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1Academic Journal
Συγγραφείς: O. N. Titova, O. N. Kuzubova, E. S. Lebedeva, E. A. Surkova, T. N. Preobrazhenskaya, I. V. Dvorakovskaya, О. Н. Титова, Н. А. Кузубова, Е. С. Лебедева, Е. А. Суркова, Т. Н. Преображенская, И. В. Двораковская
Πηγή: PULMONOLOGIYA; Том 28, № 2 (2018); 169-176 ; Пульмонология; Том 28, № 2 (2018); 169-176 ; 2541-9617 ; 0869-0189 ; 10.18093/0869-0189-2018-28-2
Θεματικοί όροι: целекоксиб, inflammation, hypoxia-inducible factor-1α, cyclooxygenase-2, interleukin-17, surfactant protein D, celecoxib, воспаление, гипоксия-индуцибельный фактор-1, циклооксигеназа-2, интерлейкин-17, сурфактантный протеин D
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Relation: https://journal.pulmonology.ru/pulm/article/view/982/818; Clerici C., Planès C. Gene regulation in the adaptive process to hypoxia in lung epithelial cells. Am. J. Physiol. Lung Cell Mol. Physiol. 2009; 296 (3): 267–274. DOI:10.1152/ajplung.90528.2008.; Rey S., Semenza G.L. Hypoxia-inducible factor-1-dependent mechanisms of vascularization and vascular remodelling. Cardiovasc. Res. 2010; 86 (2): 236–242. DOI:10.1093/cvr/cvq045.; Lee S.H., Lee S.H., Kim C.H. et al. Increased expression of vascular endothelial growth factor and hypoxia inducible factor-1α in lung tissue of patients with chronic bronchitis. Clin. Biochem. 2014; 47 (7-8): 552–559. DOI:10.1016/j.clinbiochem.2014.01.012.; Hoenderdos K., Lodge K.M., Hirs R.A. et al. Hypoxia upregulates neutrophil degranulation and potential for tissue injury. Thorax. 2016; 71 (11): 1030–1038. DOI:10.1136/thoraxjnl-2015-207604.; Polke M., Seiler F., Lepper P.M. et al. Hypoxia and the hypoxia-regulated transcription factor HIF-1α suppress the host defence of airway epithelial cells. J. Innate Immun. 2017; 23 (4): 373–380. DOI:10.1177/1753425917698032.; Ito Y., Ahmad A., Kewley E. et al. Hypoxia-inducible factor regulates expression of surfactant protein in alveolar type II cells in vitro. Am. J. Respir. Cell Mol. Biol. 2011; 45 (5): 938–945. DOI:10.1165/rcmb.2011-0052OC.; Zhong H., Willard M., Simons J. NS398 reduces hypoxia-inducible factor (HIF)-1alpha and HIF-1 activity: multiple-level effects involving cyclooxygenase-2 dependent and independent mechanisms. Int. J. Cancer. 2004; 112 (4): 585–595. DOI:10.1002/ijc.20438.; Bruning U., Fitzpatrick S.F., Frank T. et al. NFκB and HIF display synergistic behaviour during hypoxic inflammation. Cell. Mol. Life Sci. 2012; 69 (8): 1319–1329. DOI:10.1007/s00018-011-0876-2.; Лебедева Е.С., Кузубова Н.А., Данилов Л.Н. и др. Воспроизведение в эксперименте хронической обструктивной болезни легких. Бюллетень экспериментальной биологии и медицины. 2012; 152 (5): 659–663. DOI:10.1007/s10517-012-1601-3.; Gao W., Li L., Wang Y. et al. Bronchial epithelial cells: The key effector cells in the pathogenesis of chronic obstructive pulmonary disease? Respirology. 2015; 20 (5): 722–729. DOI:10.1111/resp.12542.; Yanagisawa H., Hashimoto M., Minagawa S. et al. Role of IL-17A in murine models of COPD airway disease. Am. J. Phisiol. Lung Cell Mol. Physiol. 2017; 312 (1): 122–130. DOI:10.1152/ajplung.00301.2016.; Лямина С.В., Малышев И.Ю. Сурфактантный белок D в норме и при заболеваниях легких. Российский медицинский журнал. 2012; (1): 50–55.; Levänen B., Glader P., Dahlén B. et al. Impact of tobacco smoking on cytokine signaling via interleukin-17A in the peripheral airways. Int. J. Chron. Obstruct. Pulmon. Dis. 2016; 11: 2109–2116. DOI:10.2147/COPD.S99900.; Pappu R., Rutz S., Ouyang W. Regulation of epithelial immunity by IL-17 family cytokines. Trends Immunol. 2012; 33 (7): 343–349. DOI:10.1016/j.it.2012.02.008.; Duan M.C., Zhang J.Q., Liang Y. et al. Infiltration of IL-17-producing T cells and Treg cells in a mouse model of smoke-induced emphysema. Inflammation. 2016; 39 (4): 1334–1344. DOI:10.1007/s10753-016-0365-8.; Yadava K., Bollyky P., Lawson M.A. The formation and function of tertiary lymphoid follicles in chronic pulmonary inflammation. Immunology. 2016; 149 (3): 262–269. DOI:10.1111/imm.12649.; Chen X., Cao J., Chen B.Y. [Interleukin-17 expression and clinical significance in the lung tissue of patients with stable chronic obstructive pulmonary disease]. Zhonghua Yi Xue Za Zhi. 2016; 96 (26): 2086–2090. DOI:10.3760/cma.j.issn.0376-2491.2016.26.010 (in Chinese).; Roos A.B., Sethi S., Nikota J. et al. IL-17A and the promotion of neutrophilia in acute exacerbation of chronic obstructive pulmonary disease. Am. J. Respir. Crit. Care Med. 2015; 192 (4): 428–437. DOI:10.1164/rccm.201409-1689OC.; Zhang M., Fei X., Zhang G.Q. et al. Role of neutralizing anti-murine interleukin-17A monoclonal antibody on chronic ozone-induced airway inflammation in mice. Biomed. Pharmacother. 2016; 83: 247–256. DOI:10.1016/j.biopha.2016.06.041.; Xu F., Xu Z., Zhang R. et al. Nontypeable Haemophilus influenzae induces COX-2 and PGE2 expression in lung epithelial cells via activation of p38 MAPK and NF-kappa B. Respir. Res. 2008; 9: 16. DOI:10.1186/1465-9921-9-16.; Zago M., Rico de Souza A., Hecht E. et al. The NF-κB family member RelB regulates microRNA miR-146a to suppress cigarette smoke-induced COX-2 protein expression in lung fibroblasts. Toxicol. Lett. 2014; 226 (2): 107–116. DOI:10.1016/j.toxlet.2014.01.020.; Gu W., Song L., Li X.M. et al. Mesenchymal stem cells alleviate airway inflammation and emphysema in COPD through down-regulation of cyclooxygenase-2 via p38 and ERK MAPK pathways. Sci. Rep. 2015; 5: 8733. DOI:10.1038/srep08733.; Sheridan J.A., Zago M., Nair P. et al. Decreased expression of the NF-κB family member RelB in lung fibroblasts from Smokers with and without COPD potentiates cigarette smoke-induced COX-2 expression. Respir. Res. 2015; 16: 54. DOI:10.1186/s12931-015-0214-6.; He X.Y., Shi X.Y., Yuan H.B. et al. Propofol attenuates hypoxia-induced apoptosis in alveolar epithelial type II cells through down-regulating hypoxia-inducible factor-1α. Injury. 2012; 43 (3): 279–283. DOI:10.1016/j.injury.2011.05.037.; Jiang H., Zhu Y.S., Xu H. et al. Inflammatory stimulation and hypoxia cooperatively activate HIF-1{alpha} in bronchial epithelial cells: involvement of PI3K and NF-{kappa}B. Am. J. Physiol. Lung Cell. Mol. Physiol. 2010; 298 (5): 660–669. DOI:10.1152/ajplung.00394.2009.; Lu D., Li N., Yao X., Zhou L. Potential inflammatory markers in obstructive sleep apnea-hypopnea syndrome. Bosn. J. Basic. Med. Sci. 2017; 17 (1): 47–53. DOI:10.17305/bjbms.2016.1579.; Huang M., Wang L., Chen J. et al. Regulation of COX-2 expression and epithelial-to-mesenchymal transition by hypoxia-inducible factor-1α is associated with poor prognosis in hepatocellular carcinoma patients post TACE surgery. Int. J. Oncol. 2016; 48 (5): 2144–2154. DOI:10.3892/ijo.2016.3421.; Eurlings I.M., Reynaert N.L., van den Beucken T. et al. Cigarette smoke extract induces a phenotypic shift in epithelial cells; involvement of HIF1α in mesenchymal transition. PLoS One. 2014; 9 (10): e107757. DOI:10.1371/journal.pone.0107757.; Li H., Wu Q., Xu L. et al. Increased oxidative stress and disrupted small intestinal tight junctions in cigarette smoke-exposed rats. Mol. Med. Rep. 2015; 11 (6): 4639–4644. DOI:10.3892/mmr.2015.3234.; Liu X.H., Kirschenbaum A., Lu M. et al. Prostaglandin E2 induces hypoxia-inducible factor-1alpha stabilization and nuclear localization in a human prostate cancer cell line. J. Biol. Chem. 2002; 277 (51): 50081–50086. DOI:10.1074/jbc.M201095200.; Roh G.S., Yi C.O., Cho Y.J. et al. Anti-inflammatory effects of celecoxib in rat lungs with smoke-induced emphysema. Am. J. Physiol. Lung Cell. Mol. Physiol. 2010; 299 (2): 184–191. DOI:10.1152/ajplung.00303.2009.; https://journal.pulmonology.ru/pulm/article/view/982
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2Academic Journal
Συγγραφείς: Шилова, Наталия, Чаша, Татьяна, Попова, Ирина, Хорошилова, Анна
Θεματικοί όροι: ГЛУБОКОНЕДОНОШЕННЫЕ ДЕТИ, ДЫХАТЕЛЬНЫЕ РАССТРОЙСТВА, СУРФАКТАНТНЫЙ ПРОТЕИН D, БЕЛОК КЛЕТОК КЛАРА
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3Academic Journal
Πηγή: Российский педиатрический журнал.
Θεματικοί όροι: 03 medical and health sciences, 0302 clinical medicine, ГЛУБОКОНЕДОНОШЕННЫЕ ДЕТИ, ДЫХАТЕЛЬНЫЕ РАССТРОЙСТВА, СУРФАКТАНТНЫЙ ПРОТЕИН D, БЕЛОК КЛЕТОК КЛАРА, 3. Good health
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