Εμφανίζονται 1 - 20 Αποτελέσματα από 131 για την αναζήτηση '"НЕВРОЛОГИЧЕСКИЕ ЗАБОЛЕВАНИЯ"', χρόνος αναζήτησης: 0,83δλ Περιορισμός αποτελεσμάτων
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    Academic Journal

    Πηγή: One Health & Risk Management ; Vol. 6 No. 4 (2025) ; Vol 6 Nr 4 (2025) ; Том 6 № 4 (2025) ; 2587-3466 ; 2587-3458 ; 10.38045/ohrm.2025.6(4)

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    Academic Journal

    Θέμα γεωγραφικό: USPU

    Relation: Специальное образование. 2022. № 4 (68)

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    Academic Journal

    Πηγή: Psychological and Pedagogical Support of General, Special and Inclusive Education of Children and Adults; 152-154 ; Психолого-педагогическое сопровождение общего, специального и инклюзивного образования детей и взрослых; 152-154

    Περιγραφή αρχείου: text/html

    Relation: info:eu-repo/semantics/altIdentifier/isbn/978-5-907688-23-0; https://phsreda.com/e-articles/10482/Action10482-105899.pdf; Архипова Е.Ф. Коррекционно-логопедическая работа по преодолению стертой дизартрии у детей / Е.Ф. Архипова. ‒ М.: Астрель, 2008. ‒ 72 с.; Власенко И.Т. Методы обследования речи детей. Выявление и преодоление речевых нарушений в дошкольном возрасте / И.Т. Власенко, Г.В. Чиркина, И.Ю. Кондратенко. ‒ М.: Айрис-пресс, 2005. ‒ 177 с.; Волкова Г.А. Методика психолого-логопедического обследования детей с нарушениями речи. Вопросы дифференциальной диагностики / Г.А. Волкова. ‒ СПб., 2005. ‒ 144 с.; Гаркуша Ю.Ф. Коррекционно-педагогическая работа в дошкольных учреждениях для детей с нарушениями речи / Ю.Ф. Гаркуша. ‒ М.: Владос, 2000. ‒ 158 с.; Гвоздев А.Н. Вопросы изучения детской речи / А.Н. Гвоздев. ‒ М.: АПИ РСФСР, 1961. ‒ 471 с.; Грибова О.Е. Технология организации логопедического обследования / О.Е. Грибова. ‒ М.: АИРИШ, 2008. ‒ 96 с.; Степанова О.А. Дошкольная логопедическая служба / О.А. Степанова. ‒ М.: Сфера, 2006. ‒ 190 с.; Шаховская Е.И. Обследование детей с нарушением речи в условиях медико-педагогических комиссий / Е.И. Шаховская. ‒ М.: МГПИ, 1978. – С. 4–16.; https://phsreda.com/article/105899/discussion_platform

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    Academic Journal

    Συνεισφορές: The work was supported by a grant of the Russian Science Foundation (project No. 23-21-00154 “Development of methods for predicting the properties of pharmacological preparations based on their molecular structure using the theory of topological analysis of chemographs”), FRC “Computer Science and Control”, RAS., Работа выполнена при поддержке гранта Российского научного фонда (проект № 23-21-00154 «Разработка методов прогноза свойств фармакологических препаратов по их молекулярной структуре с помощью теории топологического анализа хемографов»), ФИЦ ИУ РАН.

    Πηγή: FARMAKOEKONOMIKA. Modern Pharmacoeconomics and Pharmacoepidemiology; Vol 16, No 3 (2023); 466-480 ; ФАРМАКОЭКОНОМИКА. Современная фармакоэкономика и фармакоэпидемиология; Vol 16, No 3 (2023); 466-480 ; 2070-4933 ; 2070-4909

    Περιγραφή αρχείου: application/pdf

    Relation: https://www.pharmacoeconomics.ru/jour/article/view/891/494; Shindo Y., Witt E., Han D., et al. Enzymic and non-enzymic antioxidants in epidermis and dermis of human skin. J Invest Dermatol. 1994; 102 (1): 122–4. https://doi.org/10.1111/1523-1747.ep12371744.; Garrido-Maraver J., Cordero M.D., Oropesa-Avila M., et al. Clinical applications of coenzyme Q10. Front Biosci (Landmark Ed). 2014; 19 (4): 619–33. https://doi.org/10.2741/4231.; Торшин И.Ю., Громова О.А. Альтернативные подходы к коррекции гиперхолестеринемии: эффекты стандартизированных экстрактов красного риса и его синергистов. Лечебное дело. 2021; 1: 89–98. https://doi.org/10.24412/2071-5315-2021-12283.; Белова О.В., Арефьева Т.И., Москвина С.Н. Иммуновоспалительные аспекты болезни Паркинсона. Журнал неврологии и психиатрии им. С.С. Корсакова. 2020; 120 (2): 110–9. https://doi.org/10.17116/jnevro2020120021110.; Ghorbani S., Yong V.W. The extracellular matrix as modifier of neuroinflammation and remyelination in multiple sclerosis. Brain. 2021; 144 (7): 1958–73. https://doi.org/10.1093/brain/awab059.; Torshin I.Yu., Rudakov K.V. On metric spaces arising during formalization of recognition and classification problems. Part 1: Properties of compactness. Pattern Recognit Image Anal. 2016; 26 (2): 274–84. https://doi.org/10.1134/S1054661816020255.; Torshin I.Yu., Rudakov K.V. Combinatorial analysis of the solvability properties of the problems of recognition and completeness of algorithmic models. Part 2: Metric approach within the framework of the theory of classification of feature values. Pattern Recognit Image Anal. 2017; 27 (2): 184–99. https://doi.org/10.1134/S1054661817020110.; Torshin I.Yu., Rudakov K.V. On metric spaces arising during formalization of problems of recognition and classification. Part 2: Density properties. Pattern Recognit Image Anal. 2016; 26 (3): 483–96. https://doi.org/10.1134/S1054661816030202.; Hajiluian G., Heshmati J., Jafari Karegar S., et al. Diabetes, age, and duration of supplementation subgroup analysis for the effect of coenzyme Q10 on oxidative stress: a systematic review and metaanalysis. Complement Med Res. 2021; 28 (6): 557–70. https://doi.org/10.1159/000515249.; Shimizu K., Kon M., Tanimura Y., et al. Coenzyme Q10 supplementation downregulates the increase of monocytes expressing tolllike receptor 4 in response to 6-day intensive training in kendo athletes. Appl Physiol Nutr Metab. 2015; 40 (6): 575–81. https://doi.org/10.1139/apnm-2014-0556.; Aslani Z., Shab-Bidar S., Fatahi S., Djafarian K. Effect of coenzyme Q10 supplementation on serum of high sensitivity c-reactive protein level in patients with cardiovascular diseases: a systematic review and meta-analysis of randomized controlled trials. Int J Prev Med. 2018; 9: 82. https://doi.org/10.4103/ijpvm.IJPVM_263_17.; Farsi F., Heshmati J., Keshtkar A., et al. Can coenzyme Q10 supplementation effectively reduce human tumor necrosis factor-α and interleukin-6 levels in chronic inflammatory diseases? A systematic review and meta-analysis of randomized controlled trials. Pharmacol Res. 2019; 148: 104290. https://doi.org/10.1016/j.phrs.2019.104290.; Fan L., Feng Y., Chen G.C., et al. Effects of coenzyme Q10 supplementation on inflammatory markers: a systematic review and metaanalysis of randomized controlled trials. Pharmacol Res. 2017; 119: 128–36. https://doi.org/10.1016/j.phrs.2017.01.032.; Rasoolzadeh E.A., Shidfar F., Rasoolzadeh R.A., Hezaveh Z.S. The effect of coenzyme Q10 on periodontitis: a systematic review and metaanalysis of clinical trials. J Evid Based Dent Pract. 2022; 22 (2): 101710. https://doi.org/10.1016/j.jebdp.2022.101710.; Liu Z., Tian Z., Zhao D., et al. Effects of coenzyme Q10 supplementation on lipid profiles in adults: a meta-analysis of randomized controlled trials. J Clin Endocrinol Metab. 2022; 108 (1): 232–49. https://doi.org/10.1210/clinem/dgac585.; Al Saadi T., Assaf Y., Farwati M., et al. Coenzyme Q10 for heart failure. Cochrane Database Syst Rev. 2021; 2 (2): CD008684. https://doi.org/10.1002/14651858.CD008684.pub3.; Qu H., Guo M., Chai H., et al. Effects of coenzyme Q10 on statininduced myopathy: an updated meta-analysis of randomized controlled trials. J Am Heart Assoc. 2018; 7 (19): e009835. https://doi.org/10.1161/JAHA.118.009835.; Sun I.O., Jin L., Jin J., et al. The effects of addition of coenzyme Q10 to metformin on sirolimus-induced diabetes mellitus. Korean J Intern Med. 2019; 34 (2): 365–74. https://doi.org/10.3904/kjim.2017.004.; Moradi M., Haghighatdoost F., Feizi A., et al. Effect of coenzyme Q10 supplementation on diabetes biomarkers: a systematic review and meta-analysis of randomized controlled clinical trials. Arch Iran Med. 2016; 19 (8): 588–96.; Liang Y., Zhao D., Ji Q., et al. Effects of coenzyme Q10 supplementation on glycemic control: a GRADE-assessed systematic review and dose-response meta-analysis of randomized controlled trials. EClinicalMedicine. 2022; 52: 101602. https://doi.org/10.1016/j.eclinm.2022.101602.; Izadi A., Ebrahimi S., Shirazi S., et al. Hormonal and metabolic effects of coenzyme Q10 and/or vitamin E in patients with polycystic ovary syndrome. J Clin Endocrinol Metab. 2019; 104 (2): 319–27. https://doi.org/10.1210/jc.2018-01221.; Taghizadeh S., Izadi A., Shirazi S., et al. The effect of coenzyme Q10 supplementation on inflammatory and endothelial dysfunction markers in overweight/obese polycystic ovary syndrome patients. Gynecol Endocrinol. 2021; 37 (1): 26–30. https://doi.org/10.1080/09513590.2020.1779689.; Zhang T., He Q., Xiu H., et al. Efficacy and safety of coenzyme Q10 supplementation in the treatment of polycystic ovary syndrome: a systematic review and meta-analysis. Reprod Sci. 2023; 30 (4): 1033– 48. https://doi.org/10.1007/s43032-022-01038-2.; Chen K., Chen X., Xue H., et al. Coenzyme Q10 attenuates high-fat diet-induced non-alcoholic fatty liver disease through activation of the AMPK pathway. Food Funct. 2019; 10 (2): 814–23. https://doi.org/10.1039/c8fo01236a.; Jiang Y.J., Jin J., Nan Q.Y., et al. Coenzyme Q10 attenuates renal fibrosis by inhibiting RIP1-RIP3-MLKL-mediated necroinflammation via Wnt3α/β-catenin/GSK-3β signaling in unilateral ureteral obstruction. Int Immunopharmacol. 2022; 108: 108868. https://doi.org/10.1016/j.intimp.2022.108868.; Alehagen U., Aaseth J., Alexander J., et al. Selenium and coenzyme Q10 supplementation improves renal function in elderly deficient in selenium: observational results and results from a subgroup analysis of a prospective randomised double-blind placebo-controlled trial. Nutrients. 2020; 12 (12): 3780. https://doi.org/10.3390/nu12123780.; Zahed N.S., Ghassami M., Nikbakht H. Effects of coenzyme Q10 supplementation on C-reactive protein and homocysteine as the inflammatory markers in hemodialysis patients; a randomized clinical trial. J Nephropathol. 2016; 5 (1): 38–43. https://doi.org/10.15171/jnp.2016.07.; Drovandi S., Lipska-Ziętkiewicz B.S., Ozaltin F., et al. Oral Coenzyme Q10 supplementation leads to better preservation of kidney function in steroid-resistant nephrotic syndrome due to primary Coenzyme Q10 deficiency. Kidney Int. 2022; 102 (3): 604–12. https://doi.org/10.1016/j.kint.2022.04.029.; Bakhshayeshkaram M., Lankarani K.B., Mirhosseini N., et al. The effects of coenzyme Q10 supplementation on metabolic profiles of patients with chronic kidney disease: a systematic review and metaanalysis of randomized controlled trials. Curr Pharm Des. 2018; 24 (31): 3710–23. https://doi.org/10.2174/1381612824666181112112857.; Orsucci D., Mancuso M., Ienco E.C., et al. Targeting mitochondrial dysfunction and neurodegeneration by means of coenzyme Q10 and its analogues. Curr Med Chem. 2011; 18 (26): 4053–64. https://doi.org/10.2174/092986711796957257.; Yang X., Zhang Y., Xu H., et al. Neuroprotection of coenzyme Q10 in neurodegenerative diseases. Curr Top Med Chem. 2016; 16 (8): 858–66. https://doi.org/10.2174/1568026615666150827095252.; Shinkai T., Nakashima M., Ohmori O., et al. Coenzyme Q10 improves psychiatric symptoms in adult-onset mitochondrial myopathy, encephalopathy, lactic acidosis and stroke-like episodes: a case report. Aust N Z J Psychiatry. 2000; 34 (6): 1034–5. https://doi.org/10.1080/000486700286.; Chang Y., Huang S.K., Wang S.J. Coenzyme Q10 inhibits the release of glutamate in rat cerebrocortical nerve terminals by suppression of voltage-dependent calcium influx and mitogen-activated protein kinase signaling pathway. J Agric Food Chem. 2012; 60 (48): 11909–18. https://doi.org/10.1021/jf302875k.; Lee D., Shim M.S., Kim K.Y., et al. Coenzyme Q10 inhibits glutamate excitotoxicity and oxidative stress-mediated mitochondrial alteration in a mouse model of glaucoma. Invest Ophthalmol Vis Sci. 2014; 55 (2): 993–1005. https://doi.org/10.1167/iovs.13-12564.; Lu C.J., Guo Y.Z., Zhang Y., et al. Coenzyme Q10 ameliorates cerebral ischemia reperfusion injury in hyperglycemic rats. Pathol Res Pract. 2017; 213 (9): 1191–9. https://doi.org/10.1016/j.prp.2017.06.005.; Ibrahim Fouad G. Combination of omega 3 and coenzyme Q10 exerts neuroprotective potential against hypercholesterolemia-induced Alzheimer's-like disease in rats. Neurochem Res. 2020; 45 (5): 1142– 55. https://doi.org/10.1007/s11064-020-02996-2.; Omidi G., Karimi S.A., Shahidi S., et al. Coenzyme Q10 supplementation reverses diabetes-related impairments in long-term potentiation induction in hippocampal dentate gyrus granular cells: an in vivo study. Brain Res. 2020; 1726: 146475. https://doi.org/10.1016/j.brainres.2019.146475.; Shi T.J., Zhang M.D., Zeberg H., et al. Coenzyme Q10 prevents peripheral neuropathy and attenuates neuron loss in the db-/dbmouse, a type 2 diabetes model. Proc Natl Acad Sci U S A. 2013; 110 (2): 690– 5. https://doi.org/10.1073/pnas.1220794110.; Sadeghiyan Galeshkalami N., Abdollahi M., Najafi R., et al. Alphalipoic acid and coenzyme Q10 combination ameliorates experimental diabetic neuropathy by modulating oxidative stress and apoptosis. Life Sci. 2019; 216: 101–10. https://doi.org/10.1016/j.lfs.2018.10.055.; Kandhare A.D., Ghosh P., Ghule A.E., Bodhankar S.L. Elucidation of molecular mechanism involved in neuroprotective effect of coenzyme Q10 in alcohol-induced neuropathic pain. Fundam Clin Pharmacol. 2013; 27 (6): 603–22. https://doi.org/10.1111/fcp.12003.; Jiménez-Jiménez F.J., Alonso-Navarro H., García-Martín E., Agúndez J.A.G. Coenzyme Q10 and Parkinsonian syndromes: a systematic review. J Pers Med. 2022; 12 (6): 975. https://doi.org/10.3390/jpm12060975.; Liu J., Wang L.N., Zhan S.Y., Xia Y. Coenzyme Q10 for Parkinson's disease. Cochrane Database Syst Rev. 2012; 5: CD008150. https://doi.org/10.1002/14651858.CD008150.pub3.; Markley H.G. Coenzyme Q10 and riboflavin: the mitochondrial connection. Headache. 2012; 52 (Suppl. 2): 81–7. https://doi.org/10.1111/j.1526-4610.2012.02233.x.; Sazali S., Badrin S., Norhayati M.N., Idris N.S. Coenzyme Q10 supplementation for prophylaxis in adult patients with migraine-a metaanalysis. BMJ Open. 2021; 11 (1): e039358. https://doi.org/10.1136/bmjopen-2020-039358.; Maguire Á., Hargreaves A., Gill M. Coenzyme Q10 and neuropsychiatric and neurological disorders: relevance for schizophrenia. Nutr Neurosci. 2020; 23 (10): 756–69. https://doi.org/10.1080/1028415X.2018.1556481.; https://www.pharmacoeconomics.ru/jour/article/view/891

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    Academic Journal

    Συνεισφορές: Исследование выполнено при финансовой поддержке Министерства науки и высшего образования Российской Федерации (Соглашение № 075-15-2020-901 от 13.11.2020).

    Πηγή: Journal Infectology; Том 14, № 2 (2022); 65-72 ; Журнал инфектологии; Том 14, № 2 (2022); 65-72 ; 2072-6732 ; 10.22625/2072-6732-2022-14-2

    Περιγραφή αρχείου: application/pdf

    Relation: https://journal.niidi.ru/jofin/article/view/1354/974; Center for Systems Science and Engineering // COVID-19 content portal. URL:www.systems.jhu.edu/research/public-health/ncov/ (дата обращения: 09.05.2022). Saad I. COVID-19: breaking down a global health crisis / Saad I. [et al.] // Ann Clin Microbiol Antimicrob. – 2021. Vol.20, №35.; Galea M. Neurological manifestations and pathogenic mechanisms of COVID-19 / Galea M. [et al.] // Neurological Research. – 2022. – Р. 1-12.; Лобзин, Ю.В. COVID-19-ассоциированный педиатрический мультисистемный воспалительный синдром / Ю.В. Лобзин [и др.] // Медицина экстремальных ситуаций. – 2021. – № 2. – С. 13–19.; Mahboubi M. Neurological complications associated with Covid-19; molecular mechanisms and therapeutic approaches / Mahboubi M. [et al.] // // Reviews in Medical Virology. – 2022. – Р. e2334.; Manzano G. Acute disseminated encephalomyelitis and acute hemorrhagic leukoencephalitis following COVID-19: systematic review and meta-synthesis / Manzano G. [et al.] // Neurology Neuroimmunology Neuroinflammation. – 2021. Vol.8, №6. – Р. e1080.; Ariño H. Neuroimmune disorders in COVID-19 / Ariño H. [et al.] // Journal of Neurology. – 2022. – Р. 1-13.; Molina А.Е. SARS-CoV-2, a new causative agent of Guillain-Barré syndrome? / Molina А.Е. [et al.] // Med Intensiva. – 2022. – Vol.46, № 2. – Р. 110-111. Abu-Rumeileh S. Guillain–Barré syndrome spectrum associated with COVID-19: an up-to-date systematic review of 73 cases / Abu-Rumeileh S. [et al.] // Journal of neurology. 2021. – Vol.268, № 4. – Р. 1133-1170.; Dalakas M.C. Guillain-Barré syndrome: The first documented COVID-19–triggered autoimmune neurologic disease: More to come with myositis in the offing / Dalakas M.C. // Neurology Neuroimmunology Neuroinflammation. – 2020. – Vol.7, №5.; Seyede M. Guillain-Barré/Miller Fisher overlap syndrome in a patient after coronavirus disease-2019 infection: a case report / Seyede M. [et al.] // J Med Case Rep. – 2022. – Vol.16,№1.; Keddie S. Epidemiological and cohort study finds no association between COVID-19 and Guillain-Barré syndrome / Keddie S. // Brain. – 2021. – Vol.144,№2. – Р. 682-693.; Sriwastava S. Guillain Barré Syndrome and its variants as a manifestation of COVID-19: A systematic review of case reports and case series / Sriwastava S. [et al.] // Journal of the neurological sciences. – 2021. – Vol. 15, №420. – Р. 117263.; Laved A. Neurological associations of SARS-cov-2 infection: a systematic review / Laved A. // CNS Neurol Disord Drug Targets. – 2022. – Vol. 21, №3. – P. 246-258.; Mohammad А. Guillain Barre Syndrome as a Complication of COVID-19: A Systematic Review / Mohammad А. [et al.] // Can J Neurol Sci. – 2022. – №1. – P. 1-11.; Finsterer J. Guillain-Barré syndrome is immunogenic in SARS-CoV-2 infected / Finsterer J. [et al.] // J Med Virol. 2022. – Vol. 94, №1. – P. 22-23.; Marie I. Intravenous immunoglobulin-associated arterial and venous thrombosis; report of a series and review of the literature / Marie I. [et al.] // British Journal of Dermatology. 2006. – №4. – Р. 714-721.; Hoepner R. Is COVID-19 severity associated with reduction in T lymphocytes in anti-CD20-treated people with multiple sclerosis or neuromyelitis optica spectrum disorder? / Hoepner R. [et al.] // CNS Neurosci Ther. – 2022. – Vol.28, №6. – P.971-973.; Xia H. Evasion of type I interferon by SARS-CoV-2 / Xia H. [et al.] // Cell reports. – 2020. – Vol. 33, №1. – Р. 108234.; Sormani M. DMTs and Covid-19 severity in MS: a pooled analysis from Italy and France / Sormani M. [et al.] // Annals of Clinical and Translational Neurology. – 2021. – Vol.8, №8. Р. 1738-1744.; Finsterer J. SARS-CoV-2 triggered relapse of multiple sclerosis / Finsterer J. // Clin Neurol Neurosurg. – 2022. №215. – Р.207-210.; Alroughani R. Prevalence, severity, outcomes, and risk factors of COVID-19 in multiple sclerosis: an observational study in the Middle East / Alroughani R. [et al.] // J Clin Neurosci. – 2022. – №99. – P. 311-316.; Wang Y. SARS-CoV-2-associated acute disseminated encephalomyelitis: a systematic review of the literature / Wang Y. [et al.] // Journal of Neurology. – 2021. – Р.1-22.; Wang C. Assessment and management of acute disseminated encephalomyelitis (ADEM) in the pediatric patient / Wang C. // Pediatric Drugs. – 2021. – Vol.23(3);213-221.; Esmaeili S. Acute disseminated encephalitis (ADEM) as the first presentation of COVID-19; a case report / Esmaeili S. [et al.] // Ann Med Surg (Lond). – 2022. – №77. – P. 103511.; Gilhus N.E. Myasthenia gravis: subgroup classification and therapeutic strategies / Gilhus N.E., Verschuuren J. // Lancet Neurol. 2015; Vol.14, №10. – Р. 1023-36.; Baig A.M. Evidence of the COVID-19 virus targeting the CNS: tissue distribution, host-virus interaction, and proposed neurotropic mechanisms / Baig A.M. [et al.] // ACS Chem Neurosci. – 2020. – Vol.11, №7. – Р.995-998.; Liu R. Expansion of regulatory T cells via IL-2/anti-IL-2 mAb complexes suppresses experimental myasthenia / Liu R. [et al.] // Eur J Immunol. – 2010. – Vol.40, №6. – Р. 1577-89.; Thiruppathi M. Impaired regulatory function in circulating CD4(+)CD25(high)CD127(low/-) T cells in patients with myasthenia gravis / Thiruppathi M. [et al.] // Clin Immunol. – 2012. Vol.145, №3. – Р. 209-2.; Gunes H. What chances do children have against COVID-19? Is the answer hidden within the thymus? / Gunes H. [et al.] // European journal of pediatrics. – 2021. – Vol.180, №3. – Р. 983-986.; Wang W. High-dimensional immune profiling by mass cytometry revealed immunosuppression and dysfunction of immunity in COVID-19 patients / Wang W., Su B., Pang L, Qiao L. // Cell Mol Immunol. – 2020. – Vol.17, №6. – Р. 650-652.; Quin C. Dysregulation of immune response in patients with coronavirus 2019 (COVID-19) in Wuhan, China / Quin C., Zhou L., Hu Z. // Clin Infect Dis. – 2020. – Vol.71, №15. – Р. 762-768.; Muir R. Innate lymphoid cells are the predominant source of IL-17A during the early pathogenesis of acute respiratory distress syndrome / Muir R., Osbourn M., Dubois A.V. // Am J Respir Crit Care Med. – 2016. – Vol.193, №4. – Р. 407-16.; Sriwastava S. New onset of ocular myasthenia gravis in a patient with COVID-19: a novel case report and literature review / Sriwastava S., Tandon M., Kataria S. // J Neurol. – 2021. – Vol.268, №8. – Р. 2690-2696.; Brossard-Barbosa N. Seropositive ocular myasthenia gravis developing shortly after COVID-19 infection: report and review of the literature / Brossard-Barbosa N. [et al.] // J Neuroophthalmol. – 2022.; Алексеева, Т.М. Дебют генерализованной миастении после перенесенной новой коронавирусной инфекции (COVID-19) / Т.М. Алексеева [и др.] // Журнал инфектологии. – 2021. – Т.13, № 4. – С. 127–132.; Jakubíkova M. Predictive factors for a severe course of COVID-19 infection in myasthenia gravis patients with an overall impact on myasthenic outcome status and survival / Jakubíkova M., Tyblova M., Tesar A., Horakova M. // Eur J Neurol. – 2021. – Vol.28, №10. – Р. 3418-3425.; Kim Y. Outcomes in myasthenia gravis patients: analysis from electronic health records in the United States / Kim Y. [et al.] // Front Neurol. – 2022.; Muppidi S. COVID-19-associated risks and effects in myasthenia gravis (CARE-MG) / Muppidi S. [et al.] // Lancet Neurol. – 2020. – Vol.19, №12. – Р. 970-971.; Emamikhah M. Opsoclonus-myoclonussyndrome, a post-infectious neurologic complication of COVID-19: case series and review of literature / Emamikhah M. [et al.] // Journal of neurovirology. – 2021. – Vol.1, №9.; Urrea-Mendoza E. Opsoclonus-Myoclonus-Ataxia Syndrome (OMAS) associated with SARS-CoV-2 infection: post-infectious neurological complication with benign prognosis / UrreaMendoza E., Okafor K. // J Neurovirol. – 2021. – Vol.11, №7.; Fernandes J. Opsoclonus myoclonus ataxia syndrome in severe acute respiratory syndrome coronavirus-2 / Fernandes J., Puhlmann P. // Journal of Neurovirology. – 2021. – Vol.1, №3.; Roman G. Acute transverse myelitis (ATM. clinical review of 43 patients with COVID-19-associated ATM and 3 post-vaccination ATM serious adverse events with the ChAdOx1 nCoV-19 vaccine (AZD1222) / Roman G., Gracia F. // Frontiers in immunology. – 2021. – Vol.12, №879.; https://journal.niidi.ru/jofin/article/view/1354

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    Academic Journal

    Συγγραφείς: Shkrobanets’, I. D.

    Πηγή: Bukovinian Medical Herald; Vol. 16 No. 4 (64) (2012); 218-222 ; Буковинский медицинский вестник; Том 16 № 4 (64) (2012); 218-222 ; Буковинський медичний вісник; Том 16 № 4 (64) (2012); 218-222 ; 2413-0737 ; 1684-7903

    Περιγραφή αρχείου: application/pdf

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    Academic Journal
  12. 12
    Academic Journal

    Πηγή: Буковинський медичний вісник; Том 15 № 1(57) (2011); 174-177
    Буковинский медицинский вестник; Том 15 № 1(57) (2011); 174-177
    Bukovinian Medical Herald; Vol. 15 No. 1(57) (2011); 174-177

    Περιγραφή αρχείου: application/pdf

    Σύνδεσμος πρόσβασης: http://e-bmv.bsmu.edu.ua/article/view/239094

  13. 13
    Academic Journal

    Πηγή: Bulletin of Scientific Research; No. 1 (2018) ; Вестник научных исследований; № 1 (2018) ; Вісник наукових досліджень; № 1 (2018) ; 2415-8798 ; 1681-276X ; 10.11603/2415-8798.2018.1

    Περιγραφή αρχείου: application/pdf

  14. 14
    Conference

    Συγγραφείς: Обуховская, В. Б.

    Συνεισφορές: Янковская, Анна Ефимовна, Мещерякова, Э. И.

    Relation: Перспективы развития фундаментальных наук : сборник научных трудов XV Международной конференции студентов, аспирантов и молодых ученых, г. Томск, 24-27 апреля 2018 г. Т. 3 : Математика. — Томск, 2018.; http://earchive.tpu.ru/handle/11683/50853

    Διαθεσιμότητα: http://earchive.tpu.ru/handle/11683/50853

  15. 15
  16. 16
    Academic Journal

    Πηγή: Bulletin of Scientific Research; No 3 (2017) ; Вестник научных исследований; № 3 (2017) ; Вісник наукових досліджень; № 3 (2017) ; 2415-8798 ; 1681-276X ; 10.11603/2415-8798.2017.3

    Περιγραφή αρχείου: application/pdf

  17. 17
  18. 18
    Academic Journal

    Πηγή: Meditsinskiy sovet = Medical Council; № 2 (2018); 156-161 ; Медицинский Совет; № 2 (2018); 156-161 ; 2658-5790 ; 2079-701X ; 10.21518/2079-701X-2018-2

    Περιγραφή αρχείου: application/pdf

    Relation: https://www.med-sovet.pro/jour/article/view/2306/2289; Brown CW. Sprue and Its Treatment. London: J Bale, Sons, and Danielson, 1908.; Elders C. Tropical sprue and pernicious anaemia, aetiology and treatment. Lancet, 1925, i: 75–77.; Cooke WT, Thomas-Smith W. Neurological disorders associated with adult coeliac disease. Brain, 1966, 89: 683–722.; Hadjivassiliou M, Gibson A, Davies-Jones GAB, Lobo A, Stephenson TJ, Milford-Ward A. Is cryptic gluten sensitivity an important cause of neurological illness? Lancet, 1996, 347: 369–71.; Luostarinen L, Pirttilä T, Collin P. Coeliac disease presenting with neurological disorders. Eur Neurol, 1999, 42: 132–135.; Holmes GKT. Neurological and psychiatric complications in coeliac disease. In: Gobbi G, Anderman F, Naccarato S, et al, eds. Epilepsy and Other Neurological Disorders in Celiac Disease. London: John Libbey, 1997: 251–264.; Briani C, Zara G, Alaedini A, et al. Neurological complications of coeliac disease and autoimmune mechanisms: a prospective study. J Neuroimmunol, 2008, 195: 171–75.; Chin RL, Latov N, Green P et al. Neurologic Complica tions of Celiac Disease. J Clin Neuromusc Dis, 2004, 5: 129–137.; Hu WT, Murray JA, Greenaway MC, Parisi JE, Josephs KA. Cognitive impairment and celiac disease. Arch Neurol, 2006, 63: 1440–6.; Currie S, Hadjivassiliou M, Clark M, et al. Should we be ‘nervous’ about coeliac disease? Brain abnormalities in patients with coeliac disease referred for neurological opinion. J Neurol Neurosurg Psychiatry, 2012, 83: 1216–21.; Zelnik N, Pacht A, Obeid R, Lerner A. Range of neurological disorders in patients with celiac disease. Pediatrics, 2004, 113: 1672–6.; Всероссийский консенсус по диагностике и лечению целиакии у детей и взрослых. Принят на 42-й Научной сессии ЦНИИГ (2–3 марта 2016 г.). Consilium Medicum. Педиатрия. (Прил.), 2016, 01: 6-19.; Abele M, Bu¨rk K, Schöls L, et al. The aetiology of sporadic adult-onset ataxia. Brain, 2002, 125: 961–968.; Kaplan JG, Pack D, Horoupian D, et al. Distal axonopathy associated with chronic gluten enteropathy: a treatable disorder. Neurology, 1988, 38: 642–645.; Muller AF, Donnelly MT, Smith CM, et al. Neurological complications of celiac disease: a rare but continuing problem. Am J Gastroenterol, 1996, 91: 1430–1435.; Lionetti E, Francavilla R, Pavone P et al. The neurology of coeliac disease in childhood: what is the evidence? A systematic review and metaanalysis. Dev Med Child Neurol, 2010, 52(8): 700-7.; Luostarinen L, Himanen SL, Luostarinen M, et al. Neuromuscular and sensory disturbances in patients with well-treated celiac disease. J Neurol Neurosurg Psychiatry, 2003, 74: 490–494.; Chin RL, Sander HW, Brannagan TH, et al. Celiac neuropathy. Neurology, 2003, 60: 1581–1585.; Parisi P, Pietropaoli N, Ferretti A et al. Role of the gluten-free diet on neurological-EEG findings and sleep disordered breathing in children with celiac disease. Seizure, 2015 Feb, 25: 181-3.; Işikay S, Hizli Ş, Çoşkun S, Yilmaz K Increased tissue transglutaminase levels are associated with increased epileptiform activity in electroencephalography among patients with celiac disease. Arq Gastroenterol, 2015 Dec, 52(4): 272-7.; Lichtwark IT, Newnham ED, Robinson SR, et al. Cognitive impairment in coeliac disease improves on a gluten-free diet and correlates with histological and serological indices of disease severity. Aliment Pharmacol Ther, 2014, 40: 160–70.; Volta U, Caio G, De Giorgio R et al. Non-celiac gluten sensitivity: a work-in-progress entity in the spectrum of wheat-related disorders. Best Pract Res Clin Gastroenterol, 2015 Jun, 29(3): 477-91.; Volta U, Bardella MT, Calabr A et al. Study Group for Non-Celiac Gluten Sensitivity. An Italian prospective multicenter survey on patients suspected of having non-celiac gluten sensitivity. BMC Med, 2014, 12: 85.; Francavilla R, Cristofori F, Castellaneta S et al. Clinical, serologic, and histologic features of gluten sensitivity in children. J Pediatr, 2014, 164: 463e7.; McElhanon BO, McCracken C, Karpen S, Sharp WG (2014) Gastrointestinal symptoms in autism spectrum disorder: a metaanalysis. Pediatrics, 133: 872–883.; Piwowarczyk A, Horvath A, Łukasik J et al. Glutenand casein-free diet and autism spectrum disorders in children: a systematic review. Eur J Nutr, 2017 Jun 13. doi:10.1007/s00394-017-1483-2.; Lyra L, Rizzo LE, Sunahara CS et al. What do Cochrane systematic reviews say about interventions for autism spectrum disorders? Sao Paulo Med J, 2017 Mar-Apr, 135(2): 192-201.; Sathe N, Andrews JC, McPheeters ML et al. Nutritional and Dietary Interventions for Autism Spectrum Disorder: A Systematic Review. Pediatrics, 2017 Jun, 139(6). pii: e20170346.; Bender L. 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Biol Psychiatry, 1982, 17(5): 627–629.; Arroll MA, Wilder L, Neil J. Nutritional interventions for the adjunctive treatment of schizophrenia: a brief review. Nutrition Journal, 2014, 13: 91.; Murray-Kolb LE, Beard JL. Iron treatment normalizes cognitive functioning in young women. Am J Clin Nutr, 2007, 85: 778–87.; Balion C, Griffith L, Strifler L, et al. Vitamin D, cognition, and dementia: a systematic review and meta-analysis. Neurology, 2012, 79: 1397–405.; Ramos M, Allen L, Mungas D, et al. Lowfolate status is associated with impaired cognitive function and dementia in the Sacramento Area Latino Study on Aging. Am J Clin Nutr, 2005, 82: 1346–52.; Kieslich M, Errázuriz G, Posselt HG, Moeller-Hartmann W, Zanella F, Boehles H. Brain white-matter lesions in celiac disease: a prospective study of 75 diet-treated patients. Pediatrics, 2001, 108(2): E21.; Husby S, Koletzko S, Korponay-Szabó IR et al. European Society for Pediatric Gastroente rology, Hepatology, and Nutrition guidelines for the diagnosis of coeliac disease. J Pediatr Gastroenterol Nutr, 2012 Jan, 54(1): 136-60.; Udit S, Gautron L. Molecular anatomy of the gutbrain axis revealed with transgenic technologies: implications in metabolic research. Front. Neurosci., 2013, 7: 134.; Karakuła-Juchnowicz H, Dzikowski M, Pelczar ska A et al. The brain-gut axis dysfunctions and hypersensitivity to food antigens in the etiopathogenesis of schizophrenia. Psychiatr Pol, 2016, 50(4): 747-760.; de Magistris L, Familiari V, Pascotto A et al (2010) Alterations of the intestinal barrier in patients with autism spectrum disorders and in their first-degree relatives. J Pediatr Gastroenterol Nutr, 51: 418–424.; D’Eufemia P, Celli M, Finocchiaro R et al. Abnormal intestinal permeability in children with autism. Acta Paediatr, 1996, 85: 1076–1079.; Lu R, Wang W, Uzzau S et al. Affinity purification and partial characterization of the zonulin/zonula occludens toxin (zot) receptor from human brain. J. Neurochem., 2000, 74: 320–326.; Shattock P, Kennedy A, Rowell F, Berney T. Role of neuropeptides in autism and their relationships with classical neurotransmitters. Brain Dysfunct, 1990, 3: 328–345.; Ly V, Bottelier M, Hoekstra PJ et al. Elimination diets’ efficacy and mechanisms in attention deficit hyperactivity disorder and autism spectrum disorder. Eur Child Adolesc Psychiatry, 2017 Feb 11. doi:10.1007/s00787-017-0959-1.; Choi S, DiSilvio B, Fernstrom MH, Fernstrom JD. Meal ingestion, aminoacids and brain neurotransmitters: effects of dietary protein source on serotonin and catecholamine synthesis rats. Physiol Behav, 2009, 98: 156–62.; Schuppan D, Pickert G, Ashfaq-Khan M et al. Non-celiac wheat sensitivity: differential diagnosis, triggers and implications. Best Pract Res Clin Gastroenterol, 2015 Jun, 29(3): 469-76.; Turrin NP, Plata-Salam_an CR. Cytokine-cytokine interactions and the brain. Brain Res Bull, 2000, 51: 3–9.; Banks WA, Farr SA, Morley JE. Entry of bloodborne cytokines into the central nervous system: effects on cognitive processes. Neuroimmunomodulation, 2002, 10: 319.; Karakuła-Juchnowicz H, Dzikowski M, Pelczarska A et al. The brain-gut axis dysfunctions and hypersensitivity to food antigens in the etiopathogenesis of schizophrenia. Psychiatr Pol, 2016, 50(4): 747-760.; Wang L, Christophersen CT, Sorich MJ et al. Elevated fecal short chain fatty acid and ammonia concentrations in children with autism spectrum disorder. Dig. Dis. Sci., 2012, 57: 2096–2102.; Thomas RH, Meeking MM, Mepham JR et al. The enteric bacterial metabolite propionic acid alters brain and plasma phospholipid molecular species: further development ofa rodent model of autism spectrum disorders. J. Neuroinflammation, 2012, 9: 153.; Sandler RH, Finegold SM, Bolte ER, Buchanan CP, Maxwell AP, Vaisanen ML. et al. 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Consumption of fermented milk product with probiotic modulates brain activity. Gastroenterology, 2013, 144: 1394–401.

  19. 19
  20. 20
    Conference

    Συνεισφορές: Пеккер, Яков Семенович

    Relation: Молодежь и современные информационные технологии : сборник трудов XIV Международной научно-практической конференции студентов, аспирантов и молодых ученых, г. Томск, 7-11 ноября 2016 г. Т. 1. — Томск, 2016.; http://earchive.tpu.ru/handle/11683/37009

    Διαθεσιμότητα: http://earchive.tpu.ru/handle/11683/37009