Αποτελέσματα αναζήτησης - "обратное ремоделирование"

1

Academic Journal

The value of percutaneous transcatheter mitral valve regurgitation repair in the combination treatment of chronic heart failure patients: Results from a 6-month observational prospective study ; Роль чрескожной транскатетерной коррекции митральной недостаточности в комплексном лечении пациентов с хронической сердечной недостаточностью: результаты 6-месячного наблюдения

Συγγραφείς: Karamova Y.S., Uskach T.M., Imaev T.E., Tereshchenko S.N.

Συνεισφορές: 1

Πηγή: Almanac of Clinical Medicine; Vol 52, No 2 (2024); 77-84 ; Альманах клинической медицины; Vol 52, No 2 (2024); 77-84 ; 2587-9294 ; 2072-0505

Θεματικοί όροι: heart failure, secondary mitral regurgitation, transcatheter mitral valve repair, clipping, drug therapy, remodeling, хроническая сердечная недостаточность (ХСН), вторичная митральная регургитация, транскатетерная пластика митрального клапана, оптимальная медикаментозная терапия, обратное ремоделирование

Περιγραφή αρχείου: application/pdf

Διαθεσιμότητα: https://almclinmed.ru/jour/article/view/17241
https://doi.org/10.18786/2072-0505-2024-52-011

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2

Academic Journal

Physiological aspects of myocardial function improving during mechanical circulatory support ; Физиологические аспекты улучшения функции миокарда на фоне механической поддержки кровообращения

Συγγραφείς: D. V. Shumakov, D. I. Zybin, M. A. Popov, Д. В. Шумаков, Д. И. Зыбин, М. А. Попов

Πηγή: Transplantologiya. The Russian Journal of Transplantation; Том 11, № 4 (2019); 311-319 ; Трансплантология; Том 11, № 4 (2019); 311-319 ; 2542-0909 ; 2074-0506 ; 10.23873/2074-0506-2019-11-4

Θεματικοί όροι: трансплантация сердца, mechanical circulatory support, myocardial remodeling, reverse myocardial remodeling, heart transplantation, механическая поддержка кровообращения, ремоделирование миокарда, обратное ремоделирование миокарда

Περιγραφή αρχείου: application/pdf

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Regression of cellular hypertrophy after left ventricular assist device support. Circulation. 1998;98(7):656–662. PMID: 9715858 https://doi.org/10.1161/01.cir.98.7.656; Razeghi P, Taegtmeyer H. Hypertrophy and atrophy of the heart: the other side of remodeling. Ann N Y Acad Sci. 2006;1080:110–119. PMID: 17132779 https://doi.org/10.1196/annals.1380.011; Wohlschlaeger J, Sixt SU, Stoeppler T, Schmitz KJ, Levkau B, Tsagakis K, et al. Ventricular unloading is associated with increased 20s proteasome protein expression in the myocardium. J Heart Lung Transplant. 2010;29(1):125–132 PMID: 19837610 https://doi.org/10.1016/j.healun.2009.07.022; Soppa GK, Barton PJ, Terracciano CM, Yacoub MH. Left ventricular assist device-induced molecular changes in the failing myocardium. Curr Opin Cardiol. 2008;23(3):206–18. PMID: 18382208 https://doi.org/10.1097/HCO.0b013e3282fc7010; Terracciano CM, Hardy J, Birks EJ, Khaghani A, Banner NR, Yacoub MH. Clinical recovery from end-stage heart failure using left-ventricular assist device and pharmacological therapy correlates with increased sarcoplasmic reticulum calcium content but not with regression of cellular hypertrophy. Circulation. 2004;109(19):2263–2265. PMID: 15136495 https://doi.org/10.1161/01. CIR.0000129233.51320.92; Hall JL, Birks EJ, Grindle S, Cullen ME, Barton PJ, Rider JE, et al. Molecular signature of recovery following combination left ventricular assist device (LVAD) support and pharmacologic therapy. Eur Heart J. 2007;28(5):613–627. PMID: 17132651 https://doi.org/10.1093/eurheartj/ehl365; Vatta M, Stetson SJ, Perez-Verdia A, Entman ML, Noon GP, TorreAmione G, et al. Molecular remodelling of dystrophin in patients with endstage cardiomyopathies and reversal in patients on assistance-device therapy. Lancet. 2002;359(9310):936–941. PMID: 11918913 https://doi.org/10.1016/S0140-6736(02)08026-1; Birks EJ, Hall JL, Barton PJ, Grindle S, Latif N, Hardy JP, et al. Gene profiling changes in cytoskeletal proteins during clinical recovery after left ventricular-assist device support. Circulation. 2005;112(9Suppl):I57–I64. PMID: 16159866 https://doi.org/10.1161/CIRCULATIONAHA.104.526137; Latif N, Yacoub MH, George R, Barton PJR, Birks EJ. Changes in sarcomeric and non-sarcomeric cytoskeletal proteins and focal adhesion molecules during clinical myocardial recovery after left ventricular assist device support. J Heart Lung Transplant. 2007;26(3):230–235. PMID: 17346624 https://doi.org/10.1016/j.healun.2006.08.011; de Jonge N, van Wichen DF, Schipper ME, Lahpor JR, Gmelig- Meyling FH, Robles de Medina EO. Left ventricular assist device In end-stage heart failure: persistence of structural myocyte damage after unloa ding: Animmunohistochemical analysis of the contractile myofilaments. J Am Coll Cardiol. 2002;39(6):963–969. 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PMID: 11092662 https://doi.org/10.1016/S0735-1097(00)00948-7; Heerdt PM, Schlame M, Jehle R, Barbone A, Burkhoff D, Blanck TJ. Diseasespecific remodeling of cardiac mitochondria after a left ventricular assist device. Ann Thorac Surg. 2002;73(4):1216–1221. PMID: 11996266 https://doi.org/10.1016/S0003-4975(01)03621-9; Cullen ME, Yuen AH, Felkin LE, Smolenski RT, Hall JL, Grindle S, et al. Myocardial expression of the arginine: glycineamidinotransferase gene is elevated in heart failure and normalized after recovery: potential implications for local creatine synthesis. Circulation. 2006;114(1Suppl):I16–I20. PMID: 16820567 https://doi.org/10.1161/CIRCULATIONAHA.105.000448; Doenst T, Abel ED. Spotlight on metabolic remodelling in heart failure. Cardiovasc Res. 2011;90(2):191–193. PMID: 21429943 https://doi.org/10.1093/cvr/cvr077; Kassiotis C, Ballal K, Wellnitz K, Vela D, Gong M, Salazar R, et al. Markers of autophagy are down regulated in failing human heart after mechanical unloading. Circulation. 2009;120(11Suppl):S191–S197. PMID: 19752367 https://doi.org/10.1161/CIRCULATIONAHA.108.842252; Baba HA, Grabellus F, August C, Plenz G, Takeda A, Tijan TD, et al. Reversal of metallothionein expression is different throughout the human myocardium after prolonged left-ventricular mechanical support. J Heart Lung Transplant. 2000;19(7):668–674. PMID: 10930816 https://doi.org/10.1016/S1053-2498(00)00074-7; Grabellus F, Schmid C, Levkau B, Breukelmann D, Halloran PF, August C, et al. Reduction of hypoxia-inducible heme oxygenase-1 in the myocardium after left ventricular mechanical support. J Pathol. 2002;197(2):230–237. PMID: 12015748 https://doi.org/10.1002/path.1106; Drakos SG, Kfoury AG, Hammond EH, Reid BB, Revelo MP, Rasmusson BY, et al. Impact of mechanical unloading on microvasculature and associated central remodeling features of the failing human heart. J Am Coll Cardiol. 2010;56(5):382–391. PMID: 20650360 https://doi.org/10.1016/j.jacc.2010.04.019; Manginas A, Tsiavou A, Sfyrakis P, Giamouzis G, Tsourelis L, Leontiadis E, et al. Increased number of circulating progenitor cells after implantation of ventricular assist devices. J Heart Lung Transplant. 2009;28(7):710–717. PMID: 19560700 https://doi.org/10.1016/j.healun.2009.04.006; Wohlschlaeger J, Levkau B, Brockoff G, Schmitz K J, von Winterfeld M, Takeda A, et al. Hemodynamic support by left ventricular assist devices reduces cardiomyocyte DNA content in the failing human heart. Circulation. 2010;121(8):989–996. PMID: 20159834 https://doi.org/10.1161/CIRCULATIONAHA.108.808071; Klotz S, Foronjy RF, Dickstein ML, Gu A, Garrelds IM, Danser AH, et al. Mechanical unloading during left ventricular assist device support increases left ventricular collagen cross-linking and myocardial stiffness. Circulation. 2005;112(3):364–374. 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PMID:19237659 https://doi.org/10.1161/CIRCULATIONAHA.108.813576; de Weger RA, Schipper ME, Sierade Koning E, van der Weide P, Quadir R, Lahpor JR, et al. Proteomic profiling of the human failing heart after left ventricular assist device support. J Heart Lung Transplant. 2011;30(5):497–506. PMID: 21211997 https://doi.org/10.1016/j.healun.2010.11.011; Ramani R, Vela D, Segura A, McNamara D, Lemster B, Samarendra V, et al. A micro-ribonucleic acid signature associated with recovery from assist device support in 2 groups of patients with severe heart failure. J Am Coll Cardiol. 2011;58(22):2270–2278. PMID: 22093502 https://doi.org/10.1016/j.jacc.2011.08.041; Bruggink AH, de Jonge N, van Oosterhout MF, van Wichen DF, de Ko ning E, Lahpor JR, et al. Brain natriuretic peptide is produced both by cardiomyocytes and cells infiltrating the heart in patients with severe heart failure supported by a left ventricular assist device. J Heart Lung Transplant. 2006;25(2):174–180. 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PMID: 7586406 https://doi.org/10.1161/01.CIR.92.9.191; Drakos SG, Athanasoulis T, Malliaras KG, Terrovitis JV, Diakos N, Koudoumas D, et al. Myocardial sympathetic innervation and long-term left ventricular mechanical unloading. JACC Cardiovasc Imaging. 2010;3(1):64–70. PMID: 20129533 https://doi.org/10.1016/j.jcmg.2009.10.008; Mann DL, Bristow MR. Mechanisms and models in heart failure: the biomechanical model and beyond. Circulation. 2005;111(21):2837–2849. PMID: 15927992 https://doi.org/10.1161/CIRCULATIONAHA.104.500546; Mann DL, Burkhoff D. Myocardial expression levels of micro-ribonucleic acids in patients with left ventricular assist devices signature of myocardial recovery, signature of reverse remodeling, or signature with no name? J Am Coll Cardiol. 2011;58(22):2279–81. PMID: 22093503 https://doi.org/10.1016/j.jacc.2011.09.007; Koitabashi N, Kass DA. Reverse remodeling in heart failure-mechanisms and therapeutic opportunities. Nat Rev Cardiol. 2011;9(3):147–157. PMID: 22143079 https://doi.org/10.1038/nrcardio.2011.172; Drakos SG, Kfoury AG, Selzman CH, Verma DR, Nanas JN, Li DY, et al. Left ventricular assist device unloading effects on myocardial structure and function: current status of the field and call for action. Curr Opin Cardiol. 2011;26(3):245–255. PMID: 21451407 https://doi.org/10.1097/HCO.0b013e328345af13; https://www.jtransplantologiya.ru/jour/article/view/463

Διαθεσιμότητα: https://www.jtransplantologiya.ru/jour/article/view/463
https://doi.org/10.23873/2074-0506-2019-11-4-311-319

View record from BASE

3

Academic Journal

FLEXIBLE RINGS IN COMPARISON WITH RIGID RINGS IN THE TREATMENT OF FUNCTIONAL TRICUSPID INSUFFICIENCY: TWO DIFFERENT MODELS OF REVERSE REMODELING OF THE RIGHT HEART ; СРАВНЕНИЕ ГИБКИХ И ЖЕСТКИХ КОЛЕЦ ДЛЯ КОРРЕКЦИИ ФУНКЦИОНАЛЬНОЙ ТРИКУСПИДАЛЬНОЙ НЕДОСТАТОЧНОСТИ: ДВЕ РАЗНЫЕ МОДЕЛИ ОБРАТНОГО РЕМОДЕЛИРОВАНИЯ ПРАВЫХ ОТДЕЛОВ СЕРДЦА

Συγγραφείς: M. A. Ovcharov, A. V. Bogachev-Prokofiev, D. A. Astapov, A. N. Pivkin, K. S. Malozemov, T. N. Podsosnikova, O. Yu. Malakhova, A. M. Karaskov, М. А. Овчаров, А. В. Богачев-Прокофьев, Д. А. Астапов, А. Н. Пивкин, К. С. Малоземов, Т. Н. Подсосникова, О. Ю. Малахова, А. М. Караськов

Πηγή: Siberian Journal of Clinical and Experimental Medicine; Том 34, № 2 (2019); 89-98 ; Сибирский журнал клинической и экспериментальной медицины; Том 34, № 2 (2019); 89-98 ; 2713-265X ; 2713-2927 ; 10.29001/2073-8552-2019-34-2

Θεματικοί όροι: трикуспидальная недостаточность, tricuspid valve annuloplasty, reverse remodeling, tricuspid insufficiency, пластика трикуспидального клапана, обратное ремоделирование

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https://doi.org/10.29001/2073-8552-2019-34-2-89-98

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