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1Academic Journal
Authors: Barkovskyi, D. Ye.
Source: Patologìâ, Vol 15, Iss 3, Pp 302-308 (2018)
Pathologia; Vol. 15 No. 3 (2018): Pathologia
Патология; Том 15 № 3 (2018): Патологія
Патологія; Том 15 № 3 (2018): ПатологіяSubject Terms: pre-eclampsia, преэклампсия, цитокины, цитокіни, імунна система, neuroendocrine system, cytokines, 3. Good health, иммунная система, immune system, 03 medical and health sciences, гомеостаз, прееклампсія, нейроендокринна система, 0302 clinical medicine, insulin resistance, інсулінорезистентність, homeostasis, Pathology, инсулино-резистентность, вагітність, RB1-214, pregnancy, нейроэндокринная система, беременность
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2Academic Journal
Authors: I. A. Khripun, S. V. Vorobyev, M. I. Kogan, И. А. Хрипун, С. В. Воробьев, М. И. Коган
Source: Medical Herald of the South of Russia; № 1 (2016); 84-86 ; Медицинский вестник Юга России; № 1 (2016); 84-86 ; 2618-7876 ; 2219-8075 ; 10.21886/2219-8075-2016-1
Subject Terms: инсулино- резистентность, men, metabolic disorders, lipid accumulation product, insulin resistance, мужчины, метаболические нарушения, продукт накопления липидов
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Relation: https://www.medicalherald.ru/jour/article/view/314/316; Аметов А.С. Избранные лекции по эндокринологии / Москва: МИА. 2012. 543с.; Canoy D. Distribution of body fat and risk of coronary heart disease in men and women // Curr. Opin. Cardiol. 2008. Vol. 23, N 6. P.591-598.; Аметов А.С., Камынина Л.Л. Проблема висцерального ожирения в диабетологии (патогенетические, клинические и эпидемиологические аспекты) // Эндокринология. 2012. № 1. С. 1-8.; Дедов И.И., Балаболкин М.И., Мамаева Г.Г., Клебанова Е.М., Креминская В.М. Инсулиновая резистентность и роль гормонов жировой ткани в развитии сахарного диабета: пособие для врачей / Эндокринологический научный центр РАМН и кафедра эндокринологии ММА им И.М. Сеченова. Москва, 2005. 88 с.; Hoube A.J., Eringa E.C., Jonk A.M. et al. Perivascular Fat and the Microcirculation: Relevance to Insulin Resistance, Diabetes, and Cardiovascular Disease // Curr. Cardiovasc. Risk Rep. 2012. Vol. 6, N 1. P. 80-90.; Reaven G.M. Insulin resistance: the link between obesity and cardiovascular disease // Med. Clin. North Am. 2011. Vol. 95, N 5. P. 875-892.; Mirmiran P., Bahadoran Z., Azizi F. Lipid accumulation product is associated with insulin resistance, lipid peroxidation, and systemic inflammation in type 2 diabetic patients // Endocrinol Metab (Seoul). 2014. Vol. 29, № 4. Р. 443-449.; https://www.medicalherald.ru/jour/article/view/314
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3Academic Journal
Source: Journal Infectology; Том 4, № 4 (2012); 46-51 ; Журнал инфектологии; Том 4, № 4 (2012); 46-51 ; 2072-6732 ; 10.22625/2072-6732-2012-4-4
Subject Terms: фактор Виллебрандта, insulin resistance, vascular cellular adhesion molecule-1, von Willebrand factor, инсулино-резистентность, молекула сосудистой клеточной адгезии-1
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Relation: https://journal.niidi.ru/jofin/article/view/132/127; Giron-Gonzalez, J.A. Serial analysis of serum and ascitic fluid levels of soluble adhesion molecules and chemokines in patients with spontaneous bacterial peritonitis / J.A.Giron- Gonzalez [et al.] // Clin. Exp. Immunol. – 2001. – V. 123, № 1. – P. 56–61.; Thomson, A.W. Circulating intercellular adhesion molecule-1 (ICAM-1) in autoimmune liver disease and evidence for the production of ICAM-1 by cytokine-stimulated human hepatocytes / A.W. Thomson [et al.] // Clin. Exp. Immunol. – 1994. – V. 95, № 1. – P. 83–90.; Tortorella, C. sICAM-1, sCD95 and sCD95L levels in chronic liver diseases of different etiology / C. Tortorella [et al.] // Immunopharmacol Immunotoxicol. – 2000. – V. 22, № 1. – P. 19–33.; de Caestecker, J.S. Bile acid therapy and markers of immune-mediated damage in primary biliary cirrhosis / J.S. de Caestecker // Eur. J. Gastroenterol. Hepatol. – 1997. – V. 9, № 2. – P. 145–147.; Granot, E. Cell adhesion molecules and hyaluronic acid as markers of inflammation, fibrosis and response to antiviral therapy in chronic hepatitis C patients / E. Granot, D. Shouval, Y. Ashur // Mediators Inflamm. – 2001. – V. 10, № 5. – P. 253–258.; Marui, A. Serum levels of soluble intercellular adhesion molecule-1 and soluble vascular cell adhesion molecule-1 in liver disease, and their changes by treatment with interferon / A. Marui [et al.] // J. Int. Med. Res. – 1996. – V. 24, № 3. – P. 258–265.; Kukla, M. sPECAM-1 and sVCAM-1: Role in Pathogenesis and Diagnosis of Chronic Hepatitis C and Association with Response to Antiviral Therapy / M. Kukla [et al.] // Therap. Adv. Gastroenterol. – 2009. – V. 2, № 2. – P. 79–90.; Петрищев, Н.Н. Типовые формы дисфункции эндотелия / Н.Н. Петрищев [и др.] // Клин. лаб. консилиум. – 2007. – № 18. – С. 31–35.; Ley, K. VCAM–1 is critical in atherosclerosis / K. Ley, Y. Huo // J. Clin. Invest. – 2001. – № 107 (10). – P. 1209–1210.; Blann, A.D. Von Willebrand factor as a marker of injury to the endothelium in inflammatory vascular disease /A.D. Blann // J. Reumatol. – 1993. – № 20. – P. 1469–1471.; Булатова, И.А. Функциональное состояние эндотелия при хроническом гепатите С / И.А. Булатова, В.В. Щёкотов, А.П. Щёкотова // РЖГГК. – 2009. – Т. 3. – С. 42–46.; Kulwas, A. Von Willebrand factor and thrombomodulin as markers of endothelial cell functions in children with chronic viral hepatitis / A. Kulwas [et al.] // Med. Wieku Rozwoj. – 2004. – V. 8, № 1. – P. 107–114.; Hui, J. Insulin resistance is associated with chronic hepatitis C and fibrosis progression / J. Hui [et al.] // Gastroenterology. – 2003. – V. 125, № 6. – P. 1695–1704.; D’Sousa, R. Insulin resistance plays a significant role in liver fibrosis in chronic hepatitis C and in the response to antiviral therapy / R. D’Sousa, A. Sabin, G. R. Foster // Am. J. Gastroenterol. – 2005. – V. 100. – P. 1509–1515.; Cersosimo, E. Insulin resistance and endothelial dysfunction: the road map to cardiovascular diseases / E. Cersosimo, R.A. DeFronzo // Diabetes Metab Res Rev. – 2006. – V. 22, № 6. – P. 423–436.; Hsueh, W.A. Insulin resistance and the endothelium /W.A. Hsueh, C.J. Lyon, M.J. Quiñones // Am, J, Med. –2004. – V. 117, № 2. – P. 109–117.; Bruno, C.M. Circulating adhesion molecules in patients with virus-related chronic diseases of the liver / C.M. Bruno [et al.] // World J. Gastroenterol. – 2005. – V. 11, № 29. – P. 4566–4569.; Lo Iacono, O. Soluble adhesion molecules correlate with liver inflammation and fibrosis in chronic hepatitis C treated with interferon-alpha / O. Lo Iacono [et al.] // Aliment Pharmacol Ther. – 1998. – V. 12, № 11. – P. 1091–1099.; Fukuda, Y. Serum levels of soluble intercellular adhesion molecule-1 and soluble vascular cell adhesion molecule-1 in asymptomatic carriers of hepatitis C virus / Y. Fukuda [et al.] // J. Int. Med. Res. – 1998. – V. 26, № 6. – P. 313–318.; La Mura, V. Von Willebrand factor levels predict clinical outcome in patients with cirrhosis and portal hypertension /V. La Mura [et al.] // Gut. – 2011. – V. 60, № 8. – P. 1133– 1138.; Ferlitsch, M. Von Willebrand factor as new noninvasive predictor of portal hypertension, decompensation and mortality in patients with liver cirrhosis / M. Ferlitsch [et al.] // Hepatology. – 2012. – V. 56, № 4. – P. 1439–1447.; https://journal.niidi.ru/jofin/article/view/132
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4Academic Journal
Authors: T. V. Antonova, M. A. Romanova, E. G. Sergeeva, Т. В. Антонова, М. А. Романова, Е. Г. Сергеева
Source: Journal Infectology; Том 3, № 3 (2011); 91-96 ; Журнал инфектологии; Том 3, № 3 (2011); 91-96 ; 2072-6732 ; 10.22625/2072-6732-2011-3-3
Subject Terms: дислипидемия, metabolic syndrome, abdominal obesity, insulin resistance, dyslipidemia, метаболический синдром, абдоминальное ожирение, инсулино- резистентность
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Relation: https://journal.niidi.ru/jofin/article/view/48/43; Poynard, T. Rates and risk factors of liver fibrosis progression in patients with chronic hepatitis C / T. Poynard [et al.] // Hepatology. – 2001. – V. 34. – P. 730–739.; Kahn, R. The metabolic syndrome: time for a critical appraisal: joint statement from the American Diabetes Association and the European Association for the Study of Diabetуs / R. Kahn [et al.] // Diabetes Care. – 2005. – V. 28. – P. 2289–2304.; Hanouneh, I.A. Клиническое значение метаболического синдрома у больных хроническим гепатитом С /I.A. Hanouneh [et al.] // Клиническая гастроэнтерология и гепатология. – 2010. – Т. 3, № 3. – С. 158–164.; Monto, A. Steatosis in chronic hepatitis C: relative contributions of obesity, diabetes mellitus, and alcohol / A. Monto [et al.] // Hepatology. – 2002 . – V. 36. – P. 729–736; Adinolfi, L.E. Steatosis accelerates the progression of liver damage of chronic hepatitis C patients and correlates with specific HCV genotype and visceral obesity / L.E. Adinolfi [et al.] // Hepatology. – 2001. – V. 33 № 6. – P. 1358–1364.; Lo Iacono, O. The impact of insulin resistance, serum adipocytokines and visceral obesity on steatosis and fibrosis in patients with chronic hepatitis C / O. Lo Iacono [et al.] //Aliment. Pharmacol. Ther. – 2007. – V. 25. – P. 1181–1191.; Kawaguchi, T. Hepatitis C virus down-regulates insulin receptor substrates 1 and 2 through up-regulation of suppressor of cytokine signaling 3 / T. Kawaguchi [et al.] //Am. J. Pathol. –2004. – V. 165. – P. 1499–1508.; Shintani, Y. Hepatitis C virus infection and diabetes: direct involvement of the virus in the development of insulin resistance / Y. Shintani [et al.] // Gastroenterology. – 2004. –V. 126. – P. 840–848.; Hotamisligil, G.S. Tumor necrosis factor alpha inhibits signaling from the insulin receptor / G.S. Hotamisligil [et al.] //Proc. Natl. Acad. Sci. USA. – 1994. – V. 91. – P. 4854–4858.; Romero-Gomez, M. Insulin resistance impairs sustained response rate to peginterferon plus ribavirin in chronic hepatitis C patients / M. Romero-Gomez [et al.] // Gastroenterology. –2005. – V. 128. – P. 636–641.; Paradis, V. High glucose and hyperinsulinemia stimulate connective tissue growth factor expression: a potential mechanism involved in progression to fibrosis in nonalcoholic steatohepatitis / V. Paradis [et al.] // Hepatology. – 2001. –V. 34. – P. 738–744.; Sanyal, A.J. Nonalcoholic steatohepatitis: association of insulin resistance and mitochondrial abnormalities / A.J. Sanyal [et al.] // Gastroenterology. – 2001. – V. 120, № 5. – P. 1183–1192.; Pirro, M. Plasma free fatty acid levels and the risk of ischemic heart disease in men: prospective results from the Québec Cardiovascular Study / M. Pirro [et al.] //Atherosclerosis. – 2002. – V. 160 № 2. – P. 377–378.; https://journal.niidi.ru/jofin/article/view/48
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5Academic Journal
Authors: Сорокiна, О. Ю.
Source: Pain, Anaesthesia and Intensive Care; № 3(72) (2015); 9-23
PAIN, ANAESTHESIA & INTENSIVE CARE; № 3(72) (2015); 9-23
Біль, знеболення та інтенсивна терапія; № 3(72) (2015); 9-23Subject Terms: стресс-индуцированная гипергликемия, инсулино-резистентность при критических состояниях, ERAS-стратегия, нутритивная поддержка, стрес-iндукована гiперглiкемiя, iнсулiнорезистентнiсть при критичних станах, ERAS-стратегiя, нутритивна пiдтримка, 616-083.98-008.9:612.122-085
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Access URL: http://jpaic.aaukr.org/article/view/84437
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6Academic Journal
Authors: Тащук, Віктор Корнійович, Хребтій, Г.І., Савчук, О.В.
Subject Terms: гіпертонічна хвороба, інсулінорезистентність, L-аргінін, ендотеліальна дисфункція, ліпідний обмін, гипертоническая болезнь, инсулино резистентность, L-аргинин, эндотелиальная дисфункция, липидный обмен, hypertension, insulin resistance, L-arginine, endothelial dysfunction, lipid metabolism
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