Εμφανίζονται 1 - 20 Αποτελέσματα από 118 για την αναζήτηση '"деформация позвоночника"', χρόνος αναζήτησης: 0,87δλ Περιορισμός αποτελεσμάτων
  1. 1
    Academic Journal

    Πηγή: Modern Rheumatology Journal; Том 19, № 2 (2025); 70-77 ; Современная ревматология; Том 19, № 2 (2025); 70-77 ; 2310-158X ; 1996-7012

    Περιγραφή αρχείου: application/pdf

    Relation: https://mrj.ima-press.net/mrj/article/view/1742/1586; Эрдес ШФ, Бадокин ВВ, Бочкова АГ и др. О терминологии спондилоартритов. Научно-практическая ревматология. 2015; 53(6):657-660.; Эрдес ШФ, Коротаева ТВ. Прогрессирование аксиального спондилоартрита. Современная ревматология. 2021; 15(3):7-14. doi:10.14412/1996-7012-2021-3-7-14; Эрдес ШФ, Бочкова АГ, Дубинина ТВ и др. Проект рабочей классификации анкилозирующего спондилита. Научно-практическая ревматология. 2013;51(6): 604-8. doi:10.14412/1995-4484-2013-604-8; Braun J, Sieper J. Ankylosing spondylitis. Lancet. 2007 Apr 21;369(9570):1379-90. doi:10.1016/S0140-6736(07)60635-7; Yang Y, Huang L, Zhao G, et al. Influence of kyphosis in ankylosing spondylitis on cardiopulmonary functions. Medicine. 2023 Oct 27;102(43):e35592. doi:10.1097/md.0000000000035592; Lorente A, Barrios C, Lorente R, et al. Severe hyperkyphosis reduces the aerobic capacity and maximal exercise tolerance in patients with Scheuermann disease. Spine J. 2019 Feb;19(2):330-8. doi:10.1016/j.spinee.2018.07.002. Epub 2018 Jul 17.; Szabo SM, Levy AR, Rao SR, et al. In creased risk of cardiovascular and cerebrovascular diseases in individuals with ankylosing spondylitis: A population-based study. Arthritis Rheum. 2011 Nov;63(11):3294-304. doi:10.1002/art.30581; Kaprove RE, Hugh Little A, Graham DC, Rosen PS. Ankylosing spondylitis: survival in men with and without radiotherapy. Arthritis Rheum. 1980 Jan;23(1):57-61. doi:10.1002/art.1780230110; Moltу A, Etcheto A, Van Der Heijde D, et al. Prevalence of comorbidities and evaluation of their screening in spondyloarthritis: results of the international cross-sectional ASAS-COMOSPA study. Ann Rheum Dis. 2016 Jun;75(6):1016-23. doi:10.1136/annrheumdis-2015-208174. Epub 2015 Oct 21.; Sari I, Okan T, Akar S, et al. Impaired endothelial function in patients with ankylo sing spondylitis. Rheumatology (Oxford). 2006 Mar;45(3):283-6. doi:10.1093/rheumatology/kei145. Epub 2005 Oct 4.; Van Eijk IC, Peters MJL, Serne EH, et al. Microvascular function is impaired in ankylosing spondylitis and improves after tumour necrosis factor alpha blockade. Ann Rheum Dis. 2009 Mar;68(3):362-6. doi:10.1136/ard.2007.086777. Epub 2008 Apr 4.; O’Leary DH, Polak JF, Kronmal RA, et al. Carotid-artery intima and media thickness as a risk factor for myocardial infarction and stroke in older adults. Cardiovascular Health Study Collaborative Research Group. N Engl J Med. 1999 Jan 7;340(1):14-22. doi:10.1056/nejm199901073400103; Ramiro S, Nikiphorou E, Sepriano A, et al. ASAS-EULAR recommendations for the management of axial spondyloarthritis: 2022 update. Ann Rheum Dis. 2023 Jan;82(1):19-34. doi:10.1136/ard-2022-223296. Epub 2022 Oct 21; Baigent C, Bhala N, Emberson J, et al. Vascular and upper gastrointestinal effects of non-steroidal anti-inflammatory drugs: metaanalyses of individual participant data from randomised trials. Lancet. 2013 Aug 31; 382(9894):769-79. doi:10.1016/S0140-6736(13)60900-9. Epub 2013 May 30; Haroon NN, Paterson JM, Li P, et al. Patients With Ankylosing Spondylitis Have Increased Cardiovascular and Cerebrovascular Mortality: A Population-Based Study. Ann Intern Med. 2015 Sep 15;163(6):409-16. doi:10.7326/M14-2470.; Bakland G, Gran JT, Nossent JC. Increased mortality in ankylosing spondylitis is related to disease activity. Ann Rheum Dis. 2011 Nov;70(11):1921-5. doi:10.1136/ard.2011.151191. Epub 2011 Jul 21.; Эрдес ШФ, Бочкова АГ, Дубинина ТВ и др. Ранняя диагностика анкилозирующего спондилита. Научно-практическая ревматология. 2013;51(4):365-367.; Bengtsson K, Forsblad-D’Elia H, Lie E, et al. Risk of cardiac rhythm disturbances and aortic regurgitation in different spondylo arthritis subtypes in comparison with general population: a register-based study from Sweden. Ann Rheum Dis. 2018 Apr;77(4):541-548. doi:10.1136/annrheumdis-2017-212189. Epub 2017 Dec 19.; Lopez-Medina C, Moltу A. Update on the epidemiology, risk factors, and disease outcomes of axial spondyloarthritis. Best Pract Res Clin Rheumatol. 2018 Apr;32(2):241-253. doi:10.1016/j.berh.2018.10.006. Epub 2018 Nov 16.; Shi LH, Lam SH, So H, et al. High inflammatory burden predicts cardiovascular events in patients with axial spondyloarthritis: a long-term follow-up study. Ther Adv Musculoskelet Dis. 2022 Sep 8:14:1759720X221122401. doi:10.1177/1759720X221122401. eCollection 2022.; Navarini L, Currado D, Marino A, et al. Persistence of C-reactive protein increased levels and high disease activity are predictors of cardiovascular disease in patients with axial spondyloarthritis. Sci Rep. 2022 May 7;12(1): 7498. doi:10.1038/s41598-022-11640-8.; Поддубный ДА, Ребров АП. Кардиоваскулярный риск у больных анкилозирующим спондилитом: роль системного воспаления и дисфункции эндотелия. Рациональная фармакотерапия в кардиологии. 2008;4(5):71-76.; Mattar Valente RL, Mattar Valente J, Werner de Castro GT, et al. Subclinical athero sclerosis in ankylosing spondylitis: is there a role for inflammation? Rev Bras Reumatol. 2013 Sep-Oct;53(5):377-81.; Ozdowska P, Kowalik I, Sadowski K, et al. Patterns of dyslipidemia in young patients with seronegative spondyloarthropathies without cardiovascular. Reumatologia. 2021;59(5): 285-291. doi:10.5114/reum.2021.110610. Epub 2021 Nov 7.; Liew JW, Reveille JD, Castillo M, et al. Cardiovascular risk scores in axial spondyloarthritis versus the general population: A cross-sectional study. J Rheumatol. 2021 Mar;48(3):361-366. doi:10.3899/jrheum.200188. Epub 2020 Jul 1.; Zhang J, Qi J, Li Y, et al. Association between type 1 diabetes mellitus and ankylosing spondylitis: a two-sample Mendelian randomi zation study. Front Immunol. 2023 Dec 20:14: 1289104. doi:10.3389/fimmu.2023.1289104. eCollection 2023.; American Diabetes Association Professional Practice Committee. 2. Classification and Diagnosis of Diabetes: Standards of Medi cal Care in Diabetes-2022. Diabetes Care. 2022 Jan 1;45(Suppl 1):S17-S38. doi:10.2337/dc22-S002.; Maas F, Arends S, Van Der Veer E, et al. Obesity Is Common in Axial Spondyloarthritis and Is Associated with Poor Clinical Outcome. J Rheumatol. 2016 Feb;43(2):383-7. doi:10.3899/jrheum.150648. Epub 2015 Dec 15.; Chen CH, Chen HA, Liu CH, et al. Association of obesity with inflammation, disease severity and cardiovascular risk factors among patients with ankylosing spondylitis. Int J Rheum Dis. 2020 Aug;23(9):1165-1174. doi:10.1111/1756-185X.13912. Epub 2020 Aug 23.; Liew JW, Gianfrancesco MA, Heckbert SR, Gensler LS. Relationship Between Body Mass Index, Disease Activity, and Exercise in Ankylosing Spondylitis. Arthritis Care Res (Hoboken). 2022 Aug;74(8):1287-1293. doi:10.1002/acr.24565. Epub 2022 Apr 26.; Kesikburun B, Eksioglu E, Cakci A. Meta bolic Syndrome in Rheumatoid Arthritis and Ankylosing Spondylitis Romatoid Artrit ve Ankilozan Spondilitli Hastalarda Metabolik Sendrom. Ankara Med J. 2018;(2):198-206. doi: 1017098/amj.435258; Bayartai ME, Luomajoki H, Tringali G, et al. Differences in spinal posture and mobility between adults with obesity and normal weight individuals. Sci Rep. 2023 Aug 17;13(1): 13409. doi:10.1038/s41598-023-40470-5.; Arima H, Togawa D, Hasegawa T, et al. Hypertension Is Related to Positive Global Sagittal Alignment: A Cross-Sectional Cohort Study. Asian Spine J. 2019 Dec 31;13(6): 895-903. doi:10.31616/asj.2018.0308. Epub 2019 Jul 9.; Almasi S, Farahani B, Samiei N, et al. Echocardiographic and Electrocardiographic Findings in Patients with Ankylosing Spondylitis without Cardiovascular Risk Factors. J Tehran Heart Cent. 2020 Apr;15(2): 43-49. doi:10.18502/jthc.v15i2.4182.; Yuan Y, Yang J, Zhang X, et al. Carotid Intima-Media Thickness in Patients with Ankylosing Spondylitis: A Systematic Review and Updated Meta-Analysis. J Atheroscler Thromb. 2019 Mar 1;26(3):260-71. doi:10.5551/jat.45294. Epub 2018 Aug 8.

  2. 2
  3. 3
    Academic Journal

    Πηγή: PAEDIATRIC SURGERY. UKRAINE; № 4(69) (2020): Paediatric Surgery. Ukraine; 67-71
    ХИРУРГИЯ ДЕТСКОГО ВОЗРАСТА; № 4(69) (2020): Хирургия детского возраста; 67-71
    Хірургія дитячого віку; № 4(69) (2020): Хірургія дитячого віку; 67-71

    Περιγραφή αρχείου: application/pdf

  4. 4
    Academic Journal

    Συνεισφορές: Not specified, Отсутствует

    Πηγή: Current Pediatrics; Том 21, № 6S (2022); 577-582 ; Вопросы современной педиатрии; Том 21, № 6S (2022); 577-582 ; 1682-5535 ; 1682-5527

    Περιγραφή αρχείου: application/pdf

    Relation: https://vsp.spr-journal.ru/jour/article/view/3088/1256; Ockerman PA, Lund MD. A generalised storage disorder resembling Hurler’s syndrome. Lancet. 1967;290(7509):239–241. doi: https://doi.org/10.1016/S0140-6736(67)92303-3; Ockerman PA, Autio S, Norden NE. Diagnosis of mannosidosis. Lancet. 1973;301(7796):207–208. doi: https://doi.org/10.1016/s0140-6736(73)90045-7; Malm D, Nilssen Ø. Alpha-Mannosidosis. October 11, 2001 [Updated July 18, 2019]. In: GeneReviews® [Internet]. Adam MP, Ardinger HH, Pagon RA, et al., eds. Seattle (WA): University of Washington, Seattle; 1993–2021. Available online: https://www.ncbi.nlm.nih.gov/books/NBK1396. Accessed on December 12, 2022.; Beck M, Olsen KJ, Wraith JE, et al. Natural history of alphamannosidosis a longitudinal study. Orphan J Rare Dis. 2013;8:88. doi: https://doi.org/10.1186/1750-1172-8-88; Malm D, Nilssen Ø. Alpha-mannosidosis. Orphan J Rare Dis. 2008;3(1):21. doi: https://doi.org/10.1186/1750-1172-3-21; Poupetová H, Ledvinová J, Berná L, et al. The birth prevalence of lysosmal storage disorders in the Czech republic: Comparison with data in different populations. J Inherit Metab Dis. 2010;33(4): 387–396. doi: https://doi.org/10.1007/s10545-010-9093-7; Menendez-Sainz C, Gonzalez-Quevedo A, Gonzalez-Garcia S, et al. High proportion of mannosidosis and fucosidosis among lysosomal storage diseases in Cuba. Genet Mol Res. 2012; 11(3):2352–2359. doi: https://doi.org/10.4238/2012; Riise Stensland HM, Klenow HB, Van Nguyen L, et al. Identification of 83 novel alpha-mannosidosis-associated sequence variants: Functional analysis of MAN2B1 missense mutations. Hum Mutat. 2012;33(3):511–520. doi: https://doi.org/10.1002/humu.22005; Paciotti S, Codini M, Tasegian A, et al. Lysosomal alphamannosidase and alpha-mannosidosis. Front Biosci (Landmark Ed). 2017;22(1):157–167. doi: https://doi.org/10.2741/4478; Noll RB, Netzloff ML, Kulkarni R. Long-term follow-up of biochemical and cognitive functioning in patients with mannosidosis. Arch Neurol. 1989;46(5):507–509. doi: https://doi.org/10.1001/archneur.1989.00520410041020; Ara JR, Mayayo E, Marzo ME, et al. Neurological impairment in alpha-mannosidosis: a longitudinal clinical and MRI study of a brother and sister. Childs Nerv Syst. 1999;15(8):369–371. doi: https://doi.org/10.1007/s003810050416; Santoro L, Zampini L, Padella L, et al. Early biochemical effects of velmanase alfa in a 7-month-old infant with alpha-mannosidosis. JIMD Rep. 2020;55(1):15–21. doi: https://doi.org/10.1002/jmd2.12144; Aylsworth AS, Taylor HA, Stuart CM, Thomas GH. Mannosidosis: phenotype of a severely affected child and characterization of alphamannosidase activity in cultured fibroblasts from the patient and his parents. J Pediatr. 1976;88(5):814–818. doi: https://doi.org/10.1016/s0022-3476(76)81120-1; Malm D, Halvorsen DS, Tranebjaerg L, Sjursen H. Immunodeficiency in alpha-mannosidosis: a matched case-control study on immunoglobulins, complement factors, receptor density, phagocytosis and intracellular killing in leucocytes. Eur J Pediatr. 2000;159(9):699–703. doi: https://doi.org/10.1007/s004310000545; Matlach J, Zindel T, Amraoui Y, et al. Retinal and optic nerve degeneration in α-mannosidosis. Orphanet J Rare Dis. 2018;13(1):88. doi: https://doi.org/10.1186/s13023-018-0829-z; Autio S, Louhimo T, Helenius M. The clinical course of mannosidosis. Ann Clin Res. 1982;14(2):93–97.; Guffona N, Tylki-Szymanska A, Borgwardtc L, et al. Recognition of alpha-mannosidosis in paediatric and adult patients: Presentation of a diagnostic algorithm from an international working group. Mol Genet Metab. 2019;126(4):470–474. doi: https://doi.org/10.1016/j.ymgme.2019.01.024; Chester MA, Lundblad A, Öckerman PA, Autio S. Mannosidosis. In: Genetic Errors of Glycoprotein Metabolism. Durand P, O’Brian J, eds. Milan, Italy: Edi-Ermes; 1982. pp. 89–120.; Hennermann JB, Raebe EM, Donà F, et al. Mortality in patients with alpha-mannosidosis: a review of patients’ data and the literature. Orphanet J Rare Dis. 2022;17(1):287. doi: https://doi.org/10.1186/s13023-022-02422-6

  5. 5
    Academic Journal

    Πηγή: PAEDIATRIC SURGERY. UKRAINE; No. 1(74) (2022): Paediatric Surgery (Ukraine); 34-39
    ХИРУРГИЯ ДЕТСКОГО ВОЗРАСТА; № 1(74) (2022): Хирургия детского возраста; 34-39
    Хірургія дитячого віку; № 1(74) (2022): Хірургія дитячого віку (Україна); 34-39

    Περιγραφή αρχείου: application/pdf

    Σύνδεσμος πρόσβασης: http://psu.med-expert.com.ua/article/view/254960

  6. 6
  7. 7
    Academic Journal

    Συνεισφορές: Бюджетное финансирование осуществлялось в рамках выполнения поискового экспериментального исследования на тему: «Создание экспериментальной модели идиопатического сколиоза на курином эмбрионе путем ингибирования PAX3 гена липофильными интерферирующими siРНК».

    Πηγή: Acta Biomedica Scientifica; Том 6, № 3 (2021); 193-208 ; 2587-9596 ; 2541-9420

    Περιγραφή αρχείου: application/pdf

    Relation: https://www.actabiomedica.ru/jour/article/view/2868/2181; Kouwenhoven JW, Castelein R. The Pathogenesis of Adolescent Idiopathic Scoliosis: Review of the Literature. Spine. 2008; 33 (26): 2898–2908. doi:10.1097/BRS.0b013e3181891751; Gorman KF, Julien C, Moreau A. The genetic epidemiology of idiopathic scoliosis. Eur. Spine J. 2012; 21 (10): 1905–1919. doi:10.1007/s00586-012-2389-6; Kotwicki T, Durmala J, Czaprowski D, Glowacki M, Kolban M, Snela S, et al. Conservative management of idiopathic scoliosis – guidelines based on SOSORT 2006 consensus. Ortop Traumatol Rehabil. 2009; 11 (5): 379–395; Tarrant RC, Queally JM, Moore DP, Kiely PJ. Prevalence and impact of low body mass index on outcomes in patients with adolescent idiopathic scoliosis: a systematic review. Eur J Clin Nutr. 2018; 72 (11): 1463–1484. doi:10.1038/s41430-018-0095-0; Normand E, Francoa A, Marcil V. Nutrition and physical activity level of adolescents with idiopathic scoliosis: a narrative review. The Spine Journal. 2020; 20 (5): 785–799. doi.org/10.1016/j.spinee.2019.11.012; Yagci G, Karatel M, Yakut Y. Body Awareness and its Relation to Quality of Life in Individuals with Idiopathic Scoliosis. Percept Mot Skills. 2020; 127 (5): 841–857.; Dunn J, Henrikson NB, Morrison CC, Blasi PR, Nguyen M, Lin JS. USPSTF Evidence Review: Screening for Adolescent Idiopathic Scoliosis. JAMA. 2018; 319 (2): 173–187. doi:10.1001/jama.2017.11669; Sharma S, Gao X, Londono D, Devroy SE, Mauldin KN, Frankel JT, et al. Genome-wide association studies of adolescent idiopathic scoliosis suggest candidate susceptibility genes. Hum. Mol. Genet. 2011; 20 (7): 1456–1466. doi:10.1093/hmg/ddq571; Fadzan M, Bettany-Saltikov J. Etiological theories of adolescent idiopathic scoliosis: Past and Present. The Open Orthopaedics Journal. 2017; 11 (9): 1466–1489. doi:10.2174/1874325001711011466; Lonstein JE. Scoliosis: surgical versus nonsurgical treatment. Clin Orthop. 2006; (443): 248–259. doi:10.1097/01.blo.0000198725.54891.73; Negrini S, Aulisa AG, Aulisa L, Circo AB, de Mauroy JC, Durmala J, et al. SOSORT guidelines: Orthopaedic and rehabilitation treatment of idiopathic scoliosis during growth. Scoliosis. 2012; 7 (3): 3. doi.org/10.1186/1748-7161-7-3; Зайдман А. М. Что же такое идиопатический сколиоз? Хирургия позвоночника. 2016; 13 (4): 104–110. doi.org/10.14531/ss2016.4.104–110; Trobisch P, Suess O, Schwab F. Idiopathic Scoliosis. Dtsch Arztebl Int. 2010; 107 (49): 875–883. doi:10.3238/arztebl.2010.0875; Чаклин В. Д. От эксперимента к клинической хирургии и ортопедии позвоночника. Ортопедия, травматология и протезирование. 1962; (5): 3–8; Михайловский М. В., Садовой М. А., Новиков В. В., Васюра А. С., Садовая Т. Н., Удалова И. Г. Современная концепция раннего выявления и лечения идиопатического сколиоза. Хирургия позвоночника. 2015; 12 (3): 13–18. doi.org/10.14531/ss2015.3.13–18; Perez-Machado G, Berenguer-Pascual E, Bovea-Marco M, Rubio-Belmar PA, Garcia-Lopez E, Garzon MJ, et al. From genetics to epigenetics to unravel the etiology of adolescent idiopathic scoliosis. Bone. 2020; (140): 115563. doi.org/10.1016/j.bone.2020.115563; Good CR. The Genetic Basis of Adolescent Idiopathic Scoliosis. Journal of The Spinal Research Foundation. 2009; 4 (1): 13–17. Доступно на: https://studylib.net/doc/8650381/thegenetic-basis-of-adolescent-idiopathic-scoliosis. [Дата доступа: 01.05.2021]; Di Felice F, Zaina F, Donzelli S, Negrini S. The Natural History of Idiopathic Scoliosis During Growth. A Meta-Analysis. American Journal of Physical Medicine & Rehabilitation. 2018; 97 (5): 346–356. doi:10.1097/PHM.0000000000000861; Jada A, Mackel CE, Hwang SW, Samdani AF, Stephen JH, Bennett JT, et al. Evaluation and management of adolescent idiopathic scoliosis: A review. Neurosurgical Focus. 2017; 43 (4): Е2 doi.org/10.3171/2017.7.FOCUS17297; Perry M, Starkweather A, Baumbauer K, Young E. Factors Leading to Persistent Postsurgical Pain in Adolescents Undergoing Spinal Fusion: An Integrative Literature Review. Journal of Pediatric Nursing. 2018; (38): 74–80. doi.org/10.1016/j.pedn.2017.10.013; Seki H, Ideno S, Ishihara T, Watanabe K, Matsumoto M, Morisaki H. Postoperative pain management in patients undergoing posterior spinal fusion for adolescent idiopathic scoliosis: a narrative review. eCollection. 2018; 12 (13): 17. doi:10.1186/s13013-018-0165-z; Menger RP, Sin AH. Adolescent and Idiopathic Scoliosis. [Updated 2020 Aug 21]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2021 Jan-.Доступно на: https://pubmed.ncbi.nlm.nih.gov/29763083/. [Дата доступа: 01.05.2021]; James JI, Lloyd-Roberts GC, Pilcher MF. Infantile structural scoliosis. J Bone Joint Surg Br. 1959; (41B): 719–735. doi:10.1302/0301-620X.41B4.719; Spencer GS, Zorab PA. Spinal muscle in scoliosis. Comparison of normal and scoliotic rabbits. J Neurol Sci. 1976; 30 (2–3): 405–410. doi.org/10.1016/0022-510X(76)90143-X; Yarom R, Robin GC. Studies of spinal and peripheral muscles from patients with scoliosis. Spine. 1979; 4 (1): 12–21. doi:10.1097/00007632-197901000-00003; Zetterberg C, Aniansson A, Grimby G. Morphology of the paravertebral muscles in adolescent idiopathic scoliosis. Spine. 1983; 8 (5): 457–462. doi:10.1097/00007632-198307000-00003; Казьмин А. И., Кон И. И., Беленький В. Е. Сколиоз: монография. Медицина; 1981: 272; Miller NH, Mims B, Child A, Milewicz DM, Sponseller P, Blanton SH. Genetic analysis of structural elastic fiber and collagen genes in familial adolescent idiopathic scoliosis. J Orthop Res. 1996; 14 (6): 994–999. doi:10.1002/jor.1100140621; Shi L, Wang D, Chu WCW, Burwell RG, Freeman BJC, Heng PA, et al. Volume-based morphometry of brain MR images in adolescent idiopathic scoliosis and healthy control subjects. AJNR Am J Neuroradiol. 2009; 30 (7): 1302–1307. doi:10.3174/ajnr.A1577; Burwell RG, Freeman BJC, Dangerfield PH, Aujla RK, Cole AA, Kirby AS, et al. Etiologic theories of idiopathic scoliosis: neurodevelopmental concept of maturational delay of the CNS body schema (“body-in-the-brain”). Stud Health Technol Inform. 2006; (123): 72–79; Beaulieu M, Toulotte C, Gatto L, Rivard CH, Teasdale N, Simoneau M, et al. Postural imbalance in non-treated adolescent idiopathic scoliosis at different periods of progression. Eur Spine J. 2009; 18 (1): 38–44. doi:10.1007/s00586-008-0831-6; Hitier M, Hamon M, Denise P, Lacoudre J, Thenint MA, Mallet JF, et al. PLoS One. 2015; 10 (7): e0131120. doi:10.1371/journal.pone.0131120; Hawasli AH, Hullar TE, Dorward IG. Idiopathic scoliosis and the vestibular system. European spine journal: Official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society. 2015; 24 (2): 227–233. doi.org/10.1007/s00586-014-3701-4; Porter RW. The pathogenesis of idiopathic scoliosis: Uncoupled neuro-osseous growth? Eur Spine J. 2001; 10 (6): 473–481. doi:10.1007/s005860100311; Burwell RG, Dangerfield PH, Freeman BJ. Concepts on the pathogenesis of adolescent idiopathic scoliosis. Bone growth and mass, vertebral column, spinal cord, brain, skull, extra-spinal left-right skeletal length asymmetries, disproportions and molecular pathogenesis. Stud Health Technol Inform. 2008; (135); –52; Дудин М. Г., Михайловский М. В., Садовой М. А., Пинчук Д. Ю., Фомичев Н. Г. Идиопатический сколиоз: кто виноват и что делать? Хирургия позвоночника. 2014; (2): 8–20. doi.org/10.14531/ss2014.2.8–20; Дудин М. Г., Пинчук Д. Ю., Михайловский М. В. Сколиоз: вопросы и ответы. Учебное пособие. Спб.: Изд-во СЗГМУ им. И. И. Мечникова; 2019: 124; Гайворонский И. В. Нормальная анатомия человека. Учебник для ВУЗов в 2-х томах, 8-е издание. СПб.: СпецЛит; 2013: (1): 567, (2): 456; Chu WCW, Man GCW, Lam WWM, Yeung BHY, Chau WW, Ng BKW, Lam TP, Lee K, et al. Morphological and functional electrophysiological evidence of relative spinal cord tethering in adolescent idiopathic scoliosis. Spine. 2008; 33 (6): 673–680. doi.org/10.1097/BRS.0b013e318166aa58; Stokes IA. Mechanical effects on skeletal growth. J Musculoskelet Neuronal Interact. 2002; 2 (3): 277–280; Stokes IA, Burwell RG, Dangerfield PH. Biomechanical spinal growth modulation and progressive adolescent scoliosis--a test of the ‘vicious cycle’ pathogenetic hypothesis: Summary of an electronic focus group debate of the IBSE. Scoliosis. 2006; (1): 16. doi.org/10.1186/1748-7161-1-16; Smit TH. Adolescent idiopathic scoliosis: The mechanobiology of differential growth. JOR Spine. 2020; 3 (4): e1115. doi.org/10.1002/jsp2.1115; Castelein RM, Pasha S, Cheng JC, Dubousset J. Idiopathic Scoliosis as a Rotatory Decompensation of the Spine. J Bone Miner Res. 2020; 35 (10): 1850–1857. doi:10.1002/jbmr.4137; Burwell RG, Clark EM, Dangerfield PH, Moulton A. Adolescent idiopathic scoliosis (AIS): A multifactorial cascade concept for pathogenesis and embryonic origin. Scoliosis Spinal Disord. 2016; 11: 8. doi.org/10.1186/s13013-016-0063-1; Liu Z, Tam EMS, Sun GQ, Lam TP, Zhu ZZ, Sun X, et al. Abnormal leptin bioavailability in adolescent idiopathic scoliosis: An important new finding. Spine. 2012; 37 (7): 599–604. doi.org/10.1097/BRS.0b013e318227dd0c; Liang G, Gao W, Liang A, Ye W, Peng Y, Zhang L, et al. Normal leptin expression, lower adipogenic ability, decreased leptin receptor and hyposensitivity to leptin in adolescent idiopathic scoliosis. PLoS One. 2012; 7 (5): e36648. doi:10.1371/journal.pone.0036648; Zhou S, Qiu XS, Zhu ZZ, Wu WF, Liu Z, Qiu Y. A singlenucleotide polymorphism rs708567 in the IL-17RC gene is associated with a susceptibility to and the curve severity of adolescent idiopathic scoliosis in a Chinese Han population: a case-control study. BMC Musculoskeletal Disorders. 2012; (13): 181. doi:10.1186/1471-2474-13-181; Fendri K, Patten SA, Kaufman GN, Zaouter C, Parent S, Grimard G, et al. Microarray expression profiling identifies genes with altered expression in Adolescent Idiopathic Scoliosis. Eur Spine J. 2013; 22 (6): 1300–1311. doi:10.1007/s00586-013-2728-2; Latalski M, Danielewicz-Bromberek A, Fatyga M, Latalska M, Kröber M, Zwolak P. Current insights into the aetiology of adolescent idiopathic scoliosis. Arch Orthop Trauma Surg. 2017; (137): 1327–1333. doi:10.1007/s00402-017-2756-1; Carr AJ. Adolescent idiopathic scoliosis in identical twins. J Bone Joint Surg Br. 1990; 72 (6): 1077. doi:10.1302/0301-620X.72B6.2246294; Riseborough EJ, Wynne-Davies RA. A genetic survey of idiopathic scoliosis in Boston, Massachusetts. J Bone Joint Surg Am. 1973; 55 (5): 974–982; Harrington PR. The etiology of idiopathic scoliosis. Clin Orthop Relat Res. 1977; 126: 17–25; Roaf R. The treatment of progressive scoliosis by unilateral growth-arrest. J Bone Jt Surg [Br]. 1963; 45 (4): 637–651; Ogilvie JW, Braun J, Argyle V, Nelson L, Meade M; Ward K. The search for idiopathic scoliosis genes. Spine. 2006; 31 (6): 679–681. doi:10.1097/01.brs.0000202527.25356.90; Grauers A, Rahman I, Gerdhem P. Heritability of scoliosis. Eur Spine J. 2012; 21 (6): 1069–1074. doi.org/10.1007/s00586-011-2074-1; Watanabe K, Michikawa T, Yonezawa I, Takaso M, Minami S, Soshi S, et al. Physical activities and lifestyle factors related to adolescent idiopathic scoliosis. J Bone Joint Surg Am. 2017; 99 (4): 284–94. doi.org/10.2106/JBJS.16.00459; Morocz M, Czibula A, Grozer ZB, Szecsenyi A, Almos PZ, Rasko I, et al. Association study of BMP4, IL6, Leptin, MMP3, and MTNR1B gene promoter polymorphisms and adolescent idiopathic scoliosis. Spine (Phila Pa 1976). 2011; 36 (2): 123–130. doi:10.1097/BRS.0b013e318a511b0e; Zhao D, Qiu GX, Wang YP, Zhang JG, Shen JX, Wu ZH. Association between adolescent idiopathic scoliosis with double curve and polymorphisms of calmodulin1 gene/estrogen receptor-a gene. Orthop Surg. 2009; 1 (3): 222–230. doi:10.1111/j.1757–7861.2009.00038. x; Eun IS, Park WW, Suh KT, Kim JI, Lee JS. Association between osteoprotegerin gene polymorphism and bone mineral density in patients with adolescent idiopathic scoliosis. Eur Spine J. 2009; 18 (12): 1936–1940. doi:10.1007/s00586-009-1145-z; Nowak R, Szota J, Mazurek U. Vitamin D receptor gene (VDR) transcripts in bone, cartilage, muscles and blood and microarray analysis of vitamin D responsive genes expression in paravertebral muscles of juvenile and adolescent idiopathic scoliosis patients. BMC Musculoskelet Disord. 2012; (13): 259. doi:10.1186/1471-2474-13-259; Yin X, Wang H, Guo J, Zhang L, Zhang Y, Li L, et al. Association of vitamin D receptor BsmI rs1544410 and ApaI rs7975232 polymorphisms with susceptibility to adolescent idiopathic scoliosis. A systematic review and metaanalysis. Medicine. 2018; 97:2 (e9627). doi.org/10.1097/MD.0000000000009627; Takahashi Y, Matsumoto M, Karasugi T, Watanabe K, Chiba K, Kawakami N, Tsuji T, Uno K, et al. Lack of association between adolescent idiopathic scoliosis and previously reported single nucleotide polymorphisms in MATN1, MTNR1B, TPH1, and IGF1 in a Japanese population. J Orthop Res. 2011; 29 (7): 1055–1058; McGregor TL, Gurnett CA, Dobbs MB, Wise CA, Morcuende JA, Morgan TM, et al. Common polymorphisms in human lysyl oxidase genes are not associated with the adolescent idiopathic scoliosis phenotype. BMC Med Genet. 2011; (12): 92. doi:10.1186/1471-2350-12-92; Qiu XS, Tang NL, Yeung HY, Qiu Y, Cheng JC. Association study between adolescent idiopathic scoliosis and the DPP9 gene which is located in the candidate region identified by linkage analysis. Postgrad Med J. 2008; 84 (995): 498–501. doi:10.1136/pgmj.2007.066639; Liu Z, Tang NL, Cao XB, Liu WJ, Qiu XS, Cheng JC, et al. Lack of association between the promoter polymorphisms of MMP-3 and IL-6 genes and adolescent idiopathic scoliosis: a casecontrol study in a Chinese Han population. Spine (Phila Pa 1976). 2010; 35 (18): 1701–1705. doi:10.1097/BRS.0b013e3181c6ba13; Jiang J, Qian B, Mao S, Zhao Q, Qiu X, Liu Z, et al. A promoter polymorphism of tissue inhibitor of metalloproteinase-2 gene is associated with severity of thoracic adolescent idiopathic scoliosis. Spine (Phila Pa 1976). 2012; 37 (1): 41–47. doi:10.1097/BRS.0b013e31820e71e3; Inoue M, Minami S, Nakata Y, Takaso M, Otsuka Y, Kitahara H, et al. Prediction of curve progression in idiopathic scoliosis from gene polymorphic analysis. Stud Health Technol Inform. 2002; (91): 90–96; Wu J, Qiu Y, Zhang L, Sun Q, Qiu X, He Y. Association of estrogen receptor gene polymorphisms with susceptibility to adolescent idiopathic scoliosis. Spine (Phila Pa 1976). 2006; 31 (10): 1131–1136. doi:10.1097/01.brs.0000216603.91330.6f; Takahashi Y, Matsumoto M, Karasugi T, Watanabe K, Chiba K, Kawakami N, et al. Replication study of the association between adolescent idiopathic scoliosis and two estrogen receptor genes. J Orthop Res. 2010; 29 (6): 834–837. doi:10.1002/jor.21322; Peng Y, Liang G, Pei Y, Ye W, Liang A, Su P. Genomic polymorphisms of G-Protein Estrogen Receptor 1 are associated with severity of adolescent idiopathic scoliosis. Int Orthop. 2011; doi:10.1007/s00264-011-1374-8; Yeung HY, Tang NL, Lee KM, Ng BK, Hung VW, Kwok R, et al. Genetic association study of insulin-like growth factor-I (IGF-I) gene with curve severity and osteopenia in adolescent idiopathic scoliosis. Stud Health Technol Inform. 2006; (123): 18–24; Yang Y, Wu Z, Zhao T, Wang H, Zhao D, Zhang J, et al. Adolescent idiopathic scoliosis and the singlenucleotide polymorphism of the growth hormone receptor and IGF-1 genes. Orthopedics. 2009; 32 (6): 411. doi:10.3928/01477447-20090511-08; Machida M, Dubousset J, Imamura Y, Iwaya T, Yamada T, Kimura J. Role of melatonin deficiency in the development of scoliosis in pinealectomized chickens. J Bone Joint Surg Br. 1995; 77 (1): 134–138; Girardo M, Bettini N, Dema E, Cervellati S. The role of melatonin in the pathogenesis of adolescent idiopathic scoliosis (AIS). Eur Spine J. 2011; 20 (1): 68–74. doi. 10.1007/s00586-011-1750-5; Nelson LM, Ward K, Ogilvie JW. Genetic variants in melatonin synthesis and signaling pathway are not associated with adolescent idiopathic scoliosis. Spine (Phila Pa 1976). 2011; 36 (1): 37–40. doi:10.1097/BRS.0b013e3181e8755b; Shyy W, Wang K, Gurnett CA, Dobbs MB, Miller NH, Wise C, et al. Evaluation of GPR50, hMel-1B, and ROR- alpha melatonin-related receptors and the etiology of adolescent idiopathic scoliosis. J Pediatr Orthop. 2010; 30 (6): 539–543. doi:10.1097/BPO.0b013e3181e7902c; Kou I, Otomo N, Takeda K, Momozawa Y, Lu HF, Kubo M, et al. Genome-wide association study identifies; previously unreported susceptibility loci for adolescent idiopathic scoliosis in Japanese. Nat Commun. 2019; 10 (3685). doi.org/10.1038/s41467-019-11596-w; Grauers A, Wang J, Einarsdottir E, Simony A, Danielsson A, Akesson K, et al. Candidate gene analysis and exome sequencing confirm LBX1 as a susceptibility gene for idiopathic scoliosis. Spine J. 2015; 15 (10): 2239–2246. doi:10.1016/j.spinee.2015.05.013; Wang W, Ma J, Li SY, Wu X, Hu B, Wang X, et al. Advance on genetic mechanism of adolescent idiopathic scoliosis and genetic relationship map. Zhongguo Gu Shang. 2015; 28 (9): 854–860; Maqsood А, Frome DK, Gibly RF, Larson JE, Patel NM, Sarwark JF. IS (Idiopathic Scoliosis) etiology: Multifactorial genetic research continues. A systematic review 1950 to 2017. Journal of Orthopaedics. 2020; (21): 421–426. doi.org/10.1016/j.jor.2020.08.005; Wang S, Qiu Y, Ma Z, Xia C, Zhu F, Zhu Z. Expression of Runx2 and Type X Collagen in Vertebral Growth Plate of Patients with Adolescent Idiopathic Scoliosis. Journal Connective Tissue Research. 2010; 51 (3): 188–196. doi.org/10.3109/03008200903215590; Zaydman AM, Strokova EL, Stepanova AO, Laktionov PP, Shevchenko AI, Subbotin VM. A New Look at Causal Factors of Idiopathic Scoliosis: Altered Expression of Genes Controlling Chondroitin Sulfate Sulfation and Corresponding Changes in Protein Synthesis in Vertebral Body Growth Plates. International Journal of Medical Sciences. 2019; 16 (2): 221–230. doi:10.7150/ijms.29312; Gorman KF, Breden F. Teleosts as models for human vertebral stability and deformity. Comp Biochem Physiol C Toxicol Pharmacol. 2007; 145 (1): 28–38. doi:10.1016/j. cbpc.2006.10.004; Bobyn JD, Little DG, Gray R, Schindeler A. Animal models of scoliosis. J Orthop Res. 2015; 33 (4): 458–67.; Guo L, Yamashita H, Kou I, Takimoto A, Meguro-Horike M, Horike S, et al. Functional investigation of a non-coding variant associated with adolescent idiopathic scoliosis in zebrafish: Elevated expression of the ladybird homeobox gene causes body axis deformation. PLoS Genet. 2016; 12 (1): e1005802. doi.org/10.1371/journal.pgen.1005802; Zaydman AM, Strokova EL, Pahomova NY, Gusev AF, Mikhaylovskiy MV, Shevchenko AI, et al. Etiopathogenesis of adolescent idiopathic scoliosis: Review of the literature and new epigenetic hypothesis on altered neural crest cells migration in early embryogenesis as the key event. Medical Hypotheses. 2021; 151 (110585). doi.org/10.1016/j.mehy.2021.110585; Zaydman AM, Strokova EL, Kiseleva EV, Suldina LA, Strunov AA, Shevchenko AI, et al. A New Look at Etiological Factors of Idiopathic Scoliosis: Neural Crest Cells. International Journal of Medical Sciences. 2018; 15 (5): 436–446. doi:10.7150/ijms.22894; Lowe TG, Edgar M, Margulies JY, Miller NH, Raso VJ, Reinker KA, et al. Current concepts review: etiology of idiopathic scoliosis: current trends in research. J Bone Joint Surg Am. 2000; 82 (8): 1157–1168. doi:10.2106/00004623-200008000-00014; Negrini S, Donzelli S, Aulisa AG, Czaprowski D, Schreiber S, de Mauroy JC, et al. 2016 SOSORT guidelines: Orthopaedic and rehabilitation treatment of idiopathic scoliosis during growth. Scoliosis and Spinal Disorders. 2018; 13 (1): doi.org/10.1186/s13013-017-0145-8; Ward K, Ogilvie J, Argyle V, Nelson L, Meade M, Braun J, et al. Polygenic inheritance of adolescent idiopathic scoliosis: A study of extended families in Utah. Am J Med Genet A. 2010; 152 A (50): 1178–1188. doi.org/10.1002/ajmg.a.33145; Silahtaroglu A, Stenvang J. MicroRNAs, epigenetics and disease. Essays Biochem. 2010; 48 (1): 165–185. doi.org/10.1042/bse0480165; Aguilera O, Fernández AF, Muñoz A, Fraga MF. Epigenetics and environment: A complex relationship. J Appl Physiol. 2010; 109 (1): 243–251. doi.org/10.1152/japplphysiol.00068.2010; Braun JT. Twelve DNA markers accurately assess risk of progression in adolescent idiopathic scoliosis. Scoliosis. 2007; 2 (1): 41. doi:10.1186/1748-7161-2-S1-S41; Danielsson AJ, Hasserius R, Ohlih A, Nachemson AL. A prospective study of brace treatment versus observation alone in adolescent idiopathic scoliosis: A follow-up mean of 16 years after maturity. Spine. 2007; 32 (20): 2198–2207. doi:10.1097/BRS.0b013e31814b851f; Roye BD, Wright ML, Williams BA, Matsumoto H, Corona J, Hyman JE, et al. Does ScoliScore provide more information than traditional clinical estimates of curve progression? Spine. 2012; 37 (25): 2099–2103. doi:10.1097/BRS.0b013e31825eb605; Mo F, Cunningham ME. Pediatric scoliosis. Curr Rev Musculoskelet Med. 2011; 4 (4): 175–182. doi:10.1007/s12178-011-9100-0; https://www.actabiomedica.ru/jour/article/view/2868

  8. 8
    Academic Journal

    Πηγή: PAEDIATRIC SURGERY. UKRAINE; No. 4(73) (2021): Paediatric surgery. Ukraine; 66-71
    ХИРУРГИЯ ДЕТСКОГО ВОЗРАСТА; № 4(73) (2021): Хирургия детского возраста; 66-71
    Хірургія дитячого віку; № 4(73) (2021): Хірургія дитячого віку; 66-71

    Περιγραφή αρχείου: application/pdf

    Σύνδεσμος πρόσβασης: http://psu.med-expert.com.ua/article/view/252138

  9. 9
    Academic Journal

    Πηγή: Хірургія дитячого віку; № 3(72) (2021): Хірургія дитячого віку; 10-14
    PAEDIATRIC SURGERY. UKRAINE; No. 3(72) (2021): Paediatric surgery. Ukraine; 10-14
    ХИРУРГИЯ ДЕТСКОГО ВОЗРАСТА; № 3(72) (2021): Хирургия детского возраста; 10-14

    Περιγραφή αρχείου: application/pdf

    Σύνδεσμος πρόσβασης: http://psu.med-expert.com.ua/article/view/245123

  10. 10
  11. 11
    Academic Journal

    Πηγή: Traumatology and Orthopedics of Russia; Vol 26, No 4 (2020); 45-55 ; Травматология и ортопедия России; Vol 26, No 4 (2020); 45-55 ; 2542-0933 ; 2311-2905 ; 10.21823/2311-2905-2020-26-4

    Περιγραφή αρχείου: application/pdf

  12. 12
    Academic Journal

    Περιγραφή αρχείου: application/pdf

    Relation: Белая, Е. В. Ассоциация полиморфных вариантов генов VDR и LCT с риском развития сколиотической деформации позвоночника у детей школьного возраста / Е. В. Белая, М. Д. Амельянович, Е. И. Корнеева // Веснік Віцебскага дзяржаўнага ўніверсітэта імя П. М. Машэрава. – 2024. – № 3. – С. 20–26.; b2b7f92f3ff3d11034e9c652fed68d80; https://rep.vsu.by/handle/123456789/44295

    Διαθεσιμότητα: https://rep.vsu.by/handle/123456789/44295

  13. 13
  14. 14
  15. 15
  16. 16
  17. 17
  18. 18
  19. 19
  20. 20