Showing 1 - 20 results of 124 for search '"ГИПЕРАЛЬДОСТЕРОНИЗМ"', query time: 0.79s Refine Results
  1. 1
  2. 2
  3. 3
    Academic Journal

    Source: Mìžnarodnij Endokrinologìčnij Žurnal, Vol 13, Iss 8, Pp 604-611 (2017)
    INTERNATIONAL JOURNAL OF ENDOCRINOLOGY; Том 13, № 8 (2017); 604-611
    Международный эндокринологический журнал-Mìžnarodnij endokrinologìčnij žurnal; Том 13, № 8 (2017); 604-611
    Міжнародний ендокринологічний журнал-Mìžnarodnij endokrinologìčnij žurnal; Том 13, № 8 (2017); 604-611

    File Description: application/pdf

  4. 4
    Academic Journal

    Source: Mìžnarodnij Endokrinologìčnij Žurnal, Vol 13, Iss 5, Pp 354-365 (2017)
    INTERNATIONAL JOURNAL OF ENDOCRINOLOGY; Том 13, № 5 (2017); 354-365
    Международный эндокринологический журнал-Mìžnarodnij endokrinologìčnij žurnal; Том 13, № 5 (2017); 354-365
    Міжнародний ендокринологічний журнал-Mìžnarodnij endokrinologìčnij žurnal; Том 13, № 5 (2017); 354-365

    File Description: application/pdf

  5. 5
    Academic Journal

    Source: Mìžnarodnij Endokrinologìčnij Žurnal, Vol 15, Iss 4, Pp 351-357 (2019)
    INTERNATIONAL JOURNAL OF ENDOCRINOLOGY; Том 15, № 4 (2019); 351-357
    Международный эндокринологический журнал-Mìžnarodnij endokrinologìčnij žurnal; Том 15, № 4 (2019); 351-357
    Міжнародний ендокринологічний журнал-Mìžnarodnij endokrinologìčnij žurnal; Том 15, № 4 (2019); 351-357

    File Description: application/pdf

  6. 6
  7. 7
    Academic Journal

    Source: Rossiyskiy Vestnik Perinatologii i Pediatrii (Russian Bulletin of Perinatology and Pediatrics); Том 68, № 3 (2023); 12-20 ; Российский вестник перинатологии и педиатрии; Том 68, № 3 (2023); 12-20 ; 2500-2228 ; 1027-4065

    File Description: application/pdf

    Relation: https://www.ped-perinatology.ru/jour/article/view/1820/1371; An online catalog of human genes and genetic disorders Electronic resource. Electronic data. Baltimore: Johns Hopkins Univ., 2022; Mode of access. https://www.google.com/search?q=OMIM+Bartter+syndrome&rlz=1C1GCEU_ruRU1012RU1012&oq=OMIM+Bartter+syndrome+&aqs=chrome.69i57j69i64.25867j0j7&sourceid=chrome&ie=UTF-8 / Ссылка активна на 21.12.2022; The portal for rare diseases and orphan drugs: https://www.orpha.net/consor/cgi-bin/index.php / Ссылка активна на 21.12.2022.; Vargas-Poussou R. Pathophysiological aspects of the thick ascending limb and novel genetic defects: HELIX syndrome and transient antenatal Bartter syndrome. Pediatr Nephrol 2022; 37(2): 239–252. DOI:10.1007/s00467–021–05019–6; Савенкова Н.Д., Левиашвили Ж.Г., Андреева Э.Ф., Семенова О.А., Папаян К.А. Наследственные болезни почек у детей. Под ред. Н.Д. Савенковой. СПб.: Левша. Санкт-Петербург, 2020; 440.; Satlin L.M., Bockenhauer D. Physiology of the developing kidney: potassium homeostasis and its disorder. In: Pediatric Nephrology. Editors E.D. Avner, W.E. Harmon, P. Niaudet, N. Yoshikawa, F. Emma, F.L. Goldstein. Springer-Verlag, Berlin, 2016; 219–246.; Madden N., Trachtman H. Physiology of the developing kidney: sodium and water homeostasis and its disorders. In: Pediatric Nephrology. Editors E.D. Avner, W.E. Harmon, P. Niaudet, N. Yoshikawa, F. Emma, F.L. Goldstein. Springer-Verlag, Berlin, 2016; 181–217.; Palmer B.F., Clegg D.J. Physiology and pathophysiology of potassium homeostasis. Adv Physiol Educ 2016; 40(4): 480–490. DOI:10.1152/advan.00121.2016; Mount D.B. Thick ascending limb of the loop of Henle. Clin J Am Soc Nephrol 2014; 9(11): 1974–1986. DOI:10.2215/CJN.04480413; Kermond R., Mallett A., McCarthy H. A clinical approach to tubulopathies in children and young adults. Pediatr Nephrol 2023; 38(3): 651–662. DOI:10.1007/s00467–022–05606–1; Guo W., Ji P., Xie Y. Genetic diagnosis and treatment of hereditary renal tubular disease with hypokalemia and alkalosis. J Nephrol 2023; 36(2): 575–591. DOI:10.1007/s40620–022–01428–4; Besouw M.T.P., Kleta R., Bockenhauer D. Bartter and Gitelman syndromes: Questions of class. Pediatr Nephrol 2020; 35(10): 1815–1824. DOI:10.1007/s00467–019–04371-y; Kleta R., Bockenhauer D. Salt-Losing Tubulopathies in Children: What’s New, What’s Controversial? J Am Soc Nephrol 2018; 29(3): 727–739. DOI:10.1681/ASN.2017060600; Plumb L.A., Van’t Hoff W., Kleta R., Reid C., Ashton E., Samuels M. et al. Renal apnoea: extreme disturbance of homoeostasis in a child with Bartter syndrome type IV. Lancet 2016; 388(10044): 631–632. DOI:10.1016/S0140–6736(16)00087–8; Baum M., Anslow M. Postnatal Renal Maturation In: Pediatric Nephrology Editors Emma F., Goldstein S.L., Bagga A., Bates CM., Shroff R. Springer Naturee Switzerland AG, 2022; 1: 37–78. DOI:10.1007/978–3–030–52719–8; Krämer B.K., Bergler T., Stoelcker B., Waldegger S. Mechanisms of Disease: the kidney-specific chloride channels Cl- CKA and ClCKB, the Barttin subunit, and their clinical relevance. Nat Clin Pract Nephrol 2008; 4(1): 38–46. DOI:10.1038/ncpneph0689; Koulouridis E., Koulouridis I. Molecular pathophysiology of Bartter’s and Gitelman’s syndromes. World J Pediatr 2015; 11(2): 113–125. DOI:10.1007/s12519–015–0016–4; Gong Y., Hou J. Claudins in barrier and transport function-the kidney. Pflugers Arch 2017; 469(1): 105–113. DOI:10.1007/s00424–016–1906–6; Muto S. Physiological roles of claudins in kidney tubule paracellular transport. Am J Physiol Renal Physiol 2017; 312(1): F9–F24. DOI:10.1152/ajprenal.00204.2016; Prot-Bertoye C., Houillier P. Claudins in Renal Physiology and Pathology. Genes (Basel) 2020; 11(3): 290. DOI:10.3390/genes11030290; Gong Y., Renigunta V., Himmerkus N., Zhang J., Renigunta A., Bleich M. et al. Claudin-14 regulates renal Ca2⁺ transport in response to CaSR signalling via a novel microRNA pathway. EMBO J 2012; 31(8): 1999–2012. DOI:10.1038/emboj.2012.49; Dimke H., Desai P., Borovac J., Lau A., Pan W., Alexander R.T. Activation of the Ca2+-sensing receptor increases renal claudin-14 expression and urinary Ca2+ excretion. Am J Physiol Renal Physiol 2013; 304(6): F761–769. DOI:10.1152/ajprenal.00263.2012; Simon D.B., Lu Y., Choate K.A., Velazquez H., Al-Sabban E., Praga M. et al. Paracellin-1, a renal tight junction protein required for paracellular Mg2+ resorption. Science 1999; 285(5424): 103–106. DOI:10.1126/science. 285.5424. 103; Konrad M., Schaller A., Seelow D., Pandey A.V., Waldegger S., Lesslauer A. et al. Mutations in the tight-junction gene claudin 19 (CLDN19) are associated with renal magnesium wasting, renal failure, and severe ocular involvement. Am J Hum Genet 2006; 79(5): 949–957. DOI:10.1086/508617; Thorleifsson G., Holm H., Edvardsson V., Walters G.B., Styrkarsdottir U., Gudbjartsson D.F. et al. Sequence variants in the CLDN14 gene associate with kidney stones and bone mineral density. Nat Genet 2009; 41(8): 926–930. DOI:10.1038/ng.404; Guha M., Bankura .B, Ghosh S., Pattanayak A.K., Ghosh S., Pal D.K. et al. Polymorphisms in CaSR and CLDN14 Genes Associated with Increased Risk of Kidney Stone Disease in Patients from the Eastern Part of India. PLoS One 2015; 10(6): e0130790. DOI:10.1371/journal.pone.0130790; Corre T., Olinger E., Harris S.E., Traglia M., Ulivi S., Lenarduzzi S. et al. Common variants in CLDN14 are associated with differential excretion of magnesium over calcium in urine. Pflugers Arch 2017; 469(1): 91–103. DOI:10.1007/s00424–016–1913–7; Lifton R.P, School I. Molecular Genetics of Gitelman’s and Bartter’s Syndromes and their lmplications for Blood Pressure Variation. In: Genetic Disease of the Kidney. Editors R.P. Lifton, S. Somlo, G.H. Giebisch, D.W. Seldin Elsevier 2009; 229–247; Waldegger S., Konrad M., Bartter-, Gitelman-, and Related Syndromes In Pediatric Kidney Disease Chapter Editors D.F. Geary, F. Schaefer Springer-Verlag Berlin Heidelberg, 2016; 905–920; Emma F., van’t Hoff W.G., Vici C.D. Renal Manifestations of Metabolic Disordersin Children. In: Pediatric Nephrology. Editors E.D. Avner, W.E. Harmon, P. Niaudet, N. Yoshikawa, F. Emma, S.L. Goldstein Springer-Verlag, Berlin Heidelberg 2016; 2730. DOI:10.1007/978–3–662–43596–07; Sardani Y., Qin K., Haas M., Aronson A.J., Rosenfield R.L. Bartter syndrome complicated by immune complex nephropathy. Case report and literature review. Pediatr Nephrol 2003; 18(9): 913–9188. DOI:10.1007/s00467–003–1194–1; Seyberth H.W. An improved terminology and classification of Bartter-like syndromes. Nat Clin Pract Nephrol 2008; 4(10): 560–567. DOI: 10. 1038/ ncpneph0912; Seyberth H.W., Weber S., Kömhoff M. Bartter’s and Gitelman’s syndrome. Curr Opin Pediatr 2017; 29(2): 179–186. DOI:10.1097/MOP.0000000000000 447; Bongers E.M.H.F., Shelton L.M., Milatz S., Verkaart S., Bech A.P., Schoots J. et al. Novel Hypokalemic-Alkalotic Salt-Losing Tubulopathy in Patients with CLDN10 Mutations. J Am Soc Nephrol 2017; 28(10): 3118–3128. DOI:10.1681/ASN.2016080881; Hadj-Rabia S., Brideau G., Al-Sarraj Y., Maroun RC., Figueres M.L., Leclerc-Mercier S. et al. Multiplex epithelium dysfunction due to CLDN10 mutation: the HELIX syndrome. Genet Med 2018; 20(2): 190–201. DOI:10.1038/ gim.2017.71; Klar J., Piontek J., Milatz S., Tariq M., Jameel M., Breiderhoff T. et al. Altered paracellular cation permeability due to a rare CLDN10B variant causes anhidrosis and kidney damage. PLoS Genet 2017; 13(7): e1006897. DOI:10.1371/journal.pgen.1006897; Laghmani K., Beck B.B., Yang S.S., Seaayfan E., Wenzel A., Reusch B. et al. Transient Antenatal Bartter’s Syndrome, and MAGED2 Mutations. N Engl J Med 2016; 374(19): 1853–1863. DOI:10.1056/NEJMoa1507629; Левиашвили Ж.Г., Савенкова Н.Д. Барттер синдром у детей. Обзор литературы. Нефрология 2012; 16(3): 25–33. Leviashvili Zh.G., Savenkova N.D. Bartter syndrome in children. Literature review. Nefrologija 2012; 16(3): 25–33. (in Russ.); Левиашвили Ж.Г., Савенкова Н.Д., Левичева О.В., Снежкова Е.А. Катамнестическое наблюдение детей с синдромом Bartter и Gitelman. Нефрология 2013; 17(3): 80–87.; Левиашвили Ж.Г., Савенкова Н.Д. Справочник по наследственным синдромам с патологией почек у детей. Под ред. Н.Д. Савенковой. СПб.: Левша. Санкт-Петербург, 2015; 104.; Mrad F.C.C., Soares S.B.M., de Menezes Silva L.A.W., Dos Anjos Menezes P.V., Simões-E-Silva A.C. Bartter’s syndrome: clinical findings, genetic causes and therapeutic approach. World J Pediatr 2021; 17(1): 31–39. DOI:10.1007/s12519–020–00370–4; Devuyst O., Zennaro M.C., Vargas-Poussou R., Satlin L.M. Inherited Disorders of Sodium and Potassium Handling. In: Pediatric Nephrology. Editors Emma F., Goldstein S., Bagga A., Bates, C.M., Shroff, R. Springer, Berlin, Heidelberg. 2022; 987–1032. DOI:10.1007/978–3–642–27843–3_108–1; Левиашвили Ж.Г., Савенкова Н.Д. Клинико-генетические характеристики Bartter и Gitelman синдромов у детей. Нефрология. 2020; 24(3): 42–53.; Левиашвили Ж.Г., Савенкова Н.Д. Тубулопатии с ведущим синдромом алкалоза Bartter, Gitelman, Lidlle синдромы у детей. В книге: Наследственные болезни почек у детей. Руководство для врачей. Редакторы Н.Д. Савенкова, Ж.Г. Левиашвили, Э.Ф. Андреева, О.А. Семенова, К.А. Папаян. СПб.: Левша. Санкт-Петербург, 2020; 153–186.; Seys E., Andrini O., Keck M., Mansour-Hendili L., Courand P.Y., Simian C. et al. Clinical and Genetic Spectrum of Bartter Syndrome Type 3. J Am Soc Nephrol 2017; 28(8): 2540–2552. DOI:10.1681/ASN.2016101057; Wingo C.S., Stockand J.D. Alkaline activation of ClC-K2 chloride channels switches renal cells from reabsorbing to secreting. J Gen Physiol 2016; 148(3): 195–199. DOI:10.1085/jgp.201611669; Walsh P.R., Tse Y., Ashton E., Iancu D., Jenkins L., Bienias M., et all. Clinical and diagnostic features of Bartter and Gitelman syndromes. Clin Kidney J 2018; 11(3): 302–309. DOI:10.1093/ckj/sfx118; Legrand A., Treard C., Roncelin I., Dreux S., Bertholet-Thomas A., Broux F. et al. Prevalence of Novel MAGED2 Mutations in Antenatal Bartter Syndrome. Clin J Am Soc Nephrol 2018; 13(2): 242–250. DOI:10.2215/CJN. 05670517; Günzel D., Stuiver M., Kausalya P.J., Haisch L., Krug S.M., Rosenthal R. et al. Claudin-10 exists in six alternatively spliced isoforms that exhibit distinct localization and function. J Cell Sci 2009; 122(Pt 10): 1507–1517. DOI:10.1242/jcs.040113; Наследственные болезни органов мочевой системы у детей: Руководство для врачей. Под ред. М.С. Игнатовой, В.В. Длина, П.В. Новикова. М.: Оверлей, 2014; 348.; Meyers N., Nelson-Williams C., Malaga-Dieguez L., Kaufmann H., Loring E., Knight J. et al. Hypokalemia Associated With a Claudin 10 Mutation: A Case Report 2019; 73(3): 425–428. DOI:10.1053/j.ajkd.2018.08.015; Vaisbich M.H., Fujimura M.D., Koch V.H. Bartter syndrome: benefits and side effects of long-term treatment. Pediatr Nephrol 2004; 19(8): 858–863. DOI:10.1007/s00467–004–1527–8; Peters M., Jeck N., Reinalter S., Leonhardt A., Tönshoff B., Klaus G. et al. Clinical presentation of genetically defined patients with hypokalemic salt-losing tubulopathies. Am J Med 2002; 112(3): 183–190. DOI:10.1016/s0002–9343(01)01086–5; Mazaheri M., Assadi F., Sadeghi-Bojd S. Adjunctive acetazolamide therapy for the treatment of Bartter syndrome. Int Urol Nephrol 2020; 52(1): 121–128. DOI:10.1007/s11255–019–02351–7

  8. 8
    Academic Journal

    Source: Medical Herald of the South of Russia; Том 12, № 2 (2021); 81-91 ; Медицинский вестник Юга России; Том 12, № 2 (2021); 81-91 ; 2618-7876 ; 2219-8075 ; 10.21886/2219-8075-2021-12-2

    File Description: application/pdf

    Relation: https://www.medicalherald.ru/jour/article/view/1369/828; https://www.medicalherald.ru/jour/article/downloadSuppFile/1369/469; Фомин И.В. Хроническая сердечная недостаточность в Российской Федерации: что сегодня мы знаем и что должны делать. // Российский кардиологический журнал. – 2016. - №8. – С.7-13. DOI:10.15829/1560-4071-2016-8-7-13; Steinberg B.A., Zhao X., Heidenreich P.A., Peterson E.D., Bhatt D.L., et al. Trends in patients hospitalized with heart failure and preserved left ventricular ejection fraction: prevalence, therapies, and outcomes. // Circulation. – 2012. – V.126(1). – P. 65-75. DOI:10.1161/CIRCULATIONAHA.111.080770; Oktay A.A., Rich J.D., Shah S.J. The emerging epidemic of heart failure with preserved ejection fraction. // Curr Heart Fail Rep. – 2013. – V. 10(4). DOI:10.1007/s11897-013-0155-7; Mentz R.J., Bakris G.L., Waeber B., McMurray J.J., Gheorghiade M., et al. The past, present and future of renin-angiotensin aldosterone system inhibition. // Int J Cardiol. – 2013. – V. 167(5). – P. 1677-1687. doi:10.1016/j.ijcard.2012.10.007; Milliez P., Girerd X., Plouin P.-F., Blacher J., Safar M.E., Mourad J.J. Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism. // J Am Coll Cardiol. – 2005. – V. 45(8). – P. 1243-1248. DOI:10.1016/j.jacc.2005.01.015; Murin J. Cardiovascular effects of aldosterone. // Bratisl Lek Listy. – 2005. – V. 106(1). – P. 3-19.; Sata M., Fukuda D. Crucial role of renin-angiotensin system in the pathogenesis of atherosclerosis. // J Med Invest. – 2010. – V. 57(1-2). – P. 12-25. DOI:10.2152/jmi.57.12; Catena C., Colussi G., Nadalini E., Chiuch A., Baroselli S., et al. Cardiovascular outcomes in patients with primary aldosteronism after treatment. // Arch Intern Med. – 2008. – V. 168(1). – P. 80-85. DOI:10.1001/archinternmed.2007.33; Young W.F. Primary aldosteronism: renaissance of a syndrome. // Clin Endocrinol (Oxf). – 2007. – V. 66(5). – P. 607-618. DOI:10.1111/j.1365-2265.2007.02775.x; Шевелёк А.Н. Влияние терапии блокаторами ренин-ангиотензиновой системы на уровень альдостерона крови у больных хронической сердечной недостаточностью с сохраненной фракцией выброса левого желудочка. // Медицинский вестник Юга России. – 2020. – Т.11, №4. – С. 67-73. DOI:10.21886/2219-8075-2020-11-4-67-73; Шевелёк А.Н. Взаимосвязь плазменных уровней альдостерона и показателей углеводного обмена при хронической сердечной недостаточности с сохраненной фракцией выброса. // Российский кардиологический журнал. – 2021. – Т.26(1). – С. 3991. DOI:10.15829/1560-4071-2021-3991; Шевелёк А.Н. Взаимосвязь гиперальдостеронемии с электрофизиологической активностью миокарда у больных хронической сердечной недостаточностью с сохраненной фракцией выброса. // Архивъ внутренней медицины. – 2020. – Т.10(5). – С. 382-389. DOI:10.20514/2226-6704-2020-10-5-382-389; Шевелёк А.Н. Взаимосвязь уровня альдостерона крови со структурно-функциональным состоянием левого желудочка у пациентов с хронической сердечной недостаточностью с сохраненной фракцией выброса. // Кардиоваскулярная терапия и профилактика. – 2021. – Т. 20(2). – С. 2640. DOI:10.15829/1728-8800-2021-2640; Resic N., Durak-Nalbantic A., Dzubur A., Begic A., Begic E. Serum aldosterone as predictor of progression of coronary heart disease in patients without signs of heart failure after acute myocardial infarction. // Med Arch. – 2018. – V. 72(6). – P. 406-409. doi:10.5455/medarh.2018.72.406-409; Mignano A., Pitruzzella V., Arnone G., Arnone M.T., Rotolo A. et al. Prognostic role of aldosterone in patients with acute coronary syndrome: short and medium term follow-up. // J Cardiovasc Med (Hagerstown). - 2014. – V. ;15(1). – P. 27-32. DOI:10.2459/JCM.0b013e328364129c; Hillaert M.A., Lentjes E.G., Kemperman H., van der Graaf Y., Nathoe H.M., et al. Aldosterone, atherosclerosis and vascular events in patients with stable coronary artery disease. // Int J Cardiol. – 2013. – V. 167(5). – P. 1929-1935. DOI:10.1016/j.ijcard.2012.05.034; Ivanes F., Susen S., Mouquet F., Pigny P., Cuilleret F., et al. Aldosterone, mortality, and acute ischaemic events in coronary artery disease patients outside the setting of acute myocardial infarction or heart failure. // Eur Heart J. – 2012. – V. 33(2). – P. 191-202. DOI:10.1093/eurheartj/ehr176; Beygui F., Montalescot G., Vicaut E., Rouanet S., Van Belle E., et al. Aldosterone and long-term outcome after myocardial infarction: A substudy of the french nationwide Observatoire sur la Prise en charge hospitalière, l’Evolution à un an et les caRactéristiques de patients présentant un infArctus du myocarde avec ou sans onde Q (OPERA) study. // Am Heart J. – 2009. – V. 157(4). – P. 680-687. DOI:10.1016/j.ahj.2008.12.013; Tomaschitz A., Pilz S., Ritz E., Meinitzer A., Boehm B.O., März W. Plasma aldosterone levels are associated with increased cardiovascular mortality: the Ludwigshafen Risk and Cardiovascular Health (LURIC) study. // Eur Heart J. 2010. – V. 31(10). – P. 1237-1247. DOI:10.1093/eurheartj/ehq019; Hayashi M., Tsutamoto T., Wada A., Tsutsui T., Ishii C., et al. Immediate administration of mineralocorticoid receptor antagonist spironolactone prevents post-infarct left ventricular remodeling associated with suppression of a marker of myocardial collagen synthesis in patients with first anterior acute myocardial infarction. // Circulation. – 2003. – V. 107(20). – P. 2559-2565. DOI:10.1161/01.CIR.0000068340.96506.0F; Barter P.J., Caulfield M., Eriksson M., Grundy S.M., Kastelein J.J., et al. Effects of torcetrapib in patients at high risk for coronary events. // N Engl J Med. – 2007. – V. 357(21). – P. 2109-2122. DOI:10.1056/NEJMoa0706628; Schwartz G.G., Olsson A.G., Abt M., Ballantyne C.M., Barter P.J., et al. Effects of dalcetrapib in patients with a recent acute coronary syndrome. // N Engl J Med. – 2012. – V. 367(22). – P. 2089-2099. DOI:10.1056/NEJMoa1206797; Lincoff A.M., Nicholls S.J., Riesmeyer J.S., Barter P.J., Brewer H.B., et al. Evacetrapib and cardiovascular outcomes in high-risk vascular disease. // N Engl J Med. – 2017. – V.376(20). – P. 1933-1942. DOI:10.1056/NEJMoa1609581; Swedberg K., Eneroth P., Kjekshus J., Wilhelmsen L. Hormones regulating cardiovascular function in patients with severe congestive heart failure and their relation to mortality. CONSENSUS Trial Study Group. // Circulation. – 1990. – V. 82(5). – P. 1730-1736. DOI:10.1161/01.cir.82.5.1730; Güder G., Bauersachs J., Frantz S., Weismann D., Allolio B., et al. Complementary and incremental mortality risk prediction by cortisol and aldosterone in chronic heart failure. // Circulation. – 2007. – V. 115(13). – P. 1754-1761. DOI:10.1161/CIRCULATIONAHA.106.653964; Ватутин Н.Т., Шевелёк А.Н. Влияние коморбидных состояний на уровень альдостерона крови у больных с хронической сердечной недостаточностью с сохраненной систолической функцией левого желудочка. // Кардиоваскулярная терапия и профилактика. – 2017. – Т.16(6). – С. 92-98. DOI:10.15829/1728-8800-2017-6-92-98; Ezekowitz J.A., McAlister F.A. Aldosterone blockade and left ventricular dysfunction: a systematic review of randomized clinical trials. // Eur Heart J. – 2009. – V. 30(4). – P. 469-77. DOI:10.1093/eurheartj/ehn543; Xu Y., Qiu Z., Yang R., Wu Y., Cheng X. Efficacy of mineralocorticoid receptor antagonists in postmyocardial infarction patients with or without left ventricular dysfunction: A meta-analysis of randomized controlled trials. // Medicine (Baltimore). – 2018. – V. 97(51). – P. e13690. DOI:10.1097/MD.0000000000013690; https://www.medicalherald.ru/jour/article/view/1369

  9. 9
    Academic Journal

    Source: Almanac of Clinical Medicine; Vol 49, No 5 (2021); 335-341 ; Альманах клинической медицины; Vol 49, No 5 (2021); 335-341 ; 2587-9294 ; 2072-0505

    File Description: application/pdf

  10. 10
    Academic Journal
  11. 11
    Academic Journal

    Contributors: Работа частично выполнена по Государственному заданию в рамках бюджетной темы №АААА-А17- 117112850280-2

    Source: Rational Pharmacotherapy in Cardiology; Vol 16, No 4 (2020); 557-563 ; Рациональная Фармакотерапия в Кардиологии; Vol 16, No 4 (2020); 557-563 ; 2225-3653 ; 1819-6446

    File Description: application/pdf

    Relation: https://www.rpcardio.com/jour/article/view/2253/2013; Williams B., Mancia G., Spiering W., et al.; ESC Scientific Document Group. 2018 ESC/ESH Guidelines for the management of arterial hypertension. Eur Heart J. 2018;39(33):3021-104. DOI:10.1093/eurheartj/ehy339.; Funder J.W., Carey R.M., Mantero F., et al. The management of primary aldosteronism: case detection, diagnosis, and treatment: an endocrine society clinical practice guideline. J Clin Endocrinol Metab. 2016;101(5):1889-916. DOI:10.1210/jc.2015-4061.; Stowasser M. Update in primary aldosteronism. J Clin Endocrinol Metab. 2015;100(1):1-10. DOI:10.1210/jc.2014-3663.; Mulatero P, Monticone S, Bertello C, et al. Long-term cardio- and cerebrovascular events in patients with primary aldosteronism. J Clin Endocrinol Metab. 2013;98(12):4826-33. DOI:10.1210/jc.2013-2805.; Надеева Р.А., Камалиева Г.Р., Ягфарова Р.Р. Первичный гиперальдостеронизм в структуре артериальной гипертонии: актуальность проблемы. Вестник Современной Клинической Медицины. 2015;8(6):98-102. DOI:10.20969/vskm.2015.8(6).98-102.; Young W.F. Jr. Diagnosis and treatment of primary aldosteronism: practical clinical perspectives. J Intern Med. 2019;285(2):126-48. DOI:10.1111/joim.12831.; Milliez P., Girerd X., Plouin P.F., et al. Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism. J Am Coll Cardiol. 2005;45(8):1243-8. DOI:10.1016/j.jacc.2005.01.015.; Monticone S., D'Ascenzo F., Moretti C., et al. Cardiovascular events and target organ damage in primary aldosteronism compared with essential hypertension: a systematic review and meta-analysis. Lancet Diabetes Endocrinol. 2018;6(1):41-50. DOI:10.1016/S2213-8587(17)30319-4.; Prada E.T.A., Burrello J., Reincke M., Williams T.A. Old and New Concepts in the Molecular Pathogenesis of Primary Aldosteronism. Hypertension. 2017;70:875-81. DOI:10.1161/HYPERTENSIONAHA.117.10111.; Чихладзе Н.М., Фаворова О.О., Чазова И.Е. Семейная форма гиперальдостеронизма I типа: клиническое наблюдение и обзор литературы. Терапевтический Архив. 2018;9:115-22. DOI:10.26442/terarkh2018909115-122.; Omata K., Satoh F., Morimoto R., et al. Cellular and Genetic Causes of Idiopathic Hyperaldosteronism. Hypertension. 2018;72(4):874-80. DOI:10.1161/HYPERTENSIONAHA.118.11086.; Liao C.W., Lin Y.T., Wu X.M., et al.; TAIPAI Study Group. The relation among aldosterone, galectin3, and myocardial fibrosis: a prospective clinical pilot follow-up study. J Investig Med. 2016;64(6):1109-13. DOI:10.1136/jim-2015-000014.; Lee H.H., Hung C.S., Wu X.M., et al.; Taipai Study Group. Myocardial ultrasound tissue characterization of patients with primary aldosteronism. Ultrasound Med Biol. 2013;39(1):54-61. DOI:10.1016/j.ultrasmedbio.2012.08.023.; Grytaas M.A., Sellevåg K., Thordarson H.B., et al. Cardiac magnetic resonance imaging of myocardial mass and fibrosis in primary aldosteronism. Endocr Connect. 2018;7(3):413-24. DOI:10.1530/EC18-0039.; Цыганкова О.В., Калинина Е.М., Латынцева Л.Д., Воевода М.И. Успешный опыт коррекции рефрактерной артериальной гипертонии и морбидного ожирения у пациента с синдромом обструктивного апноэ сна тяжелой степени. Российский Кардиологический Журнал. 2018;(5):74-80. DOI:10.15829/1560-4071-2018-5-74-80.; Dudenbostel T., Calhoun D.A. Resistant hypertension, obstructive sleep apnoea and aldosterone. J Hum Hypertens. 2011;26(5):281-7. DOI:10.1038/jhh.2011.4.; Calhoun D.A., Nishizaka M.K., Zaman M.A., Harding S.M. Aldosterone excretion among subjects with resistant hypertension and symptoms of sleep apnea. Chest. 2004;125(1):112-7. DOI:10.1378/chest.125.1.112.; Gonzaga C.C., Gaddam K.K., Ahmed M.I., et al. Severity of obstructive sleep apnea is related to aldosterone status in subjects with resistant hypertension. J Clin Sleep Med. 2010;6(4):363-8. DOI:10.1097/01.hjh.0000379812.66839.11.; Di Murro A., Petramala L., Cotesta D., et al. Renin-angiotensin-aldosterone system in patients with sleep apnoea: prevalence of primary aldosteronism. Journal of the Renin-Angiotensin-Aldosterone System. 2010;11(3):165-72. DOI:10.1177/1470320310366581.; Ахадов Ш.В., Рузбанова Г.Р., Молчанова Г.С. Эволюционные стадии низкорениновой артериальной гипертонии. Рациональная Фармакотерапия в Кардиологии. 2010;6(1):68-72. DOI:10.20996/1819-6446-2010-6-1-68-72.; Hanon O., Boully C., Caillard L., et al. Treatment of hypertensive patients with diabetes and microalbuminuria with combination indapamide SR/amlodipine: retrospective analysis of NESTOR. Am J Hypertens. 2015;28:1064-71. DOI:10.1093/ajh/hpu297.; Маркель А.Л. Генетика и патофизиология низкорениновой артериальной гипертонии. Вавиловский Журнал Генетики и Селекции. 2018;22(8):1000-8. DOI:10.18699/vj18.443.; Мельниченко Г.А., Платонова Н.М., Бельцевич Д.Г. и др. Первичный гиперальдостеронизм: диагностика и лечение. Новый взгляд на проблему. По материалам Проекта клинических рекомендаций Российской ассоциации эндокринологов по диагностике и лечению первичного гиперальдостеронизма. Consilium Medicum. 2017;19(4):75-85. DOI:10.26442/2075-1753_19.4.75-85.; Williams T.A., Reincke M. Management of endocrine disease: Diagnosis and management of primary aldosteronism: the Endocrine Society guideline 2016 revisited. Eur J Endocrinol. 2018;179(1):R19- R29. DOI:10.1530/EJE-17-0990.; Mulatero P., Stowasser M., Loh K.C., et al. Increased diagnosis of primary aldosteronism, including surgically correctable forms, in centers from five continents. Journal of Clinical Endocrinology and Metabolism. 2004;89(3):1045-50. DOI:10.1210/jc.2003-031337.; Дедов И.И., Мельниченко Г.А., ред. Инциденталома надпочечников (диагностика и дифференциальная диагностика). Клинические рекомендации для врачей первичного звена. М.: ФГБУ ЭНЦ; 2015.; Young W.F., Stanson A.W., Thompson G.B., et al. Role for adrenal venous sampling in primary aldosteronism. Surgery. 2004;136(6):1227-35. DOI:10.1016/j.surg.2004.06.051.; Nwariaku F.E., Miller B.S., Auchus R., et al. Primary hyperaldosteronism: effect of adrenal vein sampling on surgical outcome. Arch Surg. 2006;141(5):497-502. DOI:10.1001/archsurg.141.5.497.; Цыганкова О.В., Худякова А.Д., Латынцева Л.Д., Ложкина Н.Г. Сердечно-сосудистый континуум: от факторов риска до систолической сердечной недостаточности. Атеросклероз. 2017;13(4):42-6. DOI:10.15372/ATER20170407.; https://www.rpcardio.com/jour/article/view/2253

  12. 12
    Academic Journal

    Source: Medical Herald of the South of Russia; Том 11, № 4 (2020); 67-73 ; Медицинский вестник Юга России; Том 11, № 4 (2020); 67-73 ; 2618-7876 ; 2219-8075 ; 10.21886/2219-8075-2020-11-4

    File Description: application/pdf

    Relation: https://www.medicalherald.ru/jour/article/view/1098/779; Мареев В.Ю., Фомин И.В., Агеев Ф.Т., Беграмбекова Ю.Л., Васюк Ю.А., и др. Клинические рекомендации ОССН - РКО - РНМОТ. Сердечная недостаточность: хроническая (ХСН) и острая декомпенсированная (ОДСН). Диагностика, профилактика и лечение. // Кардиология. – 2018. – T.58(6S). – C. 8-158. https://doi.org/10.18087/cardio.2475; Мареев В. Ю., Фомин И. В., Агеев Ф. Т., Арутюнов Г.П., Беграмбекова Ю.Л., и др. Хроническая сердечная недостаточность. // Сердечная недостаточность. – 2017. - №18(1)ю – С. 3-40. DOI:10.18087/rhfj.2017.1.2346; Catena C., Colussi G., Brosolo G., Novello M., Sechi L.A. Aldosterone and Left Ventricular Remodeling. // Horm Metab Res Horm Stoffwechselforschung Horm Metab. – 2015. – V.47(13). – P.981-986. doi:10.1055/s-0035-1565055; He B.J., Anderson M.E. Aldosterone and cardiovascular disease: the heart of the matter. // Trends Endocrinol Metab. – 2013. – V.24(1). – P. 21–30. https://doi.org/10.1016/j.tem.2012.09.004; Gueder G., Hammer F., Deutschbein T. Prognostic value of aldosterone and cortisol in patients hospitalized for acutely decompensated chronic heart failure with and without mineralocorticoid receptor antagonism. // J. Card. Fail. – 2015. – V.21(3). – P. 208-16. https://doi.org/10.1016/j.cardfail.2014.12.011; Ватутин НТ, Шевелёк АН. Влияние коморбидных состояний на уровень альдостерона крови у больных с хронической сердечной недостаточностью с сохраненной систолической функцией левого желудочка. // Кардиоваскулярная терапия и профилактика. – 2017. – Т. 16, №6. – С.92-98. doi:10.15829/1728-8800-2017-6-92-98; Lantis A.C., Ames M.K., Atkins C.E., DeFrancesco T.C., Keene B.W., Werre S.R. Aldosterone breakthrough with benazepril in furosemide-activated renin-angiotensinaldosteronesystem in normal dogs. // J Vet Pharmacol Ther. – 2015. – V.38(1). – P.65-73. doi:10.1111/jvp.12154; Prakash E.S. Aldosterone Escape or Refractory Hyperaldosteronism. // Medscape Gen Med. – 2005. – V.7(3). – P.25. PMID: 16369251; PMCID: PMC1681639.; Packer M. The neurohormonal hypothesis: a theory to explain the mechanisms of disease progression in heart failure. // J Am Coll Cardiol. – 1992. – V.20(1). – P. 248-254. doi:10.1016/0735-1097(92)90167-l; Fukuta H., Goto T., Wakami K., Ohte N. Effect of reninangiotensin system inhibitors on mortality in heart failure with preserved ejection fraction: a meta-analysis of observational cohort and randomized controlled studies. // Heart Fail Rev. – 2017. – V.22(6). – P. 775-782. doi:10.1007/s10741-017-9637-0; MacFadyen R.J., Lee A.F., Pringle S.D., Pringle S.D., Struthers A.D. How often are angiotensin II and aldosterone concentrations raised during chronic ACE inhibitor treatment in cardiac failure? // Heart. – 1999. – V.82(1). – P.57-61. doi:10.1136/hrt.82.1.57.; Fuller P.J., Young M.J. Endocrine Affairs of the Heart. / Endocrinology. – 2016. – V.157(7). – P.:2578-2582. doi:10.1210/en.2016-1375; Pitt B., Zannad F., Renne W.J., Cody R., Castaigne A., et al. The effect of spironolactone on morbidity and mortality in patients with severe heart failure: randomized Aldactone Evaluation study investigators. N Engl J Med. 1999;341(10):709- 717. doi:10.1056/NEJM199909023411001; Pitt B. Effect of aldosterone blockade in patients with systolic left ventricular dysfunction: implications of the RALES and EPHESUS studies. // Mol Cell Endocrinol. – 2004. – V.217(1- 2). – P. 53-58. doi:10.1016/j.mce.2003.10.009; Pitt B., Williams G., Remme W., Martinez F., Lopez-Sendon J., et al. The EPHESUS trial: eplerenone in patients with heart failure due to systolic dysfunction complicating acute myocardial infarction. Eplerenone Post-AMI Heart Failure Efficacy and Survival Study. // Cardiovasc Drugs Ther. – 2001. – V.15(1). – P. 79-87. doi:10.1023/a:1011119003788; Desai A.S., Jhund P.S. After TOPCAT: What to do now in Heart Failure with Preserved Ejection Fraction. // Eur Heart J. – 2016. – V.37(41). – P. 3135-3140. doi:10.1093/eurheartj/ehw114; DeFilippis E.M., Desai A.S. Treatment of Hyperkalemia in Heart Failure. // Curr Heart Fail Rep. – 2017. – V.14(4). – P. 266-274. doi:10.1007/s11897-017-0341-0; Almeida S.S., Corgosinho F.C., Amorim C.E., Gregnani M.F., Campos R.M., et al. Different metabolic responses induced by long-term interdisciplinary therapy in obese adolescents related to ACE I/D polymorphism. // J ReninAngiotensin-Aldosterone Syst JRAAS. – 2017. – V.18(2). – P.1470320317703451. doi:10.1177/1470320317703451; Cicoira M., Zanolla L., Rossi A., Golia G., Franceschini L., et al. Failure of aldosterone suppression despite angiotensin-converting enzyme (ACE) inhibitor administration in chronic heart failure is associated with ACE DD genotype. // J. Am. Coll. Card. - 2001. – V.37(7). – P.1808-1812. https://doi.org/10.1016/S1062-1458(01)00481-0; https://www.medicalherald.ru/jour/article/view/1098

  13. 13
    Academic Journal

    Source: The Russian Archives of Internal Medicine; Том 10, № 5 (2020); 382-389 ; Архивъ внутренней медицины; Том 10, № 5 (2020); 382-389 ; 2411-6564 ; 2226-6704

    File Description: application/pdf

    Relation: https://www.medarhive.ru/jour/article/view/1091/944; https://www.medarhive.ru/jour/article/view/1091/952; Fomin I.V. Chronic heart failure in Russian Federation: what do we know and what to do. Russian Journal of Cardiology. 2016; (8): 7-13. doi:10.15829/1560-4071-2016-8-7-13 [in Russian].; Ho K.K., Pinsky J.L., Kannel W.B. et al. The epidemiology of heart failure: the Framingham Study. J Am Coll Cardiol. 1993;22(4 Suppl A):6A-13A. doi:10.1016/0735-1097(93)90455-a.; Manolis A.S., Manolis A.A., Manolis T.A. et al. Sudden death in heart failure with preserved ejection fraction and beyond: an elusive target. Heart Fail Rev. 2019; 24(6): 847-866. doi:10.1007/s10741-019-09804-2.; Shlyahto E.V., Arutyunov G.P., Belenkov Yu.N. National guidelines for risk assessment and prevention of sudden cardiac death (2nd edition). M.: Publishing House «Medpraktika-M». 2018; 247 p. [in Russian].; Beygui F, Collet J-P., Benoliel J-J. et al. High plasma aldosterone levels on admission are associated with death in patients presenting with acute ST-elevation myocardial infarction. Circulation. 2006;114(24):2604-2610. doi:10.1161/CIRCULATIONAHA.106.634626.; Beygui F., Montalescot G., Vicaut E. et al. Aldosterone and longterm outcome after myocardial infarction: A substudy of the french nationwide Observatoire sur la Prise en charge hospitalière, l’Evolution à un an et les caRactéristiques de patients présentant un infArctus du myocarde avec ou sans onde Q (OPERA) study. Am Heart J. 2009;157(4):680-687. doi:10.1016/j.ahj.2008.12.013.; Hundemer GL, Curhan GC, Yozamp N, et al. Incidence of atrial fibrillation and mineralocorticoid receptor activity in patients with medically and surgically treated primary aldosteronism. JAMA Cardiol. 2018;3(8):768-774. doi:10.1001/jamacardio.2018.2003.; Reil J-C, Hohl M, Selejan S, et al. Aldosterone promotes atrial fibrillation. Eur Heart J. 2012;33(16):2098-2108. doi:10.1093/eurheartj/ehr266.; Vatutin N.T., Shevelyok A.N. Infuence of comorbidities on blood aldosterone level in chronic heart failure with preserved systolic function of the left ventricle. Cardiovascular Therapy and Prevention. 2017; 16(6): 92-98. doi:10.15829/1728-8800-2017-6-92-98 [in Russian].; Abdo A., Bebb R.A., Wilkins G.E. Ventricular fibrillation: an extreme presentation of primary hyperaldosteronism. Can J Cardiol. 1999;15(3):347-348.; Shimony A., Bereza S., Shalev A. et al. Ventricular fibrillation as the presenting manifestation of adrenocortical carcinoma. Am Heart Hosp J. 2009;7(1):65-66. doi:10.15420/ahhj.2009.7.1.65.; Zern N.K., Eaton K.D., Roth M.Y. Aldosterone-Secreting Adrenocortical Carcinoma Presenting With Cardiac Arrest. J Endocr Soc. 2019;3(9):1678-1681. doi:10.1210/js.2019-00092.; Vatutin N.T., Shevelok A.N., Kravchenko I.N. The role of aldosterone in the development of atrial fibrillation: modern understanding of problem. The Russian Archives of Internal Medicine. 2019; 9(2): 107-16. doi:10.20514/2226-6704-2019-9-2-107-116 [in Russian].; Briones A.M., Touyz R.M. Aldosterone/MR Signaling, Oxidative Stress, and Vascular Dysfunction. Aldosterone-Miner Recept — Cell Biol Transl Med. Published online September 25, 2019. doi:10.5772/intechopen.87225.; Wu F., Lin Y., Liu Q. The emerging role of aldosterone/mineralocorticoid receptors in the pathogenesis of erectile dysfunction. Endocrine. 2018;61(3):372-382. doi:10.1007/s12020-018-1610-8.; Lalevée N., Rebsamen M.C., Barrère-Lemaire S. et al. Aldosterone increases T-type calcium channel expression and in vitro beating frequency in neonatal rat cardiomyocytes. Cardiovasc Res. 2005;67(2):216-224. doi:10.1016/j.cardiores.2005.05.009; Stepanov D.A., Luiko D.I., Abramov M.A. et al. Mechanisms of sudden cardiac death. Russian Biomedical Research. 2018;3(2):3-14 [in Russian].; Yan Y., Wang C., Lu Y. et al. Mineralocorticoid receptor antagonism protects the aorta from vascular smooth muscle cell proliferation and collagen deposition in a rat model of adrenal aldosterone-producing adenoma. J Physiol Biochem. 2018;74(1):17-24. doi:10.1007/s13105-017-0600-2.; Milliez P., Girerd X., Plouin P.F. et al. Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism. J Am Coll Cardiol. 2005;45(8):1243-1248. doi:10.1016/j.jacc.2005.01.015.; Vatutin N.T., Shevelok A.N., Kravchenko I.N. Blood aldosterone level in various types of atrial fibrillation. Cardiovascular Therapy and Prevention. 2016;15(1):40-44. doi:10.15829/1728-8800-2016-1-40-44 [in Russian].; Goette A., Hoffmanns P., Enayati W. et al. Effect of successful electrical cardioversion on serum aldosterone in patients with persistent atrial fibrillation. Am J Cardiol. 2001;88(8):906-909, A8. doi:10.1016/s0002-9149(01)01905-1.; https://www.medarhive.ru/jour/article/view/1091

  14. 14
  15. 15
  16. 16
  17. 17
  18. 18
  19. 19
  20. 20