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Потенциальные биомаркеры процесса заживления после фистулизирующих антиглаукоматозных операций

Authors: Волкова, Н.В., Юрьева, Т.Н., Курсакова, Ю.В., Малышева, Ю.В.

Source: FYODOROV JOURNAL OF OPHTHALMIC SURGERY ; No. 2 (2020): Офтальмохирургия; 69-76 ; ОФТАЛЬМОХИРУРГИЯ; № 2 (2020): Офтальмохирургия; 69-76 ; 2312-4970 ; 0235-4160

Subject Terms: трабекулэктомия, рубцевание, цитокины, регенераторно-репаративный процесс, неоангиогенез, заживление, лечение рубцевания

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Relation: https://ophthalmosurgery.ru/index.php/ophthalmosurgery/article/view/64/506; https://ophthalmosurgery.ru/index.php/ophthalmosurgery/article/view/64/53; https://ophthalmosurgery.ru/index.php/ophthalmosurgery/article/view/64

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2

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Dynamics of the Activity of MAP-Kinase Cascades in the Healing Process of Postoperative Musculocutaneous Wounds ; Динамика активности MAP-киназных каскадов в процессе заживления послеоперационной кожно-мышечной раны

Authors: I. A. Shurygina, V. A. Umanets, M. G. Shurygin, И. А. Шурыгина, В. А. Уманец, М. Г. Шурыгин

Source: Acta Biomedica Scientifica; Том 4, № 5 (2019); 55-59 ; 2587-9596 ; 2541-9420

Subject Terms: ERK-каскад, healing process, adhesive process, р38 cascade, JNK cascade, ERK cascade, репаративный процесс, спаечный процесс, р38-каскад, JNK-каскад

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Relation: https://www.actabiomedica.ru/jour/article/view/2170/1934; Peng LG, Kerolus JL. Management of Surgical Scars. Facial Plast Surg Clin North Am. 2019; 27(4): 513-517. doi:10.1016/j.fsc.2019.07.013; Goutos I. Intralesional excision as a surgical strategy to manage keloid scars: what’s the evidence? Scars Burn Heal. 2019; 5: 2059513119867297. doi:10.1177/2059513119867297; Shurygin MG, Shurygina IA, Granina GB, Zelenin NV, Ayushinova NI. Using laser confocal microscopy to assess the activity of map kinase systems in the reparative process. Bull Russ Acad Sci Phys. 2016; 80(1): 14-16. https://doi.org/10.3103/S1062873816010214; Шурыгина И.А., Шурыгин М.Г., Зеленин Н.В., Аюшинова Н.И. Воздействие на митогенактивируемые протеинкиназы как новое направление регуляции роста соединительной ткани. Бюллетень сибирской медицины. 2017; 16(4): 86-93. DOI:10.20538/1682-0363-2017-4-86-93; Bhattacharya D, Tiwari R, Bhatia T, Purohit MP, Pal A, Jagdale P, et al. Accelerated and scarless wound repair by a multicomponent hydrogel through simultaneous activation of multiple pathways. Drug Deliv Transl Res. 2019. doi:10.1007/s13346-019-00660-z; Liang CJ, Yen YH, Hung LY, Wang SH, Pu C.M., Chien HF, et al. Thalidomide inhibits fibronectin production in TGF-β1-treated normal and keloid fibroblasts via inhibition of the p38/Smad3 pathway. Biochem Pharmacol. 2013; 85(11): 1594-1602. doi:10.1016/j.bcp.2013.02.038; Song J, Xu H, Lu Q, Xu Z, Bian D, Xia Y, et al. Madecassoside suppresses migration of fibroblasts from keloids: involvement of p38 kinase and PI3K signaling pathways. Burns. 2012; 38(5): 677-684. doi:10.1016/j.burns.2011.12.017; He S, Liu X, Yang Y, Huang W, Xu S, Yang S, et al. Mechanisms of transforming growth factor beta(1)/Smad signalling mediated by mitogen-activated protein kinase pathways in keloid fibroblasts. Br J Dermatol. 2010; 162(3): 538-546. doi:10.1111/j.1365-2133.2009.09511.x; Xia W, Longaker MT, Yang GP. P38 MAP kinase mediates transforming growth factor-beta2 transcription in human keloid fibroblasts. Am J Physiol Regul Integr Comp Physiol. 2006; 290(3): 501-508. DOI:10.1152/ajpregu.00472.2005; Unahabhokha T, Sucontphunt A, Nimmannit U, Chanvorachote P, Yongsanguanchai N, Pongrakhananon V. Molecular signallings in keloid disease and current therapeutic approaches from natural based compounds. Pharm Biol. 2015; 53(3): 457-463. doi:10.3109/13880209.2014.918157; Shurygina IA, Shurygin MG, Ayushinova NI, Granina GB, Zelenin NV. Mechanisms of connective tissue formation and blocks of mitogen activated protein kinase. Front Chem Sci Eng. 2012; 6(2): 232-237. DOI:10.1007/s11705-012-1286-1; Shurygina IA, Shurygin MG, Granina GB, Zelenin NV. Application of mitogen-activated protein kinase inhibitor SP 600125 for wound healing control. J Regen Med Tissue Eng. 2013; 2: 9. DOI:10.7243/2050-1218-2-9; Dolivo DM, Larson SA, Dominko T. FGF2-mediated attenuation of myofibroblast activation is modulated by distinct MAPK signaling pathways in human dermal fibroblasts. J Dermatol Sci. 2017; 88(3): 339-348. doi:10.1016/j.jdermsci.2017.08.013; Lee BC, Song J, Lee A, Cho D, Kim TS. Visfatin promotes wound healing through the activation of ERK1/2 and JNK1/2 pathway. Int J Mol Sci. 2018; 19(11): pii: E3642. doi:10.3390/ijms19113642; https://www.actabiomedica.ru/jour/article/view/2170

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https://doi.org/10.29413/ABS.2019-4.5.9

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