Αποτελέσματα αναζήτησης - "ДОФАМИНЕРГИЧЕСКАЯ СИСТЕМА"

1

Academic Journal

УЧАСТИЕ АДРЕНЕРГИЧЕСКИХ СИСТЕМ ГОЛОВНОГО МОЗГАВ УСЛОВНОРЕФЛЕКТОРНОЙ ДЕЯТЕЛЬНОСТИ

Θεματικοί όροι: условнорефлекторная деятельность, дофаминергическая система, стресс, раздражение, адренергическая стимуляция, катехоламины

2

Academic Journal

УЧАСТИЕ АДРЕНЕРГИЧЕСКИХ СИСТЕМ ГОЛОВНОГО МОЗГАВ УСЛОВНОРЕФЛЕКТОРНОЙ ДЕЯТЕЛЬНОСТИ

Θεματικοί όροι: условнорефлекторная деятельность, дофаминергическая система, стресс, раздражение, адренергическая стимуляция, катехоламины

3

Academic Journal

Substantia nigra alterations in mice modeling Parkinson’s disease ; Изменения в черной субстанции головного мозга у мышей, моделирующих болезнь Паркинсона

Συγγραφείς: I. N. Rozhkova, S. V. Okotrub, E. Yu. Brusentsev, T. A. Rakhmanova, D. A. Lebedeva, V. S. Kozeneva, N. A. Shavshaeva, N. V. Khotskin, S. Ya. Amstislavsky, И. Н. Рожкова, С. В. Окотруб, Е. Ю. Брусенцев, Т. А. Рахманова, Д. А. Лебедева, В. С. Козенева, Н. А. Шавшаева, Н. В. Хоцкин, С. Я. Амстиславский

Συνεισφορές: The work was carried out with the support of RNF No. 23-25-00123.

Πηγή: Vavilov Journal of Genetics and Breeding; Том 28, № 7 (2024); 744-751 ; Вавиловский журнал генетики и селекции; Том 28, № 7 (2024); 744-751 ; 2500-3259 ; 10.18699/vjgb-24-75

Θεματικοί όροι: альфа-синуклеин, Parkinson’s disease, motor coordination, dopaminergic brain system, alpha-synuclein, болезнь Паркинсона, координация движений, дофаминергическая система мозга

Περιγραφή αρχείου: application/pdf

Relation: https://vavilov.elpub.ru/jour/article/view/4347/1881; Bae Y.J., Kim J.M., Sohn C.H., Choi J.H., Choi B.S., Song Y.S., Nam Y., Cho S.J., Jeon B., Kim J.H. Imaging the substantia nigra in Parkinson disease and other Parkinsonian syndromes. Radiology. 2021;300:260-278. DOI 10.1148/radiol.2021203341; Beitz J.M. Parkinson’s disease: a review. Front. Biosci. (Schol. Ed.). 2014;6:65-74. DOI 10.2741/S415; Bidesi N.S., Vang Andersen I., Windhorst A.D., Shalgunov V., Herth M.M. The role of neuroimaging in Parkinson’s disease. J. Neurochem. 2021;159:660-689. DOI 10.1111/jnc.15516; Burre J., Sharma M., Sudhof T.C. Cell biology and pathophysiology of α-synuclein. Cold. Spring. Harb. Perspect. Med. 2018;8:a024091. DOI 10.1101/cshperspect.a024091; Case L.K., Del Rio R., Bonney E.A., Zachary J.F., Blankenhorn E.P., Tung K.S., Teuscher C. The postnatal maternal environment affects autoimmune disease susceptibility in A/J mice. Cell Immunol. 2010; 260:119-127. 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DOI 10.1016/j.parkreldis.2017. 07.033; Dickson D.W., Braak H., Duda J.E., Duyckaerts C., Gasser T., Halliday G.M., Hardy J., Leverenz J.B., Del Tredici K., Wszolek Z.K., Litvan I. Neuropathological assessment of Parkinson’s disease: refining the diagnostic criteria. Lancet Neurol. 2009;8:1150-1157. DOI 10.1016/S1474-4422(09)70238-8; Graham D.R., Sidhu A. Mice expressing the A53T mutant form of human alpha-synuclein exhibit hyperactivity and reduced anxietylike behavior. J. Neurosci. Res. 2010;88:1777-1183. DOI 10.1002/jnr.22331; Grigoryan G.A., Bazyan A.S. The experimental models of Parkinson’s disease in animals. Uspekhi Fiziologicheskikh Nauk = Progress in Physiological Science. 2007;38:80-88 (in Russian); Halliday G.M., Del Tredici K., Braak H. Critical appraisal of brain pathology staging related to presymptomatic and symptomatic cases of sporadic Parkinson’s disease. J. Neural Transm. Suppl. 2006;70:99. DOI 10.1007/978-3-211-45295-0_16; Hayes M.T. Parkinson’s disease and parkinsonism. Am. J. Med. 2019; 132:802-807. DOI 10.1016/j.amjmed.2019.03.001; Holmdahl R., Malissen B. The need for littermate controls. Eur. J. Immunol. 2012;42:45-47. DOI 10.1002/eji.201142048; Jellinger K.A. Pathology of Parkinson’s disease. Mol. Chem. Neuropath. 1991;14:153-197. DOI 10.1007/bf03159935; Kalia L.V., Kalia S.K., McLean P.J., Lozano A.M., Lang A.E. α-Synuclein oligomers and clinical implications for Parkinson disease. Ann. Neurol. 2013;73:155-169. DOI 10.1002/ana.23746; Kato M., Kimura M. Effects of reversible blockade of basal ganglia on a voluntary arm movement. J. Neurophysiol. 1992;68:1516-1534. DOI 10.1152/jn.1992.68.5.1516; Korchounov A., Meyer M.F., Krasnianski M. Postsynaptic nigrostriatal dopamine receptors and their role in movement regulation. J. Neural Transm. (Vienna). 2010;117:1359-1369. DOI 10.1007/s00702-010-0454-z; Korolenko T.A., Shintyapina A.B., Belichenko V.M., Pupyshev A.B., Akopyan A.A., Fedoseeva L.A., Russkikh G.S., Vavilin V.A., Tenditnik M.V., Lin C-L., Amstislavskaya T.G., Tikhonova M.A. Early Parkinson’s disease-like pathology in a transgenic mouse model involves a decreased Cst3 mRNA expression but not neuroinflammatory response in the brain. Med. Univer. 2020;3:66-78. DOI 10.2478/medu-2020-0008; Lai T.T., Kim Y.J., Nguyen P.T., Koh Y.H., Nguyen T.T., Ma H.I., Kim Y.E. Temporal evolution of inflammation and neurodegeneration with alpha-synuclein propagation in Parkinson’s disease mouse model. Fron. Int. Neurosci. 2021;15:715190. DOI 10.3389/fnint.2021.715190; Langley M.R., Ghaisas S., Palanisamy B.N., Ay M., Jin H., Anantharam V., Kanthasamy A., Kanthasamy A.G. Characterization of nonmotor behavioral impairments and their neurochemical mechanisms in the MitoPark mouse model of progressive neurodegeneration in Parkinson’s disease. Exp. Neurol. 2021;341:113716. DOI 10.1016/j.expneurol.2021.113716; Lee F.J., Liu F., Pristupa Z.B., Niznik H.B. Direct binding andfunctional coupling of alpha-synuclein to the dopamine transporters accelerate dopamine-induced apoptosis. FASEB J. 2001;15:916-926. DOI 10.1096/fj.00-0334com; Liu Q., Xu Y., Wan W., Ma Z. An unexpected improvement in spatial learning and memory ability in alpha-synuclein A53T transgenic mice. J. Neural. Transm. (Vienna). 2018;125(2):203-210. DOI 10.1007/s00702-017-1819-3; Maric D., Jahanipour J., Li X.R., Singh A., Mobiny A., Van Nguyen H., Sedlock A., Grama K., Roysam B. Whole-brain tissue mapping toolkit using large-scale highly multiplexed immunofluorescence imaging and deep neural networks. Nat. Commun. 2021;12:1550. DOI 10.1038/s41467-021-21735-x; Nicholas L.M., Ozanne S.E. Early life programming in mice by maternal overnutrition: mechanistic insights and interventional approaches. Philos. Trans. R. Soc. Lond. B. Biol. Sci. 2019;374:20180116. DOI 10.1098/rstb.2018.0116; Oaks A.W., Frankfurt M., Finkelstein D.I., Sidhu A. Age-dependent effects of A53T alpha-synuclein on behavior and dopaminergic function. PLoS One. 2013;8:e60378. DOI 10.1371/journal.pone.0060378; Paumier K.L., Sukoff Rizzo S.J., Berger Z., Chen Y., Gonzales C., Kaftan E., Li L., Lotarski S., Monaghan M., Shen W., Stolyar P., Vasilyev D., Zaleska M., D Hirst W., Dunlop J. Behavioral characterization of A53T mice reveals early and late stage deficits related to Parkinson’s disease. PLoS One. 2013;8(8):e70274. DOI 10.1371/journal.pone.0070274.; Paxinos G., Franklin K. Mouse Brain in Stereotaxic Coordinates. 4th ed. San Diego: Acad. Press, 2012 Poewe W., Seppi K., Tanner C.M., Halliday G.M., Brundin P., Volkmann J., Schrag A.E., Lang A.E. Parkinson disease. Nat. Rev. Dis. Primers. 2017;3:17013. DOI 10.1038/nrdp.2017.13; Polymeropoulos M.H., Lavedan C., Leroy E., Ide S.E., Dehejia A., Dutra A., Pike B., Root H., Rubenstein J., Boyer R., Stenroos E.S., Chandrasekharappa S., Athanassiadou A., Papapetropoulos T., Johnson W.G., Lazzarini A.M., Duvoisin R.C., Di Iorio G., Golbe L.I., Nussbaum R.L. Mutation in the alpha-synuclein gene identified in families with Parkinson’s disease. Science. 1997;276:2045-2047. DOI 10.1126/science.276.5321.2045; Pupyshev A.B., Korolenko T.A., Akopyan A.A., Amstislavskaya T.G., Tikhonova M.A. Suppression of autophagy in the brain of transgenic mice with overexpression of А53Т-mutant α-synuclein as an early event at synucleinopathy progression. Neurosci. Lett. 2018;672: 140-144. DOI 10.1016/j.neulet.2017.12.001; Rozhkova I.N., Okotrub S.V., Brusentsev E.Yu., Rakhmanova T.A., Lebedeva D.A., Kozeneva V.S., Khotskin N.V., Amstislavsky S.Ya. Analysis of behavior and brain neuronal density in B6.Cg-Tg(PrnpSNCA*A53T)23Mkle/J mice modeling, a Parkinson’s disease. Rossiyskiy Fiziologicheskiy Zhurnal imeni I.M. Sechenova = Russian Journal of Physiology. 2023;59:1633-1647. DOI 10.31857/S0869813923090091 (in Russian); Schultz W., Ruffieux A., Aebischer P. The activity of pars compacta neurons of the monkey substantia nigra in relation to motor activation. Exp. Brain Res. 1983;51:377-387. DOI 10.1016/0304-3940(84)90456-7; Seo J.H., Kang S.W., Kim K., Wi S., Lee J.W., Cho S.R. Environmental enrichment attenuates oxidative stress and alters detoxifying enzymes in an A53T α-synuclein transgenic mouse model of Parkinson’s disease. Antioxidants (Basel). 2020;9:928. DOI 10.3390/antiox9100928; Spillantini M.G., Schmidt M.L., Lee V.M., Trojanowski J.Q., Jakes R., Goedert M. Alpha-synuclein in Lewy bodies. Nature. 1997;388: 839-840. DOI 10.1038/42166; Stern G. The effects of lesions in the substantia nigra. Brain. 1966;89: 449-478. DOI 10.1093/brain/89.3.449; Taguchi T., Ikuno M., Hondo M., Parajuli L.K., Taguchi K., Ueda J., Sawamura M., Okuda S., Nakanishi E., Hara J., Uemura N., Hatanaka Y., Ayaki T., Matsuzawa S., Tanaka M., El-Agnaf O.M.A., Koike M., Yanagisawa M., Uemura M.T., Yamakado H., Takahashi R. α-Synuclein BAC transgenic mice exhibit RBD-like behaviour and hyposmia: a prodromal Parkinson’s disease model. Brain. 2020;143:249-265. DOI 10.1093/brain/awz380; Tang H., Gao Y., Zhang Q., Nie K., Zhu R., Gao L., Feng S., Wang L., Zhao J., Huang Z., Zhang Y., Wang L. Chronic cerebral hypoperfusion independently exacerbates cognitive impairment within the pathopoiesis of Parkinson’s disease via microvascular pathologys. Behav. Brain Res. 2017;333:286-294. DOI 10.1016/j.bbr.2017.05.061; Tikhonova M.A., Tikhonova N.G., Tenditnik M.V., Ovsyukova M.V., Akopyan A.A., Dubrovina N.I., Amstislavskaya T.G., Khlestkina E.K. Effects of grape polyphenols on the life span and neuroinflammatory alterations related to neurodegenerative Parkinson disease-like disturbances in mice. Molecules. 2020;25:5339. DOI 10.3390/molecules25225339; Tran J., Anastacio H., Bardy C. Genetic predispositions of Parkinson’s disease revealed in patient-derived brain cells. NPJ Parkinsons Dis. 2020;6:8. DOI 10.1038/s41531-020-0110-8; Unger E.L., Eve D.J., Perez X.A., Reichenbach D.K., Xu Y., Lee M.K., Andrews A.M. Locomotor hyperactivity and alterations in dopamine neurotransmission are associated with overexpression of A53T mutant human alpha-synuclein in mice. Neurobiol. Dis. 2006;21:431- 443. DOI 10.1016/j.nbd.2005.08.005; Van der Putten H., Wiederhold K.H., Probst A., Barbieri S., Mistl C., Danner S., Kauffmann S., Hofele K., Spooren W.P., Ruegg M.A., Lin S., Caroni P., Sommer B., Tolnay M., Bilbe G. Neuropathology in mice expressing human alpha-synuclein. J. Neurosci. 2000;20: 6021-6029. DOI 10.1523/JNEUROSCI.20-16-06021.2000; Venda L., Cragg S., Buchman V.L., Wade-Martins R. α-Synuclein and dopamine at the crossroads of Parkinson’s disease. Trends Neurosci. 2010;12:559-568. DOI 10.1016/j.tins.2010.09.004; Wang Y., Sun Z., Du S., Wei H., Li X., Li X., Shen J., Chen X., Cai Z. The increase of α-synuclein and alterations of dynein in A53T transgenic and aging mouse. J. Clin. Neurosci. 2022;96:154-162. DOI 10.1016/j.jocn.2021.11.002; Wu D., Dean J. Maternal factors regulating preimplantation development in mice. Curr. Top. Dev. Biol. 2020;140:317-340. DOI 10.1016/bs.ctdb.2019.10.006; Zhang Yu., Wu Q., Zhang L., Wang Q., Yang Z., Liu J., Feng L. Caffeic acid reduces A53T α-synuclein by activating JNK/Bcl-2-mediated autophagy in vitro and improves behaviour and protects dopaminergic neurons in a mouse model of Parkinson’s disease. Pharmacol. Res. 2019;150:104538. DOI 10.1016/j.phrs.2019.104538; Zhang Yu., Wu Q., Ren Y., Zhang Y., Feng L. A53T α-synuclein induces neurogenesis impairment and cognitive dysfunction in line M83 transgenic mice and reduces the proliferation of embryonic neural stem cells. Brain Res. Bull. 2022;182:118-129. DOI 10.1016/ j.brainresbull.2022.02.010; Zheng M., Liu Y., Xiao Z., Jiao L., Lin X. Tau knockout and α-synuclein A53T synergy modulated parvalbumin-positive neurons degeneration staging in substantia nigra pars reticulata of Parkinson’s disease-liked model. Front. Aging Neurosci. 2022;13:784665. DOI 10.3389/fnagi.2021.784665.; https://vavilov.elpub.ru/jour/article/view/4347

Διαθεσιμότητα: https://vavilov.elpub.ru/jour/article/view/4347
https://doi.org/10.18699/vjgb-24-82

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4

Academic Journal

Нарушение дофаминергической системы в патофизиологии сахарного диабета ; Disruption of the dopaminergic system in the pathophysiology of diabetes mellitus

Συγγραφείς: Bykov, Yu. V., Baturin, V. A., Быков, Ю. В., Батурин, В. А.

Θεματικοί όροι: DIABETES MELLITUS, DOPAMINERGIC SYSTEM, DOPAMINE, INSULIN, CEREBRAL INSUFFICIENCY, САХАРНЫЙ ДИАБЕТ, ДОФАМИНЕРГИЧЕСКАЯ СИСТЕМА, ДОФАМИН, ИНСУЛИН, ЦЕРЕБРАЛЬНАЯ НЕДОСТАТОЧНОСТЬ

Περιγραφή αρχείου: application/pdf

Relation: Уральский медицинский журнал. 2023. T. 22, № 4.; http://elib.usma.ru/handle/usma/15359

Διαθεσιμότητα: http://elib.usma.ru/handle/usma/15359

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5

Academic Journal

Role of the gene polymorphic variants of the dopaminergic system in the formation of the human psycho-emotional status ; Роль полиморфных вариантов генов дофаминергической системы в формировании психоэмо ционального статуса человека

Συγγραφείς: I. B. Mosse, L. V. Kukhtsinskaya, N. G. Siadlia, T. V. Dokukina, A. V. Kilchevsky, И. Б. Моссэ, Л. В. Кухтинская, Н. Г. Седляр, Т. В. Докукина, А. В. Кильчевский

Συνεισφορές: The results obtained in the project “Development of a methodology for determining the status of genetic loci and lifetime modification of DNA sites affecting the psychoemotional status of a person” of the research- and-technological program of the Union State of Russia and Belarus “DNA Identification”., Работа выполнена в рамках реализации мероприятия «Разработка методики определения статуса генетических локусов и прижизненной модификации участков ДНК, влияющих на психоэмоцио нальный статус человека» НТП Союзного государства «Разра ботка инновационных геногеографических и геном ных технологий идентификации личности и индивидуаль ных особенностей человека на основе изучения генофондов регионов Союзного государства» («ДНК-идентификация»).

Πηγή: Doklady of the National Academy of Sciences of Belarus; Том 66, № 3 (2022); 294-300 ; Доклады Национальной академии наук Беларуси; Том 66, № 3 (2022); 294-300 ; 2524-2431 ; 1561-8323 ; 10.29235/1561-8323-2022-66-3

Θεματικοί όροι: молекулярно-генетический анализ, dopaminergic system, molecular genetic analysis, дофаминергическая система

Περιγραφή αρχείου: application/pdf

Relation: https://doklady.belnauka.by/jour/article/view/1067/1064; Калимуллина, Л. Б. Анализ ассоциаций по сочетаниям генотипов полиморфных ДНК-локусов (tag 1a и Ncoi) Drd2, 256А/G гена Slc6a3 и объемных характеристик миндалевидного комплекса мозга с повышенной тревожностью / Л. Б. Калимуллина, А. В. Ахмадеев, А. Я. Ханнанова // Успехи современного естествознания. – 2011. – № 11. – С. 9–11.; Benjamin, J. Molecular Genetics and the Human Personality / J. Benjamin, R. P. Ebstein, R. H. Belmaker. – American Psychiatric Pub., 2008. – 452 р.; Dopamine multilocus genetic profiles predict sex differences in reactivity of the human reward system / E. K. Diekhof [et al.] // Brain Struct. Funct. – 2021. – Vol. 226, N 4. – Р. 1099–1114. https://doi.org/10.1007/s00429-021-02227-6; A validation study of the Hospital Anxiety and Depression Scale (HADS) in different groups of Dutch subjects / P. Spinhoven [et al.] // Psychol. Med. – 1997. – Vol. 27, N 2. – Р. 363–370. https://doi.org/10.1017/s0033291796004382; Cohen, S. A Global Measure of Perceived Stress / S. Cohen, T. Kamarck, R. Mermelstein // J. Health Soc. Behav. – 1983. – Vol. 24, N 4. – P. 385–396. https://doi.org/10.2307/2136404; Валидизация русскоязычной версии опросника «Шкала воспринимаемого стресса-10» / В. А. Абабков [и др.] // Вестн. С.-Петерб. ун-та. Сер. 16: Психология. Педагогика. – 2016. – № 2. – С. 6–15. https://doi.org/10.21638/11701/spbu16.2016.202; Look beyond Catechol-O-Methyltransferase genotype for cathecolamines derangement in migraine: the BioBIM rs4818 and rs4680 polymorphisms study / M. L. De Marchis [et al.] // J. Headache Pain. – 2015. – Vol. 16, N 1. – P. 37–45. https://doi.org/10.1186/s10194-015-0520-x; Dean, B. Associations between catechol-O-methyltransferase (COMT) genotypes at rs4818 and rs4680 and gene expression in human dorsolateral prefrontal cortex / B. Dean, G. M. Parkin, A. S. Gibbons // Exp. Brain Res. – 2020. – Vol. 238, N 2. – P. 477–486. https://doi.org/10.1007/s00221-020-05730-0; Genomic organization of the human catechol O-methyltransferase gene and its expression from two distinct promoters / J. Tenhunen [et al.] // Eur. J. Biochem. – 1994. – Vol. 223, N 3. – P. 1049–1059. https://doi.org/10.1111/j.1432-1033.1994.tb19083.x; A haplotype implicated in schizophrenia susceptibility is associated with reduced COMT expression in human brain / N. J. Bray [et al.] // Am. J. Hum. Genet. – 2003. – Vol. 73, N 1. – P. 152–161. https://doi.org/10.1086/376578; Effect of COMT Val108/158 Met genotype on frontal lobe function and risk for schizophrenia / M. F. Egan [et al.] // Proc. Natl. Acad. Sci. – 2001. – Vol. 98, N 12. – P. 6917–6922. https://doi.org/10.1073/pnas.111134598; Regulatory Polymorphisms in Human DBH Affect Peripheral Gene Expression and Sympathetic Activity / E. S. Barrie [et al.] // Circ. Res. – 2014. – Vol. 115, N 12. – P. 1017–1025. https://doi.org/10.1161/circresaha.116.304398; Association of regulatory variants of dopamine β-hydroxylase with cognition and tardive dyskinesia in schizophrenia subjects / T. J. Punchaichira [et al.] // J. Psychopharmacol. Oxf. Engl. – 2020. – Vol. 34, N 3. – P. 358–369. https://doi.org/10.1177/0269881119895539; The dopamine β-hydroxylase –1021C/T polymorphism is associated with the risk of Alzheimer’s disease in the Epistasis Project / O. Combarros [et al.] // BMC Med. Genet. – 2010. – Vol. 11, N 1. – Р. 162–172. https://doi.org/10.1186/1471-2350-11-162; Effects of cultural intensity and density regime treatment on post-thinning loblolly pine individual tree DBH increment in the lower coastal plain of the southeastern United States / J. T. Perren [et al.] // Proc. 18th Bienn. South. Silvic. Res. Conf. E-Gen Tech Rep SRS-212 Asheville NC US Dep. Agric. For. Serv. South. Res. Stn. 614 P. – 2016. – Vol. 212. – P. 288–292.; https://doklady.belnauka.by/jour/article/view/1067

Διαθεσιμότητα: https://doklady.belnauka.by/jour/article/view/1067
https://doi.org/10.29235/1561-8323-2022-66-3-294-300

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6

Academic Journal

Functional facets of dopaminergic system and cancer. Part 2

Πηγή: ZHurnal «Patologicheskaia fiziologiia i eksperimental`naia terapiia». :90-96

Θεματικοί όροι: 0301 basic medicine, серотонин, aging, серотониновые рецепторы, 3. Good health, serotonin, 03 medical and health sciences, дофаминовые рецепторы, stress, 0302 clinical medicine, дофаминергическая система, dopaminergic system, рак, serotonin receptors, cancer, стресс, старение, dopamine receptors

Σύνδεσμος πρόσβασης: https://pfiet.ru/article/view/806

7

Academic Journal

Дофаминергическая система головного мозга ; Dopaminergic brain system

Συγγραφείς: Makarov, E. A., Filonov, I. A., Izmozherova, N. V., Bakhtin, V. M., Макаров, Е. А., Филонов, И. А., Изможерова, Н. В., Бахтин, В. М.

Πηγή: Сборник статей

Θεματικοί όροι: ДОФАМИНЕРГИЧЕСКАЯ СИСТЕМА, ДОФАМИНОВЫЕ РЕЦЕПТОРЫ, DOPAMINERGIC SYSTEM, DOPAMINE RECEPTORS

Περιγραφή αρχείου: application/pdf

Relation: Актуальные вопросы современной медицинской науки и здравоохранения: Материалы VI Международной научно-практической конференции молодых учёных и студентов, посвященной году науки и технологий, (Екатеринбург, 8-9 апреля 2021): в 3-х т.; http://elib.usma.ru/handle/usma/5985

Διαθεσιμότητα: http://elib.usma.ru/handle/usma/5985

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8

Academic Journal

Functional facets of dopaminergic system and cancer. Part 1

Πηγή: ZHurnal «Patologicheskaia fiziologiia i eksperimental`naia terapiia». :116-126

Θεματικοί όροι: 0301 basic medicine, aging, 3. Good health, 03 medical and health sciences, дофаминовые рецепторы, stress, 0302 clinical medicine, дофаминергическая система, dopaminergic system, болезнь Паркинсона, депрессивное расстройство, depression, Parkinson's disease, старение, dopamine receptors

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Нейромедиаторный дисбаланс - как основа развития синдрома дефицита внимания с гиперактивностью

Συγγραφείς: С.Н. САИДХОДЖАЕВА, Ё.Н. МАДЖИДОВА

Θεματικοί όροι: поведенческая неврология, СДВГ, дети, тики, дофамин, серотонин, норадреналин, дофаминергическая система, серотонинергическая система, генетические механизмы нейромедиации

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Dopaminergic deficiency syndrome in the picture of severe brain injury in the presence of protracted depression of consciousness ; Cиндром дофаминергической недостаточности в картине тяжелой травмы мозга на фоне длительного угнетения сознания

Συγγραφείς: M. V. Chelyapina, E. V. Sharova, O. S. Zaitsev, М. В. Челяпина, Е. В. Шарова, О. С. Зайцев

Πηγή: Neurology, Neuropsychiatry, Psychosomatics; Vol 6, No 4 (2014); 31-39 ; Неврология, нейропсихиатрия, психосоматика; Vol 6, No 4 (2014); 31-39 ; 2310-1342 ; 2074-2711 ; 10.14412/2074-2711-2014-4

Θεματικοί όροι: амантадина сульфат, dopaminergic system, electroencephalography, severe brain injury, neurotransmitters, amantadine sulfate, дофаминергическая система, электроэнцефалография, тяжелая черепно-мозговая травма, нейротрансмиттерные препараты

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