. -P. 282-288.; Kavanagh K.M., Belenkie I., Duff H.J. Transmural temporospatial left ventricular activation during pacing from different sites: potential implications for optimal pacing // Cardiovasc. Res. - 2008. - Vol. 77(1). - P. 81-88.; Prinzen F.W., Cheriex E.C., Delhaas T. et al. Asymmetric thickness of the left ventricular wall resulting from asynchronous electric activation: a study in dogs with ventricular pacing and in patients with left bundle branch block // Am. Heart J. - 1995. - Vol. 130. - P. 1045-1053.; Van Oosterhout M.F.M., Prinzen F.W., Arts T. et al. Asynchronous electrical activation induces asymmetrical hypertrophy of the left ventricular wall // Circulation. - 1998. - Vol. 98. - P. 588-595.; Nielsen J.C., Andersen H.R., Thomsen P.E.B. et al. Heart failure and echocardiographic changes during long-term follow-up of patients with sick sinus syndrome randomized to singlechamber atrial or ventricular pacing // Circulation. - 1998. -Vol. 97. - P. 987-995.; Nahlawi M., Waligora M., Spies S.M. Left ventricular function during and after right ventricular pacing // J. Am. Coll. Cardiol. - 2004. - Vol. 44. - P. 1883-1888.; Wan X., Bryant S.M., Hart G. A topographical study of mechanical and electrical properties of single myocytes isolated from normal guinea-pig ventricular muscle // J. Anat. - 2003. - Vol. 202. - P. 525-536.; Cordeiro J.M., Greene L., Heilmann C. Transmural heterogeneity of calcium activity and mechanical function in the canine left ventricle // Am. J. Physiol. Heart Circ. Physiol. - 2004. -Vol. 286. - P. H1471-H1479.; Sommer J.R., Jennings R.B. Ultrastructure of Cardiac Muscle // The Heart and Cardiovascular System / Ed. by H.A. Fozzard, R.B. Jennings, E. Haber, A.M. Katz, H.E. Morgan. - New York: Raven Press, 1991.; Katra R.P., Pruvot E., Laurita K.R. Intracellular calcium handling heterogeneities in intact guinea pig hearts // Am. J. Physiol. Heart Circ. Physiol. - 2004. - Vol. 286. - P. H648-H656.; Baker C.T., Bocharov G.A., Paul C.A. Modelling and analysis of time-lags in some basic patterns of cell proliferation // J. Math. Biol. - 1998. - Vol. 37. - 341-371.; Gardner T.S., Cantor C.R., Collins J.J. Construction of a genetic toggle switch in Escherichia coli // Nature. - 2000. - Vol. 403. - P. 339-342.; Koulakov A.A., Raghavachari S., Kepecs A. et al. Model for a robust neural integrator // Nat. Neurosci. - 2002. - Vol. 5. -P. 775-782.; Solovyova O., Katsnelson L.B., Konovalov P. et al. Activation sequence as a key factor in spatio-temporal optimization of myocardial function // Philos Transact A Math Phys Eng Sci. - 2006. - Vol. 364 (1843). - P. 1367-1383.; Abraham W.T., Fisher W.G., Smith A.L. et al. Cardiac resynchronization in chronic heart failure // N. Engl. J. Med. - 2002. - Vol. 346. - P. 1845-1853.; Auricchio A., Abraham W.T. Cardiac resynchronization therapy: current state of the art: cost versus benefit // Circulation. -2004. - Vol. 109. - P. 300-307.; Blanc J.J., Etienne Y., Gilard M. et al. Evaluation of different ventricular pacing sites in patients with severe heart failure: results of an acute hemodynamic study // Circulation. - 1997. - Vol. 96. - P. 3273-3277.; Leclercq J.F., Chouty F., Cauchemez B. et al. Results of electrical fulguration in arrhythmogenic right ventricular disease // Am. J. Cardiol. - 1988. - Vol. 62. - P. 220-224.; Cazeau S., Leclercq C., Lavergne T. et al. Effects of multisite biventricular pacing in patients with heart failure and intraventricular conduction delay // N. Engl. J. Med. - 2001. -Vol. 344. - P. 873-880.; Stellbrink C., Breithardt O.A., Franke A. et al. Impact of cardiac resynchronization therapy using hemodynamically optimized pacing on left ventricular remodeling in patients with congestive heart failure and ventricular conduction disturbances // J. Am. Coll. Cardiol. - 2001. - Vol. 38. -P. 1957-1965.; Abraham W.T., Fisher W.G., Smith A.L. Cardiac resynchronisation in heart failure // N. Engl. J. Med. - 2002. -Vol. 346. - P. 1845-1853.; Bristow M., Saxon L.A., Boehmer J. Cardiac-resynchronisation therapy with or without an implantable defibrillator in advanced heart failure // N. Engl. J. Med. - 2004. - Vol. 350. -P. 2140-2150.; Nelson G.S., Berger R.D., Fetics B.J. et al. Left ventricular or biventricular pacing improves cardiac function at diminished energy cost in patients with dilated cardiomyopathy and left bundle-branch block // Circulation. - 2000. - Vol. 102. -P. 3053-3059.; Burkhoff D., Oikawa R.Y., Sagawa K. Influence of pacing site on canine left ventricular contraction // Am. J. Physiol. Heart Circ. Physiol. - 1986. - Vol. 251. - P. H428-H435.; Beshai J.F., Grimm R.A., Nagueh S.F. et al. Cardiacresynchronization therapy in heart failure with narrow QRS complexes // N. Engl. J. Med. - 2007. - Vol. 357. - P. 24612471.; Dorn G.W., Molkentin J.D. Manipulating cardiac contractility in heart failure. Data from mice and men // Circulation. - 2004. - Vol. 109(2). - P. 150-158.; Bristow M.R. β-Adrenergic receptor blockade in chronic heart failure // Circulation. - 2000. - Vol. 101. - P. 558-569.; Диденко М.В., Шорохов К.Н., Хубулава Г.Г. Современные принципы физиологической электрокардиостимуляции // Вестник аритмологии. - 2007. - № 48. - С. 58-65.; Schaldach M., Urbaszek A., Strobel J. et al. Rate-adaptive pacing using a closed-loop stimulation, autonomic nervous system controlled pacemaker // J. Am. Coll. Cardiol. - 1995. - Vol. 3. - P. 22-32.; Levine B., Kalman J., Mayer L. et al. Elevated circulating levels of tumor necrosis factor in severe chronic heart failure // N. Engl. J. Med. - 1990. - Vol. 323(4). - P. 236-241.; Torre-Amione G. Immune activation in chronic heart failure // Am. J. Cardiol. - 2005. - Vol. 95. - P. 3C-8C.; Vonhof S., Brost B., Stille-Siegener M. et al. Monocyte activation in congestive heart failure due to coronary artery disease and idiopathic dilated cardiomyopathy // Int. J. Cardiol. - 1998. -Vol. 63(3). - P. 237-244.; Carswell E.A., Old L.J., Kassel R.L. et al. An endotoxin-induced serum factor that causes necrosis of tumor // Proc. Natl. Acad. Sci. USA. - 1975. - Vol. 72. - P. 3666-3670.; Levine B., Kalman J., Mayer L. et al. Elevated circulating levels of tumor necrosis factor in severe chronic heart failure // N. Engl. J. Med. - 1990. - Vol. 323. - P. 236-241.; Беленков Ю.Н., Агеев Ф.Т., Мареев В.Ю. Нейрогормоны и цитокины при сердечной недостаточности: новая теория старого заболевания? // Сердечная недостаточностью -2000. - № 1(4). - С. 135-138.; Von Haehling S., Jankowska E.A., Anker S.D. Tumour necrosis factor-alpha and the failing heart: pathophysiology and therapeutic implications // Basic Res. Cardiol. - 2004. -Vol. 99(1). - P. 18-28.; Irwin M.W., Mak S., Mann D.L. et al. Tissue expression and immunolocalization of tumor necrosis factor-α in postinfarction dysfunctional myocardium // Circulation. - 1999. - Vol. 99. - P. 1492-1498.; Zhao S.P., Xu T.D. Elevated tumor necrosis factor alpha of blood mononuclear cells in patients with congestive heart failure // Int. J. Cardiol. - 1999. - Vol. 71(3). - P. 257-261.; Moore K., O’Garra A., de Wall Malefyt R. et al. Interleukin-10 // Ann. Rev. Immunol. - 1993. - Vol. 11. - P. 165-171.; Singh K., Sirokman G., Communal C. et al. Myocardial osteopontin expression coincides with the development of heart failure // Hypertension. - 1999. - Vol. 33. - P. 663-670.; Graf K., Do Y.S., Ashizawa N. et al. Myocardial osteopontin expression is associated with left ventricular hypertrophy // Circulation. - 1997. - Vol. 96. - P. 3063-3071.; Xie Z., Singh M., Singh K. Osteopontin modulates myocardial hypertrophy in response to chronic pressure overload in mice // Hypertension. - 2004. - Vol. 44. - P. 826-831.; Williams E.B., Halpert I., Wickline S. et al. Osteopontin expression is increased in the heritable cardiomyopathy of Syrian hamsters // Circulation. - 1995. - Vol. 92. - P. 705709.; Komatsubara I., Murakami T., Kusachi S. et al. Spatially and temporally different expression of osteonectin and osteopontin in the infarct zone of experimentally induced myocardial infarction in rats // Cardiovasc. Pathol. - 2003. -Vol. 12. - P. 186-194.; Rosenberg M., Zugck C., Nelles M. et al. Osteopontin, a new prognostic biomarker in patients with chronic heart failure // Circ. Heart Fail. - 2008. - Vol. 1(1). - P. 43-49.; Stawowy P., Blaschke F., Pfautsch P. et al. Increased myocardial expression of osteopontin in patients with advanced heart failure // Eur. J. Heart Fail. - 2002. - Vol. 4. - P. 139-146.; Satoh M., Nakamura M., Akatsu T. et al. Myocardial osteopontin expression is associated with collagen fibrillogenesis in human dilated cardiomyopathy // Eur. J. Heart Fail. - 2005. -Vol. 7. - P. 755-762.; Suezawa C., Kusachi S., Murakami T. et al. Time dependent changes in plasma osteopontin levels in patients with anteriorwall acute myocardial infarction after successful reperfusion: correlation with left-ventricular volume and function // J. Lab. Clin. Med. - 2005. - Vol. 145. - P. 33-40.; Kotlyar E., Vita J.A., Winter M.R. et al. The relationship between aldosterone, oxidative stress, and inflammation in chronic, stable human heart failure // J. Card. Fail. - 2006. - Vol. 12. -P. 122-127.; Okamoto H. Osteopontin and the cardiovascular system // Mol. Cell. Biochem. - 2007. - Vol. 300. - P. 1-7.; Bozkurt B., Kribbs S.B., Clubb F.J. et al. Pathophysiologically relevant concentrations of tumor necrosis factor-a promote progressive left ventricular dysfunction and remodeling in rats // Circulation. - 1998. - Vol. 97. - P. 1382-1391.; Kapadia S.R., Oral H., Lee J. et al. Hemodynamic regulation of tumor necrosis factor-α gene and protein expression in adult feline myocardium // Circ. Res. - 1997. - Vol. 81. - P. 187-195.; Anker S.D., Egerer K., Volk H.-D. et al. Elevated soluble CD 14 receptors and altered cytokines in chronic heart failure // Am. J. Cardiol. - 1997. - Vol. 79. - P. 1426-1430.; Neibauer J., Volk H.-D., Kemp M. et al. Endotoxin and immune activation in heart failure: a prospective cohort study // Lancet. - 1999. - Vol. 353. - P. 1838-1842.; Peschel T., Anker S.D., Ziegenbalg K. et al. Endotoxemia in congestive heart failure: highest levels in hepatic veins suggestive of intestinal bacterial and/or endotoxin translocation // Eur. J. Heart Fail. - 2000. - Vol. 2 (Suppl. 2). -P. 22/10452.; Peschel T. Invasive assessment of bacterial endotoxin and inflammatory cytokines in patients with acute heart failure // Eur. J. Heart Fail. - 2003. - Vol. 5(5). - P. 609-614.; Torre-Amione G., Kapadia S., Benedict C. et al. Proinflammatory cytokine levels in patients with depressed left ventricular ejection fraction: a report from of SOLVd // J. Am. Coll. Cardiol. - 1996. - Vol. 27. - P. 1201-1206.; Gurlek A., Kilikcap M., Dandachi R. et al. Tumor necrosis factoralpha in diastolic heart failure // Eur. J. Heart Fail. - 2000. -Vol. 2 (Suppl. 2). - P. 28/10381.; Anker S.D., Engerer K.R., Volk H.D. et al. Elevated soluble CD14 receptors and altered cytokines in chronic heart failure // Am. J. Cardiol. - 1997. - Vol. 79(10). - P. 1426-1430.; Randow F., Syrbe U., Meisel C. et al. Mechanism of endotoxin desensitization: involvement of interleukin 10 and transforming growth factor beta // J. Exp. Med. - 1995. -Vol. 181. - P. 1887-1892.; Sharma R., Bolger A., Rauchhaus M. et al. Cellular endotoxin desensitization in patients with severe chronic heart failure // Eur. J. Heart Fail. - 2005. - Vol. 7(5). - P. 865-868.; Genth-Zotz S., von Haehling S., Bolger A.P. et al. Pathophysiologic quantities of endotoxin-induced tumor necrosis factor-αlpha release in whole blood from patients with chronic heart failure // Am. J. Cardiol. - 2002. -Vol. 90(11). - P. 1226-1230.; Fan H., Kuck J.A. Review: Molecular mechanisms of endotoxin tolerance // J. Endotoxin Research. - 2004. - Vol. 10(2). -P. 71-84.; Frantz S., Kobzik L., Kim Y.D. et al. Toll4 (TLR4) expression in cardiac myocytes in normal and failing myocardium // J. Gin. Invest. - 1999. - Vol. 104. - P. 271-280.; Brown J.M., Grosso M.A., Terada L.S. et al. Endotoxin pretreatment increases endogenous myocardial catalase activity and decreases ischemia-reperfusion injury of isolated rat hearts // Proc. Natl. Acad. Sci. USA. - 1989. - Vol. 86. -P. 2516-2520.; Yao Z., Auchampach J.A., Pieper G.M. et al. Cardioprotective effects of monophosphoryl lipid-A, a novel endotoxin analogue, in dog // Cardiovasc. Res. - 1993. - Vol. 27. -P. 832-838.; Li C., Ha T., Kelley J. et al. Modulating Toll-like receptor mediated signaling by (1->3)-β-D-glucan rapidly induces cardioprotection // Cardiovasc. Res. - 2003. - Vol. 61. -P. 538-547.; Li C., Browder W., Kao R.L. Early activation of transcription factor NF-kB during ischemia in perfused rat heart // Am. J. Physiol. - 1999. - Vol. 276. - P. H543-H552.; Oyama J., Blais C. Jr., Liu X. et al. Reduced myocardial ischemia-reperfusion injury in Toll-like receptor 4-deficient mice // Circulation. - 2004. - Vol. 109. - P. 784-789.; Hua F., Ha T., Ma J. et al. Protection against myocardial ischemia/reperfusion injury in TLR4 deficient mice is mediated through a phosphoinositide 3-kinase dependent mechanism // J. Immunol. - 2007. - Vol. 178. - P. 7317-7324.; Morishita R., Sugimoto T., Aoki M. et al. In vivo transfection of cis element ‘decoy’ against nuclear factor-kappaB binding site prevents myocardial infarction // Nat. Med. - 1997. - Vol. 3. - P. 894-899.; Аниховская И.А., Кубатиев А.А., Яковлев М.Ю. Эндотоксиновая теория атеросклероза // Физиология человека. -2015. - Т. 41(1). - С. 1-11.; Ishida K., Kimura F., Imamaki M. Relation of inflammatory cytokines to atrial fibrillation after off-pump coronary artery bypass grafting // Eur. J. Cardiothorac. Surg. - 2006. - Vol. 29. - P. 501-505.; Bruins P., te Velthuis H., Yazdanbakhsh A.P. et al. Activation of the complement system during and after cardiopulmonary bypass surgery: postsurgery activation involves C-reactive protein and is associated with postoperative arrhythmia // Circulation. - 1997. - Vol. 96. - P. 3542-3548.; Abdelhadi R.H., Gurm H.S., van Wagoner D.R. et al. Relation of an exaggerated rise in white blood cells after coronary bypass or cardiac valve surgery to development of atrial fibrillation postoperatively // Am. J. Cardiol. - 2004. - Vol. 93. - P. 1176-1178.; Aviles R.J., Martin D.O., Apperson-Hansen C. et al. Inflammation as a risk factor for atrial fibrillation // Circulation. - 2003. -Vol. 108. - P. 3006-3010.; Chung M.K., Martin D.O., Sprecher D. et al. C-reactive protein elevation in patients with atrial arrhythmias: inflammatory mechanisms and persistence of atrial fibrillation // Circulation. - 2001. - Vol. 104. - P. 2886-2891.; Conway D.S., Buggins P., Hughes E. et al. Prognostic significance of raised plasma levels of interleukin-6 and C-reactive protein in atrial fibrillation // Am. Heart J. - 2004. - Vol. 148. -P. 462-466.; Conway D.S., Buggins P., Hughes E. et al. Relationship of interleukin-6 and C-reactive protein to the prothrombotic state in chronic atrial fibrillation // J. Am. Coll. Cardiol. - 2004. - Vol. 43. - P. 2075-2082.; Ellinor P.T., Low A., Patton K.K. et al. C-reactive protein in lone atrial fibrillation // Am. J. Cardiol. - 2006. - Vol. 97. - P. 1346-1350.; Rotter M., Jais P., Vergnes M.C. et al. Decline in C-reactive protein after successful ablation of long-lasting persistent atrial fibrillation // J. Am. Coll. Cardiol. - 2006. - Vol. 47. - P. 1231-1233.; Dernellis J., Panaretou M. C-reactive protein and paroxysmal atrial fibrillation: evidence of the implication of an inflammatory process in paroxysmal atrial fibrillation // Acta Cardiol. - 2001. - Vol. 56. - P. 375-380.; Wazni O.M., Martin D.O., Marrouche N.F. et al. C-reactive protein concentration and recurrence of atrial fibrillation after electrical cardioversion // Heart. - 2005. - Vol. 91. - P. 1303-1305.; Goette A., Arndt M., Rocken C. et al. Calpains and cytokines in fibrillating human atria // Am. J. Physiol. Heart Circ. Physiol. -2002. - Vol. 283. - P. H264-H272.; Madrid A.H. Atrial fibrillation and C-reactive protein // J. Am. Coll. Cardiol. - 2007. - Vol. 49. - P. 1649-1650.; Watson T., Kakar P., Lip G.Y. Cardioversion for atrial fibrillation: does inflammation matter? // Am. J. Cardiol. - 2007. -Vol. 99. - P. 1617-1618.; Murray D.R., Freeman G.L. Proinflammatory cytokines. Predictors of a failing heart? // Circulation. - 2003. - Vol. 107. - P. 1460-1462.; Haissaguerre M., Shah D.C., Jais P. et al. Mapping-guided ablation of pulmonary veins to cure atrial fibrillation // Am. J. Cardiol. - 2000. - Vol. 86. - P. K9-K19.; Fromer M., Genton C., Schlaepfer J. et al. Is there an isolated arrhythmogenic right atrial myocarditis? // Eur. Heart J. - 1990. - Vol. 11. - P. 566-571.; Frustaci A., Chimenti C., Bellocci F. et al. Histological substrate of atrial biopsies in patients with lone atrial fibrillation // Circulation. - 1997. - Vol. 96. - P. 1180-1184.; Korantzopoulos P., Kolettis T., Siogas K. et al. Atrial fibrillation and electrical remodeling: the potential role of inflammation and oxidative stress // Med. Sci. Monit. - 2003. - Vol. 9. -P. RA225-RA229.; Ishii Y., Schuessler R.B., Gaynor S.L. et al. Inflammation of atrium after cardiac surgery is associated with inhomogeneity of atrial conduction and atrial fibrillation // Circulation. - 2005. - Vol. 111. - P. 2881-2888.; Shiroshita-Takeshita A., Schram G., Lavoie J. et al. Effect of simvastatin and antioxidant vitamins on atrial fibrillation promotion by atrial-tachycardia remodeling in dogs // Circulation. - 2004. - Vol. 110. - P. 2313-2319.; Liuba I., Ahlmroth H., Jonasson L. et al. Source of inflammatory markers in patients with atrial fibrillation // Europace. - 2008. - Vol. 10(7). - P. 848-853.; Waldo A.L. Mechanisms of atrial fibrillation // J. Cardiovasc. Electrophysiol. - 2003. - Vol. 14. - P. S267-S274.; Halonen J., Halonen P., Jarvinen O. et al. Corticosteroids for the Prevention of Atrial Fibrillation After Cardiac Surgery. A Randomized Controlled Trial // J. Am. Med. Ass. - 2007. -Vol. 297. - P. 1562-1567.; Dantzer R. Cytokine-induced sickness behavior: mechanisms and implications // Ann. N. Y. Acad. Sci. - 2001. - Vol. 933. -P. 222-234.; Mann D.L. Inflammatory mediators and the failing heart: past, present, and the foreseeable future // Circ. Res. - 2002. -Vol. 91. - P. 988-998.; Mann D.L. Stress-activated cytokines and the heart: from adaptation to maladaptation // Ann. Rev. Physiol. - 2003. -Vol. 65. - P. 81-101.; Packer M. Neurohormonal interactions and adaptations in congestive heart failure // Circulation. - 1988. - Vol. 77. -P. 721-730.; Packer M. Role of the sympathetic nervous system in chronic heart failure: a historical and philosophical perspective // Circulation. - 1990. - Vol. 82, Suppl. I. - P. I1-I6.; Chung E.S., Packer M., Lo K.H. et al. Randomized, double-blind, placebo-controlled, pilot trial of infliximab, a chimeric monoclonal antibody to tumor necrosis factor-αlpha, in patients with moderate-to-severe heart failure: results of the anti-TNF Therapy against Congestive Heart Failure (ATTACH) trial // Circulation. - 2003. - Vol. 107. - P. 3133-3140.; Cohn J.N., Tognoni G. A randomized trial of the angiotensin-receptor blocker valsartan in chronic heart failure // N. Engl. J. Med. - 2001. - Vol. 345. - P. 1667-1675.; Mann D.L., McMurray J.J., Packer M. et al. Targeted anticytokine therapy in patients with chronic heart failure: results of the Randomized Etanercept Worldwide Evaluation (RENEWAL) // Circulation. - 2004. - Vol. 109. - P. 1594-1602.; Cohn J.N., Pfeffer M.A., Rouleau J. et al. Adverse mortality effect of central sympathetic inhibition with sustained-release moxonidine in patients with heart failure (MOXCON) // Eur. J. Heart Fail. - 2003. - Vol. 5. - P. 659-667.; Guz A. Chairman’s Introduction // The physiological basis of starling’s law of the heart / edited by R. Porter and D.W. Fitzsimons. - Amsterdam: Associated Scientific Publishers, 1974. - P. 1-6.; Lakatta E.G. Age-related alterations in the cardiovascular response to adrenergic mediated stress // Fed. Proc. - 1980. -Vol. 39. - P. 3173-3177.; Lakatta E.G., Gerstenblith G., Angell C.S. et al. Diminished inotropic response of aged myocardium to catecholamines // Circulation. - 1975. - Vol. 36. - P. 262-269.; Poliner L.R., Dehmer G.J., Lewis S.E. et al. Left ventricular performance in normal subjects: A comparison of the responses to exercise in the upright and supine positions // Circulation. - 1980. - Vol. 62. - P. 528-534.; Rodeheffer R.J., Gerstenblith G., Beard E. et al. Postural changes in cardiac volumes in men in relation to adult age // Exp. Gerontol. - 1986. - Vol. 21. - P. 367-378.; Granath A., Jonsson B., Strandell T. Circulation in healthy old men studied by right heart catheterization at rest and during exercise in supine and sitting position // Acta Med. Scand. -1964. - Vol. 169. - P. 125-126.; Rodeheffer R.J., Gerstenblith G., Becker L.C. et al. Exercise cardiac output is maintained with advancing age in healthy human subjects: Cardiac dilatation and increased stroke volume compensate for a diminished heart rate // Circulation. - 1984. - Vol. 69. - P. 203-213.; Braunwald E., Ross J., Jr. Applicability of Starling’s law of the heart to man // Circ. Res. - 1964. - Vol. 15. - P. 169-181.; Schwinger R.H., Bohm M., Koch A. et al. The failing human heart is unable to use the Frank-Starling mechanism // Circ. Res. - 1994. - Vol. 74. - P. 959-969.; Holubarsch C., Ruf T., Goldstein D.J. et al. Existence of the Frank-Starling mechanism in the failing human heart: investigations on the organ, tissue, and sarcomere levels // Circulation. - 1996. - Vol. 94. - P. 683-689.; Jacob R., Dierberger B., Kissling G. Functional significance of the Frank-Starling mechanism under physiological and pathophysiological conditions // Eur. Heart J. - 1992. -Vol. 13. - P. 7-14.; Berk B.C., Fujiwara K., Lehoux S. ECM remodeling in hypertensive heart disease // J. Clin. Invest. - 2007. - Vol. 117. - P. 568-575.; Warren C.M., Jordan M.C., Roos K.P. et al. Titin isoform expression in normal and hypertensive myocardium // Cardiovas. Res. - 2003. - Vol. 59. - P. 86-94.; Borbely A., van der Velden J., Papp Z. et al. Cardiomyocyte stiffness in diastolic heart failure // Circulation. - 2005. -Vol. 111. - P. 774-781.; Neagoe C., Kulke M., del Monte F. et al. Titin isoform switch in ischemic human heart disease // Circulation. - 2002. -Vol. 106. - P. 1333-1341.; Fuchs F., Martyn D.A. Length-dependent Ca2+ activation in cardiac muscle: some remaining questions // J. Muscle Res. & Cell Motility. - 2005. - Vol. 26. - P. 199-212.; https://www.sibjcem.ru/jour/article/view/135
-
3Academic Journal
Συγγραφείς: Белянцева, Елена, Федоров, Игорь, Телешева, Лариса, Степанов, Олег
Θεματικοί όροι: БРОНХОЛЕГОЧНАЯ ДИСПЛАЗИЯ, РЕМОДЕЛЛИНГ СЛИЗИСТОЙ ОБОЛОЧКИ БРОНХОВ, РОСТОВЫЕ И ПРОВОСПАЛИТЕЛЬНЫЕ ФАКТОРЫ, ЭПИТЕЛИАЛЬНО-МЕЗЕНХИМАЛЬНЫЕ ВЗАИМООТНОШЕНИЯ, РЕСПИРАТОРНЫЙ ДИСТРЕСС СИНДРОМ НОВОРОЖДЕННЫХ
Περιγραφή αρχείου: text/html
-
4Academic Journal
Πηγή: Человек. Спорт. Медицина.
Θεματικοί όροι: 0502 economics and business, 05 social sciences, БРОНХОЛЕГОЧНАЯ ДИСПЛАЗИЯ, РЕМОДЕЛЛИНГ СЛИЗИСТОЙ ОБОЛОЧКИ БРОНХОВ, РОСТОВЫЕ И ПРОВОСПАЛИТЕЛЬНЫЕ ФАКТОРЫ, ЭПИТЕЛИАЛЬНО-МЕЗЕНХИМАЛЬНЫЕ ВЗАИМООТНОШЕНИЯ, РЕСПИРАТОРНЫЙ ДИСТРЕСС СИНДРОМ НОВОРОЖДЕННЫХ, 3. Good health
Περιγραφή αρχείου: text/html
-
5Academic Journal
Πηγή: Сибирский медицинский журнал (Томск).
Θεματικοί όροι: 03 medical and health sciences, 0302 clinical medicine, ЭЛЕКТРОКАРДИОТЕРАПИЯ, ФИЗИОЛОГИЧНОСТЬ ЛЕЧЕНИЯ, НЕЙРОГУМОРАЛЬНАЯ РЕГУЛЯЦИЯ, ИНОТРОПНАЯ ФУНКЦИЯ МИОКАРДА, ПРОВОСПАЛИТЕЛЬНЫЕ ФАКТОРЫ, 3. Good health
Περιγραφή αρχείου: text/html
-
6Academic Journal
Συγγραφείς: Belyantseva, E. S., Fyodorov, I. A., Telesheva, L. F., Stepanov, O. G.
Θεματικοί όροι: ремоделлинг слизистой оболочки бронхов, ГРНТИ 76.29, ростовые и провоспалительные факторы, growth-stimulating and anti-inflammatory factors, УДК 616.23-053.32-09-07, респираторный дистресс синдром новорожденных, bronchopulmonary dysplasia, эпителиально-мезенхимальные взаимоотношения, epithelial-mesenchymal relationships, respiratory distress syndrome of the newborn, бронхолегочная дисплазия, bronchial mucosa remodeling
Σύνδεσμος πρόσβασης: http://dspace.susu.ac.ru/xmlui/handle/0001.74/7309
-
7Academic Journal
Συγγραφείς: Алтынбаева, Е. И., Теплова, С. Н., Игнатова, Г. Л., Галимова, О. В.
Θεματικοί όροι: ХОБЛ, МУКОЗАЛЬНЫЙ ИММУНИТЕТ, НЕИНВАЗИВНЫЙ МЕТОД, ПРОВОСПАЛИТЕЛЬНЫЕ ФАКТОРЫ СЛЮНЫ, АНТИМИКРОБНАЯ ЗАЩИТА
Περιγραφή αρχείου: application/pdf
Relation: Уральский медицинский журнал. 2007. Т. 33, № 5.; http://elib.usma.ru/handle/usma/17339
Διαθεσιμότητα: http://elib.usma.ru/handle/usma/17339
-
8Academic Journal
Συγγραφείς: Белянцева, Е. С., Федоров, И. А., Телешева, Л. Ф., Степанов, О. Г., Belyantseva, E. S., Fyodorov, I. A., Telesheva, L. F., Stepanov, O. G.
Θεματικοί όροι: бронхолегочная дисплазия, ремоделлинг слизистой оболочки бронхов, ростовые и провоспалительные факторы, эпителиально-мезенхимальные взаимоотношения, респираторный дистресс синдром новорожденных, bronchopulmonary dysplasia, bronchial mucosa remodeling, growth-stimulating and anti-inflammatory factors, epithelial-mesenchymal relationships, respiratory distress syndrome of the newborn, УДК 616.23-053.32-09-07, ГРНТИ 76.29
Περιγραφή αρχείου: application/pdf
Relation: Вестник ЮУрГУ. Серия Образование, здравоохранение, физическая культура; Vestnik Yuzhno-Ural'skogo Gosudarstvennogo Universiteta. Seriya Obrazovaniye , zdravookhraneniye , fizicheskaya kul'tura; Bulletin of SUSU; Bulletin of South Ural State University; Образование, здравоохранение, физическая культура;Том 15; http://dspace.susu.ac.ru/xmlui/handle/0001.74/7309
Διαθεσιμότητα: http://dspace.susu.ac.ru/xmlui/handle/0001.74/7309
-
9Academic Journal
Συγγραφείς: Хаддад Сами, Борисов, А. В.
Θεματικοί όροι: провоспалительные факторы, сыворотка крови, хронический калькулезный пиелонефрит, эйказоноиды
Περιγραφή αρχείου: application/pdf
Relation: Хаддад Сами. Диагностическое значение повышения содержания провоспалительных факторов в крови и моче больных с активно протекающим хроническим калькулезным пиелонефритом / Хаддад Сами, А. В. Борисов / Конгрес асоціації урологів України, 18–20 червня 2015, м. Київ: матер. – Київ, 2015. – С. 83–84.; https://repo.odmu.edu.ua:443/xmlui/handle/123456789/10594
Διαθεσιμότητα: https://repo.odmu.edu.ua:443/xmlui/handle/123456789/10594
